Martin R. Sprick

9.3k total citations · 4 hit papers
58 papers, 6.6k citations indexed

About

Martin R. Sprick is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Martin R. Sprick has authored 58 papers receiving a total of 6.6k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Oncology, 29 papers in Molecular Biology and 21 papers in Cancer Research. Recurrent topics in Martin R. Sprick's work include Cell death mechanisms and regulation (19 papers), Cancer Cells and Metastasis (17 papers) and Pancreatic and Hepatic Oncology Research (12 papers). Martin R. Sprick is often cited by papers focused on Cell death mechanisms and regulation (19 papers), Cancer Cells and Metastasis (17 papers) and Pancreatic and Hepatic Oncology Research (12 papers). Martin R. Sprick collaborates with scholars based in Germany, Netherlands and United States. Martin R. Sprick's co-authors include Henning Walczak, Jan Paul Medema, Giorgio Stassi, Louis Vermeulen, Kristel Kemper, Dick J. Richel, Matilde Todaro, Tijana Borovski, Peter H. Krammer and Kate Cameron and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Martin R. Sprick

53 papers receiving 6.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Wnt activity defines colon cancer stem cells and is regulated by the microenvironment

2010 1.4k citations Louis Vermeulen, Felipe de Sousa e Melo et al. Nature Cell Biology profile →
Identification of a population of blood circulating tumor cells from breast cancer patients that initiates metastasis in a xenograft assay

2013 836 citations Irène Baccelli, Andreas Schneeweiß et al. Nature Biotechnology profile →
FADD/MORT1 and Caspase-8 Are Recruited to TRAIL Receptors 1 and 2 and Are Essential for Apoptosis Mediated by TRAIL Receptor 2

2000 678 citations Martin R. Sprick, Eva Rieser et al. profile →
Single-cell cloning of colon cancer stem cells reveals a multi-lineage differentiation capacity

2008 597 citations Louis Vermeulen, Matilde Todaro et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Martin R. Sprick Germany	31	4.0k	3.6k	2.0k	1.3k	536	58	6.6k
Karen Blyth United Kingdom	43	4.7k 1.2×	1.9k 0.5×	2.2k 1.1×	939 0.7×	289 0.5×	134	7.0k
Stefano Indraccolo Italy	47	3.5k 0.9×	2.5k 0.7×	2.2k 1.1×	1.4k 1.1×	280 0.5×	209	7.0k
Pnina Brodt Canada	46	3.2k 0.8×	2.5k 0.7×	2.2k 1.1×	1.1k 0.9×	252 0.5×	115	6.8k
Emanuela Pilozzi Italy	32	3.9k 1.0×	4.9k 1.4×	2.4k 1.2×	613 0.5×	464 0.9×	143	8.4k
Lalita A. Shevde United States	41	4.5k 1.1×	2.5k 0.7×	1.6k 0.8×	1.3k 1.0×	300 0.6×	105	6.9k
Kazuhiro Mizumoto Japan	52	3.5k 0.9×	4.7k 1.3×	2.3k 1.1×	756 0.6×	224 0.4×	221	8.1k
Moorthy P. Ponnusamy United States	43	3.4k 0.9×	2.3k 0.6×	1.0k 0.5×	998 0.8×	304 0.6×	113	5.4k
Wei‐Zhong Wu China	41	3.5k 0.9×	2.1k 0.6×	1.9k 0.9×	1.0k 0.8×	264 0.5×	107	5.7k
Hu‐Liang Jia China	43	4.6k 1.1×	2.0k 0.6×	3.6k 1.8×	892 0.7×	278 0.5×	92	7.5k
Qiongzhu Dong China	49	4.5k 1.1×	1.8k 0.5×	3.6k 1.8×	1.1k 0.8×	291 0.5×	131	7.4k

Countries citing papers authored by Martin R. Sprick

Since Specialization

Citations

This map shows the geographic impact of Martin R. Sprick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin R. Sprick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin R. Sprick more than expected).

Fields of papers citing papers by Martin R. Sprick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin R. Sprick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin R. Sprick. The network helps show where Martin R. Sprick may publish in the future.

Co-authorship network of co-authors of Martin R. Sprick

This figure shows the co-authorship network connecting the top 25 collaborators of Martin R. Sprick. A scholar is included among the top collaborators of Martin R. Sprick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin R. Sprick. Martin R. Sprick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Lewis, Jennifer, et al.. (2025). Validation of a UPLC-MS/MS Method for Quantifying Intracellular Olaparib Levels in Resistant Ovarian Cancer Cells. Pharmaceuticals. 18(12). 1870–1870.

Würth, Roberto, Laura L. Michel, Verena Thewes, et al.. (2025). Biopsy‐derived organoids in personalised early breast cancer care: Challenges of tumour purity and normal cell overgrowth cap their practical utility. International Journal of Cancer. 156(11). 2200–2209.

Bozza, Matthias, Edward W. Green, Elisa Espinet, et al.. (2020). Novel Non-integrating DNA Nano-S/MAR Vectors Restore Gene Function in Isogenic Patient-Derived Pancreatic Tumor Models. Molecular Therapy — Methods & Clinical Development. 17. 957–968. 23 indexed citations

Muckenhuber, Alexander, Anne Berger, Anna Melissa Schlitter, et al.. (2017). Pancreatic Ductal Adenocarcinoma Subtyping Using the Biomarkers Hepatocyte Nuclear Factor-1A and Cytokeratin-81 Correlates with Outcome and Treatment Response. Clinical Cancer Research. 24(2). 351–359. 61 indexed citations

Muzumdar, Mandar D., Kimberly Judith Dorans, Katherine Chung, et al.. (2017). Survival of pancreatic cancer cells lacking KRAS function. Nature Communications. 8(1). 1090–1090. 128 indexed citations

Muzumdar, Mandar D., Panyu Chen, Kimberly Judith Dorans, et al.. (2017). Survival of pancreatic cancer cells lacking KRAS function. Nature. 1 indexed citations

Jabs, Julia, Franziska Maria Zickgraf, Jeongbin Park, et al.. (2017). Screening drug effects in patient‐derived cancer cells links organoid responses to genome alterations. Molecular Systems Biology. 13(11). 156 indexed citations

Lafont, Élodie, Chahrazade Kantari‐Mimoun, Peter Dráber, et al.. (2017). The linear ubiquitin chain assembly complex regulates TRAIL ‐induced gene activation and cell death. The EMBO Journal. 36(9). 1147–1166. 91 indexed citations

Wallwiener, Markus, Andreas D. Hartkopf, Sabine Riethdorf, et al.. (2015). The impact of HER2 phenotype of circulating tumor cells in metastatic breast cancer: a retrospective study in 107 patients. BMC Cancer. 15(1). 403–403. 62 indexed citations

10.

Baccelli, Irène, Andreas Schneeweiß, Sabine Riethdorf, et al.. (2013). Identification of a population of blood circulating tumor cells from breast cancer patients that initiates metastasis in a xenograft assay. Nature Biotechnology. 31(6). 539–544. 836 indexed citations breakdown →

11.

Nason, Jeffrey A., et al.. (2012). