Matem Tunçdemi̇r

1.0k total citations
45 papers, 750 citations indexed

About

Matem Tunçdemi̇r is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Nephrology. According to data from OpenAlex, Matem Tunçdemi̇r has authored 45 papers receiving a total of 750 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 9 papers in Pathology and Forensic Medicine and 7 papers in Nephrology. Recurrent topics in Matem Tunçdemi̇r's work include Chronic Kidney Disease and Diabetes (7 papers), Heavy Metal Exposure and Toxicity (6 papers) and Wound Healing and Treatments (4 papers). Matem Tunçdemi̇r is often cited by papers focused on Chronic Kidney Disease and Diabetes (7 papers), Heavy Metal Exposure and Toxicity (6 papers) and Wound Healing and Treatments (4 papers). Matem Tunçdemi̇r collaborates with scholars based in Türkiye, Mexico and Kazakhstan. Matem Tunçdemi̇r's co-authors include Melek Öztürk, Derviş Özçelik, Gönül Kanıgür Sultuybek, Fatma Kaya Dağıstanlı, Hafize Uzun, Mustafa Nazıroğlu, Manuel Flores-Arce, Ömer Çelik, Orhun Sinanoğlu and Güler Öztürk and has published in prestigious journals such as SHILAP Revista de lepidopterología, Archives of Biochemistry and Biophysics and Gene.

In The Last Decade

Matem Tunçdemi̇r

44 papers receiving 732 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matem Tunçdemi̇r Türkiye 16 179 127 108 95 79 45 750
Melek Öztürk Türkiye 16 218 1.2× 130 1.0× 187 1.7× 125 1.3× 56 0.7× 49 816
Osman Nuri Keleş Türkiye 19 180 1.0× 95 0.7× 136 1.3× 56 0.6× 65 0.8× 48 1.0k
Narayana Kilarkaje Kuwait 16 185 1.0× 82 0.6× 97 0.9× 89 0.9× 91 1.2× 45 773
Mariagrazia Rinaldi Italy 13 453 2.5× 90 0.7× 85 0.8× 53 0.6× 138 1.7× 16 1.0k
Arianna Romani Italy 17 285 1.6× 56 0.4× 64 0.6× 48 0.5× 47 0.6× 39 882
Vincenzo Trichilo Italy 13 438 2.4× 109 0.9× 86 0.8× 40 0.4× 170 2.2× 15 962
Freddy Romero United States 17 272 1.5× 67 0.5× 70 0.6× 30 0.3× 43 0.5× 36 947
Xinqing Deng China 12 378 2.1× 197 1.6× 144 1.3× 30 0.3× 25 0.3× 19 999
Nashwa Fathy Gamal El‐Tahawy Egypt 14 136 0.8× 44 0.3× 70 0.6× 76 0.8× 19 0.2× 52 571
Sara Johansson Sweden 9 368 2.1× 111 0.9× 89 0.8× 73 0.8× 34 0.4× 12 778

Countries citing papers authored by Matem Tunçdemi̇r

Since Specialization
Citations

This map shows the geographic impact of Matem Tunçdemi̇r's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matem Tunçdemi̇r with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matem Tunçdemi̇r more than expected).

Fields of papers citing papers by Matem Tunçdemi̇r

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matem Tunçdemi̇r. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matem Tunçdemi̇r. The network helps show where Matem Tunçdemi̇r may publish in the future.

