Mathew P. Leese
- Molecular Biology top 10%
- Organic Chemistry top 2%
- Genetics top 5%
- Oncology top 10%
- Cancer Research top 10%
- Co-authors
- Michael ReedBarry V. L. PotterAtul PurohitB. V. L. POTTERA. PurohitSimon P. NewmanStephen T. NewmanPaul Foster
- Topics
- Estrogen and related hormone effects (25 papers)Synthesis and biological activity (12 papers)Bioactive Compounds and Antitumor Agents (8 papers)
- Cited by
- ToxicologyOrganic ChemistryGenetics
- Partner nations
- United KingdomUnited StatesFrance
In The Last Decade
Mathew P. Leese
53 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 92
- Molecular Biology 994
- Organic Chemistry 898
- Genetics 612
- Oncology 280
- Cancer Research 209
Countries citing papers authored by Mathew P. Leese
This map shows the geographic impact of Mathew P. Leese's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathew P. Leese with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathew P. Leese more than expected).
Fields of papers citing papers by Mathew P. Leese
This network shows the impact of papers produced by Mathew P. Leese. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathew P. Leese. The network helps show where Mathew P. Leese may publish in the future.
Co-authorship network of co-authors of Mathew P. Leese
This figure shows the co-authorship network connecting the top 25 collaborators of Mathew P. Leese. A scholar is included among the top collaborators of Mathew P. Leese based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mathew P. Leese. Mathew P. Leese is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 15 | |
| 3 | 16 | |
| 4 | 25 | |
| 5 | 37 | |
| 6 | 18 | |
| 7 | 6 | |
| 8 | 38 | |
| 9 | 40 | |
| 10 | 30 | |
| 11 | Sulphamoylated derivatives of 2-methoxyestradiol induce apoptosis in breast, ovarian and prostate cancer cell lines through mitotic arrest via the intrinsic apoptotic pathway | 1 |
| 12 | 40 | |
| 13 | 29 | |
| 14 | 27 | |
| 15 | 61 | |
| 16 | 32 | |
| 17 | 24 | |
| 18 | 113 | |
| 19 | 56 | |
| 20 | 30 |
About Mathew P. Leese
Mathew P. Leese is a scholar working on Toxicology, Organic Chemistry and Genetics, having authored 53 papers that have together received 1.8k indexed citations. Recurring topics across this work include Estrogen and related hormone effects (25 papers), Synthesis and biological activity (12 papers) and Bioactive Compounds and Antitumor Agents (8 papers). The work is most often cited by research in Toxicology (138 citations), Organic Chemistry (898 citations) and Genetics (612 citations). Mathew P. Leese has collaborated with scholars based in United Kingdom, United States and France. Frequent co-authors include Michael Reed, Barry V. L. Potter, Atul Purohit, B. V. L. POTTER, A. Purohit, Simon P. Newman, Stephen T. Newman, Paul Foster, Chloe Stengel and Eric Ferrandis. Their work appears in journals such as Biochemistry, Cancer Research and Chemical Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.