Masaakira Kano

556 total citations
29 papers, 466 citations indexed

About

Masaakira Kano is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Masaakira Kano has authored 29 papers receiving a total of 466 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 17 papers in Cellular and Molecular Neuroscience and 5 papers in Physiology. Recurrent topics in Masaakira Kano's work include Ion channel regulation and function (15 papers), Neuroscience and Neural Engineering (11 papers) and Neuroscience and Neuropharmacology Research (9 papers). Masaakira Kano is often cited by papers focused on Ion channel regulation and function (15 papers), Neuroscience and Neural Engineering (11 papers) and Neuroscience and Neuropharmacology Research (9 papers). Masaakira Kano collaborates with scholars based in Japan, India and Germany. Masaakira Kano's co-authors include Yutaka Shimada, Norihiro Suzuki, Koichi Ishikawa, Yuji Ishikawa, Nobuo Tamiya, Hisayuki Ojima, Tohru Yoshioka, Yoshihisa Kudo, Hiroshi Takagi and Hiroshi Takagi and has published in prestigious journals such as Science, Brain Research and Biochemical and Biophysical Research Communications.

In The Last Decade

Masaakira Kano

29 papers receiving 422 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Masaakira Kano Japan 15 328 254 55 49 44 29 466
L. Simoncini United States 9 535 1.6× 314 1.2× 17 0.3× 20 0.4× 69 1.6× 11 791
S.J. Hong Taiwan 16 521 1.6× 283 1.1× 60 1.1× 68 1.4× 11 0.3× 37 646
John V. Milligan Canada 13 186 0.6× 250 1.0× 12 0.2× 58 1.2× 20 0.5× 22 533
Alberto L. Politoff United States 11 201 0.6× 175 0.7× 15 0.3× 39 0.8× 26 0.6× 16 396
Joshua T. Wolfe United States 10 452 1.4× 216 0.9× 14 0.3× 82 1.7× 32 0.7× 15 695
Sidney J. Socolar United States 10 497 1.5× 235 0.9× 3 0.1× 42 0.9× 83 1.9× 13 678
Francisco C. Herrera Venezuela 11 272 0.8× 125 0.5× 3 0.1× 81 1.7× 41 0.9× 26 581
Shiko Chichibu Japan 10 109 0.3× 141 0.6× 9 0.2× 25 0.5× 57 1.3× 35 357
Alfred Dorn Germany 13 189 0.6× 155 0.6× 7 0.1× 76 1.6× 19 0.4× 36 582
James O. Jackson United States 9 445 1.4× 212 0.8× 27 0.5× 208 4.2× 10 0.2× 11 597

Countries citing papers authored by Masaakira Kano

Since Specialization
Citations

This map shows the geographic impact of Masaakira Kano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masaakira Kano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masaakira Kano more than expected).

Fields of papers citing papers by Masaakira Kano

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masaakira Kano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masaakira Kano. The network helps show where Masaakira Kano may publish in the future.

Co-authorship network of co-authors of Masaakira Kano

This figure shows the co-authorship network connecting the top 25 collaborators of Masaakira Kano. A scholar is included among the top collaborators of Masaakira Kano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masaakira Kano. Masaakira Kano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kano, Masaakira, et al.. (1994). Ca2+/Calmodulin-Dependent Regulation of the Density of Na+ Channels in Embryonic Chick Skeletal Muscle Cells During Their Development in Culture. Biochemical and Biophysical Research Communications. 203(2). 852–856. 2 indexed citations
2.
Kano, Masaakira, et al.. (1992). Chronic treatment with D600 enhances development of sodium channels in cultured chick skeletal muscle cells. Neuroscience Letters. 138(2). 249–252. 3 indexed citations
3.
Kano, Masaakira, et al.. (1992). Calcium channels in embryonic chick skeletal muscle cells after cultivation with calcium channel blocker. Neuroscience Letters. 144(1-2). 161–164. 4 indexed citations
4.
Suzuki, Norihiro, et al.. (1992). Augmentation of transient low-threshold Ca2+ current induced by GTP-binding protein signal transduction system in GH3 pituitary cells. Biochemical and Biophysical Research Communications. 187(1). 529–536. 1 indexed citations
5.
Kano, Masaakira, et al.. (1991). Pharmacological properties of two types of calcium channel in embryonic chick skeletal muscle cells in culture. Neuroscience Letters. 122(2). 233–236. 5 indexed citations
6.
Kano, Masaakira, et al.. (1990). Pharmacological blockade of two types calcium channel in cultured chick skeletal muscle cells. The Japanese Journal of Physiology. 40. 112. 1 indexed citations
7.
Suzuki, Norihiro, et al.. (1990). Participation of transient‐type Ca2+ channels in the sustained increase of Ca2+ level in GH3 cells. Journal of Cellular Physiology. 144(1). 62–68. 19 indexed citations
8.
Kano, Masaakira, et al.. (1989). Two components of calcium channel current in embryonic chick skeletal muscle cells developing in culture. Developmental Brain Research. 47(1). 101–112. 20 indexed citations
9.
Kano, Masaakira, et al.. (1988). [<sup>3</sup>H]-Nitrendipine Binding in Chick Myotubes Developing in Culture. Pharmacology. 36(5). 298–304. 1 indexed citations
10.
Kano, Masaakira, et al.. (1987). Calcium channel components of action potential in chick skeletal muscle cells developing in culture. Developmental Brain Research. 32(2). 233–240. 13 indexed citations
11.
Kano, Masaakira, et al.. (1984). Brain extract induces the tetrodotoxin-sensitive action potentials in a rat skeletal muscle cell line (L6). Developmental Brain Research. 13(2). 251–256. 3 indexed citations
12.
Kano, Masaakira & Norihiro Suzuki. (1982). Inhibition by α-amanitin of development of tetrodotoxin-sensitive spike induced by brain extract in cultured chick skeletal muscle cells. Developmental Brain Research. 3(4). 674–678. 6 indexed citations
13.
Kano, Masaakira, et al.. (1981). Development and maintenance of tetrodotoxin-sensitive action potential in cultured skeletal muscle cells from dystrophic and normal chickens. Experimental Neurology. 74(2). 408–418. 3 indexed citations
14.
Kano, Masaakira, et al.. (1977). Development of spike potentials in skeletal muscle cells differentiated in vitro from chick embryo. Journal of Cellular Physiology. 90(3). 439–444. 23 indexed citations
15.
Kano, Masaakira. (1975). Development of excitability in embryonic chick skeletal muscle cells. Journal of Cellular Physiology. 86(3). 503–510. 63 indexed citations
16.
Kano, Masaakira & Koichi Ishikawa. (1972). Effect of tetanus toxin on the inhibitory neuromuscular junction of crayfish muscle. Experimental Neurology. 37(3). 550–561. 5 indexed citations
17.
Kano, Masaakira & Yutaka Shimada. (1971). Innervation of skeletal muscle cells differentiatedin vitro from chick embryo. Brain Research. 27(2). 402–405. 23 indexed citations
18.
Shimada, Yutaka & Masaakira Kano. (1971). Formation of Neuromuscular Junctions in Embryonic Cultures. Archivum histologicum japonicum. 33(2). 95–114. 8 indexed citations
19.
Kano, Masaakira, et al.. (1969). Glycine in the Spinal Cord of Cats with Local Tetanus Rigidity. Science. 164(3879). 571–572. 24 indexed citations
20.
Kano, Masaakira, et al.. (1969). GAMMA ACTIVITY OF RIGID CAT CAUSED BY TETANUS TOXIN. The Japanese Journal of Physiology. 19(1). 1–10. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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