Matthew P. Harrison

403 citations
7 papers · 336 indexed · h-index 7

Impact in

Papers in

Matthew P. Harrison

7 papers receiving 327 citations

Peers

Matthew P. Harrison
Comparison fields: 5 of 49
  • Pharmacology 271
  • Cellular and Molecular Neuroscience 181
  • Endocrine and Autonomic Systems 43
  • Biological Psychiatry 13
  • Toxicology 17
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Citations per field
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Citations per year

Countries citing papers authored by Matthew P. Harrison

Since Specialization
Citations

This map shows the geographic impact of Matthew P. Harrison's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew P. Harrison with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew P. Harrison more than expected).

Fields of papers citing papers by Matthew P. Harrison

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew P. Harrison. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew P. Harrison. The network helps show where Matthew P. Harrison may publish in the future.

Co-authors

The 25 scholars most cited alongside Matthew P. Harrison, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Matthew P. Harrison Line = papers co-authored together Matthew P. Harrison links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 201014
2 1998186
3 199815
4 199628
5
Changes in red blood cell methotrexate pharmacology and their impact on outcome when cytarabine is infused with methotrexate in the treatment of acute lymphocytic leukemia in children: a pediatric oncology group study.
199614
6 199557
7 199222

About Matthew P. Harrison

Matthew P. Harrison is a scholar working on Pharmacology, Cellular and Molecular Neuroscience, Hematology, Neurology and Public Health, Environmental and Occupational Health, having authored 7 papers that have together received 336 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (4 papers), Cannabis and Cannabinoid Research (4 papers), Acute Myeloid Leukemia Research (2 papers), Neurotransmitter Receptor Influence on Behavior (2 papers), Acute Lymphoblastic Leukemia research (2 papers), Diet and metabolism studies (1 paper), Intracranial Aneurysms: Treatment and Complications (1 paper) and Biochemical Analysis and Sensing Techniques (1 paper). The work is most often cited by research in Pharmacology (271 citations), Cellular and Molecular Neuroscience (181 citations), Endocrine and Autonomic Systems (43 citations), Biological Psychiatry (13 citations) and Toxicology (17 citations). Matthew P. Harrison has collaborated with scholars based in United States, Canada and United Kingdom. Frequent co-authors include Denise Pettit, Guy A. Cabral, Robert F. Spencer, John M. Olson, G. A. Cabral, Francine Marciano‐Cabral, Denise Toney, Krista Fischer‐Stenger, Douglas L. Anders and Elizabeth Hayter. Their work appears in journals such as Cancer Chemotherapy and Pharmacology, Life Sciences, Advances in experimental medicine and biology, Journal of Vascular Surgery and Journal of Neuroscience Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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