Matthew Grimmer

3.3k total citations
17 papers, 790 citations indexed

About

Matthew Grimmer is a scholar working on Molecular Biology, Genetics and Cancer Research. According to data from OpenAlex, Matthew Grimmer has authored 17 papers receiving a total of 790 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 7 papers in Genetics and 5 papers in Cancer Research. Recurrent topics in Matthew Grimmer's work include Glioma Diagnosis and Treatment (7 papers), Epigenetics and DNA Methylation (4 papers) and Neuroblastoma Research and Treatments (4 papers). Matthew Grimmer is often cited by papers focused on Glioma Diagnosis and Treatment (7 papers), Epigenetics and DNA Methylation (4 papers) and Neuroblastoma Research and Treatments (4 papers). Matthew Grimmer collaborates with scholars based in United States, United Kingdom and Canada. Matthew Grimmer's co-authors include William A. Weiss, J Costello, Susan M. Chang, Louis Chesler, Yu Yao, Michael Wahl, Serah Choi, Peggy Farnham, Anders I. Persson and Christopher S. Hackett and has published in prestigious journals such as Nucleic Acids Research, Journal of Clinical Oncology and Cancer Cell.

In The Last Decade

Matthew Grimmer

17 papers receiving 781 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthew Grimmer United States 12 514 255 244 216 139 17 790
Nancy E. Hasselt Netherlands 7 761 1.5× 293 1.1× 352 1.4× 298 1.4× 161 1.2× 7 1.0k
Markus Bredel United States 15 565 1.1× 199 0.8× 276 1.1× 192 0.9× 251 1.8× 28 914
Cecilia Dyberg Sweden 12 609 1.2× 272 1.1× 110 0.5× 362 1.7× 168 1.2× 20 878
Danny A. Zwijnenburg Netherlands 17 641 1.2× 303 1.2× 99 0.4× 337 1.6× 234 1.7× 23 965
Amanda Tivnan Ireland 15 875 1.7× 444 1.7× 118 0.5× 218 1.0× 187 1.3× 17 1.2k
Kah Suan Lim United States 10 538 1.0× 173 0.7× 177 0.7× 85 0.4× 157 1.1× 14 754
Carlos Clara Brazil 14 348 0.7× 181 0.7× 274 1.1× 95 0.4× 156 1.1× 38 619
Sujatmi Hariono United States 8 438 0.9× 189 0.7× 192 0.8× 80 0.4× 123 0.9× 9 658
Thale Kristin Olsen Sweden 11 335 0.7× 169 0.7× 134 0.5× 134 0.6× 144 1.0× 24 603
I‐Mei Siu United States 16 494 1.0× 190 0.7× 292 1.2× 66 0.3× 160 1.2× 22 823

Countries citing papers authored by Matthew Grimmer

Since Specialization
Citations

This map shows the geographic impact of Matthew Grimmer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew Grimmer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew Grimmer more than expected).

Fields of papers citing papers by Matthew Grimmer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew Grimmer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew Grimmer. The network helps show where Matthew Grimmer may publish in the future.

