Matthew J. Bellizzi

1.5k total citations · 1 hit paper
13 papers, 1.2k citations indexed

About

Matthew J. Bellizzi is a scholar working on Neurology, Physiology and Pathology and Forensic Medicine. According to data from OpenAlex, Matthew J. Bellizzi has authored 13 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Neurology, 4 papers in Physiology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Matthew J. Bellizzi's work include Neuroinflammation and Neurodegeneration Mechanisms (8 papers), Alzheimer's disease research and treatments (3 papers) and Multiple Sclerosis Research Studies (3 papers). Matthew J. Bellizzi is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (8 papers), Alzheimer's disease research and treatments (3 papers) and Multiple Sclerosis Research Studies (3 papers). Matthew J. Bellizzi collaborates with scholars based in United States. Matthew J. Bellizzi's co-authors include Peter G. Weyand, Seth Wright, Harris A. Gelbard, Seth W. Perry, Stephen Dewhurst, Matthew W. Bundle, Jennetta W. Hammond, Ricardo Martinez-Ruiz, Shao‐Ming Lu and Wen Qiu and has published in prestigious journals such as Journal of Clinical Investigation, Journal of Neuroscience and Neurology.

In The Last Decade

Matthew J. Bellizzi

12 papers receiving 1.1k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Faster top running speeds are achieved with greater ground forces not more rapid leg movements

2000 722 citations Peter G. Weyand, Matthew J. Bellizzi et al. Journal of Applied Physiology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew J. Bellizzi United States	11	634	526	175	123	85	13	1.2k
Michael J. Turner United States	22	414 0.7×	156 0.3×	37 0.2×	95 0.8×	344 4.0×	51	1.6k
Grant A. Robinson United States	23	154 0.2×	512 1.0×	128 0.7×	57 0.5×	13 0.2×	35	1.5k
Régis Bonnefoy France	16	525 0.8×	353 0.7×	19 0.1×	257 2.1×	119 1.4×	20	1.3k
Brian L. O’Connor United States	31	531 0.8×	268 0.5×	46 0.3×	16 0.1×	50 0.6×	52	2.5k
Tsang‐Hai Huang Taiwan	18	279 0.4×	202 0.4×	23 0.1×	31 0.3×	102 1.2×	27	1.1k
Wei Yao China	19	82 0.1×	74 0.1×	46 0.3×	386 3.1×	31 0.4×	69	969
D Maurel France	18	390 0.6×	89 0.2×	16 0.1×	30 0.2×	107 1.3×	54	1.1k
Richard C. Carlsen United States	22	101 0.2×	167 0.3×	48 0.3×	76 0.6×	26 0.3×	60	1.4k
Gavin J. Pinniger Australia	18	260 0.4×	542 1.0×	97 0.6×	18 0.1×	21 0.2×	49	1.1k
Fuminori Kawano Japan	21	103 0.2×	252 0.5×	175 1.0×	21 0.2×	114 1.3×	89	1.6k

Countries citing papers authored by Matthew J. Bellizzi

Since Specialization

Citations

This map shows the geographic impact of Matthew J. Bellizzi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Bellizzi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Bellizzi more than expected).

Fields of papers citing papers by Matthew J. Bellizzi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Bellizzi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Bellizzi. The network helps show where Matthew J. Bellizzi may publish in the future.

Co-authorship network of co-authors of Matthew J. Bellizzi

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Bellizzi. A scholar is included among the top collaborators of Matthew J. Bellizzi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Bellizzi. Matthew J. Bellizzi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Powell, Stephen Joseph, et al.. (2025). Posterior Reversible Encephalopathy Syndrome as a Presenting Finding of Multiple Endocrine Neoplasia type 2A Syndrome: A Case Report (P2-13.002). Neurology. 104(7_Supplement_1).

Hammond, Jennetta W., et al.. (2020). Complement-dependent synapse loss and microgliosis in a mouse model of multiple sclerosis. Brain Behavior and Immunity. 87. 739–750. 83 indexed citations

Bellizzi, Matthew J., et al.. (2018). The Mixed-Lineage Kinase Inhibitor URMC-099 Protects Hippocampal Synapses in Experimental Autoimmune Encephalomyelitis. eNeuro. 5(6). ENEURO.0245–18.2018. 15 indexed citations

Hammond, Jennetta W., Wen Qiu, Daniel F. Marker, et al.. (2018). HIV Tat causes synapse loss in a mouse model of HIV‐associated neurocognitive disorder that is independent of the classical complement cascade component C1q. Glia. 66(12). 2563–2574. 17 indexed citations

Bellizzi, Matthew J., et al.. (2016). Platelet-Activating Factor Receptors Mediate Excitatory Postsynaptic Hippocampal Injury in Experimental Autoimmune Encephalomyelitis. Journal of Neuroscience. 36(4). 1336–1346. 35 indexed citations

Bellizzi, Matthew J. & Harris A. Gelbard. (2013). Activated Microglia and Neuronal Excitotoxicity in EAE Gray Matter (P05.170). Neurology. 80(7_supplement). 1 indexed citations

Bellizzi, Matthew J., Shao‐Ming Lu, & Harris A. Gelbard. (2006). Protecting the Synapse: Evidence for a Rational Strategy to Treat HIV-1 Associated Neurologic Disease. Journal of Neuroimmune Pharmacology. 1(1). 20–31. 31 indexed citations

Bundle, Matthew W., Carrie L. Ernst, Matthew J. Bellizzi, Seth Wright, & Peter G. Weyand. (2006). A metabolic basis for impaired muscle force production and neuromuscular compensation during sprint cycling. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 291(5). R1457–R1464. 30 indexed citations

Bellizzi, Matthew J.. (2005). Synaptic activity becomes excitotoxic in neurons exposed to elevated levels of platelet-activating factor. Journal of Clinical Investigation. 115(11). 3185–3192. 60 indexed citations

10.

Perry, Seth W., Stephen Dewhurst, Matthew J. Bellizzi, & Harris A. Gelbard. (2002). Tumor Necrosis Factor-Alpha in Normal and Diseased Brain: Conflicting Effects Via Intraneuronal Receptor Crosstalk?. Journal of NeuroVirology. 8(6). 611–624. 89 indexed citations

11.

Weyand, Peter G., et al.. (2000). Faster top running speeds are achieved with greater ground forces not more rapid leg movements. Journal of Applied Physiology. 89(5). 1991–1999. 722 indexed citations breakdown →

12.

Weyand, Peter G., et al.. (1999). High-speed running performance is largely unaffected by hypoxic reductions in aerobic power. Journal of Applied Physiology. 86(6). 2059–2064. 70 indexed citations

13.

Bellizzi, Matthew J., et al.. (1998). Does the application of ground force set the energetic cost of cross-country skiing?. Journal of Applied Physiology. 85(5). 1736–1743. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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