Co-authorship network of co-authors of Matem Tunçdemi̇r

This figure shows the co-authorship network connecting the top 25 collaborators of Matem Tunçdemi̇r. A scholar is included among the top collaborators of Matem Tunçdemi̇r based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matem Tunçdemi̇r. Matem Tunçdemi̇r is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yenmiş, Güven, et al.. (2024). Metformin represses the carcinogenesis potentially induced by 50 Hz magnetic fields in aged mouse fibroblasts via inhibition of NF‐kB. Journal of Cellular and Molecular Medicine. 28(19). e70132–e70132. 2 indexed citations
2.
Yenmiş, Güven, Cihan Taştan, Kazım Şenol, et al.. (2021). Anti-cancer effect of metformin on the metastasis and invasion of primary breast cancer cells through mediating NF-kB activity. Acta Histochemica. 123(4). 151709–151709. 27 indexed citations
3.
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Abalı, Remzi, et al.. (2019). The apoptotic effects of bisphenol A exposure on the rat ovary: an experimental study. Environmental Science and Pollution Research. 26(10). 10198–10203. 15 indexed citations
6.
Yenmiş, Güven, et al.. (2019). Metformin suppresses the proliferation and invasion through NF-kB and MMPs in MCF-7 cell line. Turkish Journal of Biochemistry. 45(3). 295–304. 12 indexed citations
7.
Tunçdemi̇r, Matem, et al.. (2019). Protective Effects of Zinc on 2.45 GHz Electromagnetic Radiation-Induced Oxidative Stress and Apoptosis in HEK293 Cells. Biological Trace Element Research. 194(2). 368–378. 9 indexed citations
8.
Söğüt, İbrahim, et al.. (2019). Dietary curcumin and capsaicin: Relationship with hepaticoxidative stress and apoptosis in rats fed a high fat diet. Advances in Clinical and Experimental Medicine. 28(8). 1013–1020. 14 indexed citations
9.
Gelışgen, Remise, et al.. (2018). Effects of Quercetin on Lipid and Protein Damage in the Liver of Streptozotocin-Induced Experimental Diabetic Rats. Journal of Medicinal Food. 22(1). 52–56. 14 indexed citations
10.
Tunçdemi̇r, Matem, et al.. (2018). Covid-19 ve Kanser Covid-19 and Cance. 4(1). 1–14.
11.
Tunçdemi̇r, Matem, et al.. (2018). Selenium supplementation ameliorates electromagnetic field-induced oxidative stress in the HEK293 cells. Journal of Trace Elements in Medicine and Biology. 50. 572–579. 14 indexed citations
12.
Güven, Mehmet, et al.. (2017). SIRT6 expression and oxidative DNA damage in individuals with prediabetes and type 2 diabetes mellitus. Gene. 642. 542–548. 11 indexed citations
13.
Öztürk, Melek, et al.. (2016). The Effects of Colchicum baytopiorum on Regulatory Genes of Apoptotic and Autophagic Cell Death in HeLa Cells. Current Pharmaceutical Biotechnology. 17(15). 1369–1376. 9 indexed citations
14.
Tunçdemi̇r, Matem & Melek Öztürk. (2016). Regulation of the Ku70 and apoptosis-related proteins in experimental diabetic nephropathy. Metabolism. 65(10). 1466–1477. 13 indexed citations
15.
Öztürk, Güler, et al.. (2013). Protective Effect of Melatonin and 1,25-Dihydroxyvitamin D3 on Renal Ischemia–Reperfusion Injury in Rats. Renal Failure. 35(3). 374–379. 40 indexed citations
16.
Erkanlı, Korhan, Gözde Erkanlı Şentürk, Ünal Aydın, et al.. (2013). Oxytocin Protects Rat Skeletal Muscle Against Ischemia/Reperfusion Injury. Annals of Vascular Surgery. 27(5). 662–670. 22 indexed citations
17.
Sinanoğlu, Orhun, et al.. (2012). Melatonin with 1,25-Dihydroxyvitamin D3 Protects against Apoptotic Ischemia–Reperfusion Injury in the Rat Kidney. Renal Failure. 34(8). 1021–1026. 19 indexed citations
18.
Tunçdemi̇r, Matem, et al.. (2010). Antiapoptotic effect of angiotensin-II type-1 receptor blockade in renal tubular cells of hyperoxaluric rats. Urological Research. 38(2). 71–80. 13 indexed citations
19.
Öztürk, Melek, et al.. (2006). The effects of 5-aminoimidazole-4-carboxamide riboside on the pancreas in neonatal streptozotocin-diabetic rats. Acta Diabetologica. 43(3). 61–65. 3 indexed citations
20.
Öztürk, Melek, et al.. (2004). Effects of ACE inhibition on the expression of type IV collagen and laminin in renal glomeruli in experimental diabetes. Acta Histochemica. 106(4). 279–287. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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