Co-authorship network of co-authors of Matthew Grimmer

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew Grimmer. A scholar is included among the top collaborators of Matthew Grimmer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew Grimmer. Matthew Grimmer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Rodvold, Jeffrey J., Matthew Grimmer, Scot A. Marsters, et al.. (2024). ATF6 Promotes Colorectal Cancer Growth and Stemness by Regulating the Wnt Pathway. Cancer Research Communications. 4(10). 2734–2755. 7 indexed citations
2.
Mendoza, Alex de, Trung Viet Nguyen, Ethan Ford, et al.. (2022). Large-scale manipulation of promoter DNA methylation reveals context-specific transcriptional responses and stability. Genome biology. 23(1). 163–163. 64 indexed citations
3.
Mathur, Radhika, Yalan Zhang, Matthew Grimmer, et al.. (2020). MGMT promoter methylation level in newly diagnosed low-grade glioma is a predictor of hypermutation at recurrence. Neuro-Oncology. 22(11). 1580–1590. 50 indexed citations
4.
Jones, Lindsey, Stephanie Hilz, Matthew Grimmer, et al.. (2020). Patient-derived cells from recurrent tumors that model the evolution of IDH-mutant glioma. Neuro-Oncology Advances. 2(1). vdaa088–vdaa088. 16 indexed citations
5.
Bush, Nancy Ann Oberheim, Yu Yao, Javier Villanueva‐Meyer, et al.. (2020). Temozolomide-induced hypermutation is associated with high-grade transformation, distant recurrence, and reduced survival after transformation in initially low-grade IDH-mutant diffuse gliomas.. Journal of Clinical Oncology. 38(15_suppl). 2506–2506. 3 indexed citations
6.
Choi, Serah, Yu Yao, Matthew Grimmer, et al.. (2018). Temozolomide-associated hypermutation in gliomas. Neuro-Oncology. 20(10). 1300–1309. 134 indexed citations
7.
Tak, Yu Gyoung, Lijing Yao, Matthew Grimmer, et al.. (2016). Effects on the transcriptome upon deletion of a distal element cannot be predicted by the size of the H3K27Ac peak in human cells. Nucleic Acids Research. 44(9). 4123–4133. 27 indexed citations
8.
Cage, Tene A., Yvan H. Chanthery, Louis Chesler, et al.. (2015). Downregulation of MYCN through PI3K Inhibition in Mouse Models of Pediatric Neural Cancer. Frontiers in Oncology. 5. 111–111. 20 indexed citations
9.
Mazor, Tali, Brett Johnson, Matthew Grimmer, et al.. (2015). GENO-25HYPERMUTATION AND MALIGNANT PROGRESSION IN AN EXPANDED COHORT OF TEMOZOLOMIDE-TREATED LOW-GRADE GLIOMA PATIENTS. Neuro-Oncology. 17(suppl 5). v97.1–v97. 2 indexed citations
10.
Grimmer, Matthew, Sabine Stolzenburg, Ethan Ford, et al.. (2014). Analysis of an artificial zinc finger epigenetic modulator: widespread binding but limited regulation. Nucleic Acids Research. 42(16). 10856–10868. 56 indexed citations
11.
Grimmer, Matthew & Peggy Farnham. (2014). Can Genome Engineering be used to Target Cancer-Associated Enhancers?. Epigenomics. 6(5). 493–501. 7 indexed citations
12.
Swartling, Fredrik J., Anders I. Persson, Justin Chen, et al.. (2012). Distinct Neural Stem Cell Populations Give Rise to Disparate Brain Tumors in Response to N-MYC. Cancer Cell. 21(5). 601–613. 156 indexed citations
13.
Chanthery, Yvan H., W. Clay Gustafson, Melissa Itsara, et al.. (2012). Paracrine Signaling Through MYCN Enhances Tumor-Vascular Interactions in Neuroblastoma. Science Translational Medicine. 4(115). 115ra3–115ra3. 63 indexed citations
14.
Chesler, Louis, David Goldenberg, Rodney Collins, et al.. (2008). Chemotherapy-Induced Apoptosis in a Transgenic Model of Neuroblastoma Proceeds Through p53 Induction. Neoplasia. 10(11). 1268–IN34. 51 indexed citations
15.
Chesler, Louis, David Goldenberg, Ronit Satchi‐Fainaro, et al.. (2007). Malignant Progression and Blockade of Angiogenesis in a Murine Transgenic Model of Neuroblastoma. Cancer Research. 67(19). 9435–9442. 54 indexed citations
16.
Grimmer, Matthew & William A. Weiss. (2006). Childhood tumors of the nervous system as disorders of normal development. Current Opinion in Pediatrics. 18(6). 634–638. 79 indexed citations
17.
Walley, T, et al.. (1990). Adverse effects of captopril in hospital outpatients with hypertension. Postgraduate Medical Journal. 66(772). 106–109. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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