Mathilde Savall

405 citations
5 papers · 261 indexed · h-index 5
    • Cancer, Hypoxia, and Metabolism 2
    • Cancer, Lipids, and Metabolism 1
    • RNA modifications and cancer 2
    • Cancer-related gene regulation 2
    • Wnt/β-catenin signaling in development and cancer 1
    • Genomics, phytochemicals, and oxidative stress 1
    • Glutathione Transferases and Polymorphisms 1
    • Cancer-related Molecular Pathways 1

Mathilde Savall

5 papers receiving 261 citations

Peers

Mathilde Savall
Comparison fields: 5 of 42
  • Cancer Research 126
  • Hepatology 30
  • Molecular Biology 178
  • Biochemistry 14
  • Oncology 40
Replace Nadia Senni with:
Nadia Senni France
Zhengming Deng China
Xiaoguang Liu China
Juan L López-Cánovas Spain
Jiaqi Sheng China
Mujian Teng China
Hyunsoo Rho United States
Huike Jiao China
Jiaoyuan Jia China
Haofeng Lu China
Mathilde Savall relative to Nadia Senni France Nadia Senni's profile →
Citations per field
00.5×1.5×
Nadia Senni · 1×
Citations per year

Countries citing papers authored by Mathilde Savall

Since Specialization
Citations

This map shows the geographic impact of Mathilde Savall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathilde Savall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathilde Savall more than expected).

Fields of papers citing papers by Mathilde Savall

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathilde Savall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathilde Savall. The network helps show where Mathilde Savall may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mathilde Savall, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mathilde Savall Line = papers co-authored together Mathilde Savall links everyone, so they are left out of the graph.

All Works

5 of 5 papers shown
#Work
1 202111
2 201845
3 201882
4 2018108
5 201815

About Mathilde Savall

Mathilde Savall is a scholar working on Cancer Research, Molecular Biology and Cell Biology, having authored 5 papers that have together received 261 indexed citations. Recurring topics across this work include RNA modifications and cancer (2 papers), Cancer-related gene regulation (2 papers), Cancer, Hypoxia, and Metabolism (2 papers), Cancer, Lipids, and Metabolism (1 paper), Cancer-related Molecular Pathways (1 paper), Wnt/β-catenin signaling in development and cancer (1 paper), Genomics, phytochemicals, and oxidative stress (1 paper) and Glutathione Transferases and Polymorphisms (1 paper). The work is most often cited by research in Cancer Research (126 citations), Hepatology (30 citations) and Molecular Biology (178 citations). Mathilde Savall has collaborated with scholars based in France, Canada and United States. Frequent co-authors include Christine Perret, Benoît Terris, Nadia Senni, Pascale Bossard, Hélène Gilgenkrantz, Chiara Sartor, Angélique Gougèlet, Sabine Colnot, Marie‐Clotilde Alves‐Guerra and Pierre‐Alexandre Just. Their work appears in journals such as Cancer Research, Gut and Journal of Hepatology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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