Matthew K. Robinson
- Molecular Biology
- Radiology, Nuclear Medicine and Imaging top 2%
- Oncology top 10%
- Biomedical Engineering
- Health, Toxicology and Mutagenesis top 5%
- Co-authors
- Gregory P. AdamsJames D. MarksJohn A. McLachlanLouis J. GuilletteSteven F. ArnoldAngelο C. NotidesCalvin ShallerMichael Stern
- Topics
- Monoclonal and Polyclonal Antibodies Research (19 papers)HER2/EGFR in Cancer Research (15 papers)Glycosylation and Glycoproteins Research (9 papers)
- Partner nations
- United StatesUnited KingdomMalaysia
In The Last Decade
Matthew K. Robinson
49 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 117
- Molecular Biology 666
- Radiology, Nuclear Medicine and Imaging 612
- Oncology 389
- Biomedical Engineering 199
- Health, Toxicology and Mutagenesis 182
Countries citing papers authored by Matthew K. Robinson
This map shows the geographic impact of Matthew K. Robinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew K. Robinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew K. Robinson more than expected).
Fields of papers citing papers by Matthew K. Robinson
This network shows the impact of papers produced by Matthew K. Robinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew K. Robinson. The network helps show where Matthew K. Robinson may publish in the future.
Co-authorship network of co-authors of Matthew K. Robinson
This figure shows the co-authorship network connecting the top 25 collaborators of Matthew K. Robinson. A scholar is included among the top collaborators of Matthew K. Robinson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew K. Robinson. Matthew K. Robinson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 1 | |
| 3 | 6 | |
| 4 | 5 | |
| 5 | 4 | |
| 6 | 11 | |
| 7 | 3 | |
| 8 | 6 | |
| 9 | 29 | |
| 10 | 35 | |
| 11 | 8 | |
| 12 | 1 | |
| 13 | 18 | |
| 14 | 103 | |
| 15 | 15 | |
| 16 | 20 | |
| 17 | 34 | |
| 18 | 56 | |
| 19 | 54 | |
| 20 | 8 |
About Matthew K. Robinson
Matthew K. Robinson is a scholar working on Aging, Radiology, Nuclear Medicine and Imaging and Oncology, having authored 51 papers that have together received 1.6k indexed citations. Recurring topics across this work include Monoclonal and Polyclonal Antibodies Research (19 papers), HER2/EGFR in Cancer Research (15 papers) and Glycosylation and Glycoproteins Research (9 papers). The work is most often cited by research in Aging (80 citations), Radiology, Nuclear Medicine and Imaging (612 citations) and Oncology (389 citations). Matthew K. Robinson has collaborated with scholars based in United States, United Kingdom and Malaysia. Frequent co-authors include Gregory P. Adams, James D. Marks, John A. McLachlan, Louis J. Guillette, Steven F. Arnold, Angelο C. Notides, Calvin Shaller, Michael Stern, Mohan Doss and Louis M. Weiner. Their work appears in journals such as Blood, ACS Nano and The Journal of Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.