Matthew L. Andria
- Molecular Biology
- Reproductive Medicine top 2%
- Public Health, Environmental and Occupational Health top 10%
- Cellular and Molecular Neuroscience top 10%
- Immunology
- Co-authors
- A. Daniel JonesStanley MeizelEric J. SimonH Kenneth KramerShoshana LevyJacob M. HillerS TakahashiVictoria Nguyen
- Topics
- Receptor Mechanisms and Signaling (11 papers)Neuropeptides and Animal Physiology (10 papers)Pharmacological Receptor Mechanisms and Effects (6 papers)
- Partner nations
- United States
In The Last Decade
Matthew L. Andria
22 papers receiving 812 citations
Peers
Comparison fields: 5 of 71
- Molecular Biology 379
- Reproductive Medicine 305
- Public Health, Environmental and Occupational Health 269
- Cellular and Molecular Neuroscience 214
- Immunology 108
Countries citing papers authored by Matthew L. Andria
This map shows the geographic impact of Matthew L. Andria's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew L. Andria with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew L. Andria more than expected).
Fields of papers citing papers by Matthew L. Andria
This network shows the impact of papers produced by Matthew L. Andria. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew L. Andria. The network helps show where Matthew L. Andria may publish in the future.
Co-authorship network of co-authors of Matthew L. Andria
This figure shows the co-authorship network connecting the top 25 collaborators of Matthew L. Andria. A scholar is included among the top collaborators of Matthew L. Andria based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew L. Andria. Matthew L. Andria is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 2 | |
| 3 | 12 | |
| 4 | 87 | |
| 5 | 32 | |
| 6 | 20 | |
| 7 | 41 | |
| 8 | 28 | |
| 9 | 29 | |
| 10 | 15 | |
| 11 | 0 | |
| 12 | 10 | |
| 13 | 11 | |
| 14 | 61 | |
| 15 | 46 | |
| 16 | 355 | |
| 17 | 1 | |
| 18 | 5 | |
| 19 | 8 | |
| 20 | 1 |
About Matthew L. Andria
Matthew L. Andria is a scholar working on Cellular and Molecular Neuroscience, Biotechnology and Immunology and Allergy, having authored 23 papers that have together received 835 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (11 papers), Neuropeptides and Animal Physiology (10 papers) and Pharmacological Receptor Mechanisms and Effects (6 papers). The work is most often cited by research in Reproductive Medicine (305 citations), Physiology (59 citations) and Cellular and Molecular Neuroscience (214 citations). Matthew L. Andria has collaborated with scholars based in United States. Frequent co-authors include A. Daniel Jones, Stanley Meizel, Eric J. Simon, H Kenneth Kramer, Shoshana Levy, Jacob M. Hiller, S Takahashi, Victoria Nguyen, E. Benjamini and Rachel Oren. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Immunology and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.