Masahiro Matsuki

1.2k total citations · 1 hit paper
14 papers, 839 citations indexed

About

Masahiro Matsuki is a scholar working on Pulmonary and Respiratory Medicine, Cancer Research and Molecular Biology. According to data from OpenAlex, Masahiro Matsuki has authored 14 papers receiving a total of 839 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Pulmonary and Respiratory Medicine, 6 papers in Cancer Research and 4 papers in Molecular Biology. Recurrent topics in Masahiro Matsuki's work include Cancer, Hypoxia, and Metabolism (6 papers), Renal cell carcinoma treatment (5 papers) and Genetics, Aging, and Longevity in Model Organisms (4 papers). Masahiro Matsuki is often cited by papers focused on Cancer, Hypoxia, and Metabolism (6 papers), Renal cell carcinoma treatment (5 papers) and Genetics, Aging, and Longevity in Model Organisms (4 papers). Masahiro Matsuki collaborates with scholars based in Japan, United States and Canada. Masahiro Matsuki's co-authors include Junji Matsui, Yasuhiro Funahashi, Yuichi Iino, Hirofumi Kunitomo, Taisuke Hoshi, Yoichi Ozawa, Yuji Yamamoto, Yukinori Minoshima, Megumi Ikemori‐Kawada and Takayuki Kimura and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and PLoS ONE.

In The Last Decade

Masahiro Matsuki

13 papers receiving 832 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Lenvatinib plus anti-PD-1 antibody combination treatment activates CD8+ T cells through reduction of tumor-associated macrophage and activation of the interferon pathway

2019 341 citations Yu Kato, Kimiyo Tabata et al. PLoS ONE profile →

Peers

Masahiro Matsuki

ONCOL

HEPAT

AGING

PRM

Naoko Kamimura Japan

Azzura Greco Italy

Chris Siebel United States

Jamie Coupar United States

Constantinos Demetriades Germany

Tae Ho Shin United States

Constantinos Chronis United States

Francesco Antonica Italy

Yutaka Yoshida Japan

Shengda Lin United States

Naoko Kamimura Japan

Masahiro Matsuki

198 ×0.7

ONCOL

42 ×0.2

HEPAT

521 ×2.3

41 ×0.3

AGING

51 ×0.3

PRM

Citations per year, relative to Masahiro Matsuki Masahiro Matsuki (= 1×) peers Naoko Kamimura

Countries citing papers authored by Masahiro Matsuki

Since Specialization

Citations

This map shows the geographic impact of Masahiro Matsuki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masahiro Matsuki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masahiro Matsuki more than expected).

Fields of papers citing papers by Masahiro Matsuki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masahiro Matsuki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masahiro Matsuki. The network helps show where Masahiro Matsuki may publish in the future.

Co-authorship network of co-authors of Masahiro Matsuki

This figure shows the co-authorship network connecting the top 25 collaborators of Masahiro Matsuki. A scholar is included among the top collaborators of Masahiro Matsuki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masahiro Matsuki. Masahiro Matsuki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Kato, Yu, Kimiyo Tabata, Takayuki Kimura, et al.. (2019). Lenvatinib plus anti-PD-1 antibody combination treatment activates CD8+ T cells through reduction of tumor-associated macrophage and activation of the interferon pathway. PLoS ONE. 14(2). e0212513–e0212513. 341 indexed citations breakdown →

Adachi, Yusuke, Masahiro Matsuki, Hideki Watanabe, et al.. (2019). Antitumor and Antiangiogenic Activities of Lenvatinib in Mouse Xenograft Models of Vascular Endothelial Growth Factor-Induced Hypervascular Human Hepatocellular Carcinoma. Cancer Investigation. 37(4-5). 185–198. 14 indexed citations

Matsuki, Masahiro, Taisuke Hoshi, Yuji Yamamoto, et al.. (2018). Lenvatinib inhibits angiogenesis and tumor fibroblast growth factor signaling pathways in human hepatocellular carcinoma models. Cancer Medicine. 7(6). 2641–2653. 186 indexed citations

Matsuki, Masahiro, Yusuke Adachi, Yoichi Ozawa, et al.. (2017). Targeting of tumor growth and angiogenesis underlies the enhanced antitumor activity of lenvatinib in combination with everolimus. Cancer Science. 108(4). 763–771. 57 indexed citations

Ichikawa, Kenji, et al.. (2016). Lenvatinib Suppresses Angiogenesis through the Inhibition of both the VEGFR and FGFR Signaling Pathways. 2(1). 19–25. 6 indexed citations

Matsuki, Masahiro, Kiyoshi Okamoto, Zoltán Dezső, et al.. (2016). Abstract 3266: Antitumor activity of a combination of lenvatinib mesilate, ifosfamide, and etoposide against human pediatric osteosarcoma cell lines. Cancer Research. 76(14_Supplement). 3266–3266.

Adachi, Yusuke, Masahiro Matsuki, Atsumi Yamaguchi, et al.. (2016). Abstract 3264: Lenvatinib in combination with everolimus demonstrated enhanced antiangiogenesis and antitumor activity in human RCC xenograft models. Cancer Research. 76(14_Supplement). 3264–3264. 2 indexed citations

Mitsuhashi, Kaoru, Takayuki Kimura, Taisuke Hoshi, et al.. (2016). Abstract 3262: Effects of lenvatinib mesilate in combination with everolimus on VEGF and FGF-driven angiogenesis and tumor growth. Cancer Research. 76(14_Supplement). 3262–3262. 1 indexed citations

Matsuki, Masahiro, et al.. (2014). [Clinical features of six patients with pancreas metastasis from renal cell carcinoma].. PubMed. 60(3). 105–8. 3 indexed citations

10.

Matsuki, Masahiro, et al.. (2013). PATHOLOGICAL COMPLETE RESPONSE TO GEMCITABINE AND CISPLATIN CHEMOTHERAPY FOR ADVANCED UPPER TRACT UROTHELIAL CARCINOMA: A CASE REPORT. The Japanese Journal of Urology. 104(1). 33–37. 6 indexed citations

11.

Yamada, Kôji, Takaaki Hirotsu, Masahiro Matsuki, et al.. (2010). Olfactory Plasticity Is Regulated by Pheromonal Signaling in Caenorhabditis elegans. Science. 329(5999). 1647–1650. 80 indexed citations

12.

Yamada, Koji, Takaaki Hirotsu, Masahiro Matsuki, Hirofumi Kunitomo, & Yuichi Iino. (2009). GPC-1, a G Protein γ-Subunit, Regulates Olfactory Adaptation inCaenorhabditis elegans. Genetics. 181(4). 1347–1357. 19 indexed citations

13.

Ikeda, Daisuke, Yukan Duan, Masahiro Matsuki, et al.. (2008). CASY-1, an ortholog of calsyntenins/alcadeins, is essential for learning in Caenorhabditis elegans. Proceedings of the National Academy of Sciences. 105(13). 5260–5265. 56 indexed citations

14.

Matsuki, Masahiro, Hirofumi Kunitomo, & Yuichi Iino. (2006). G _o α regulates olfactory adaptation by antagonizing G _q α-DAG signaling in Caenorhabditis elegans. Proceedings of the National Academy of Sciences. 103(4). 1112–1117. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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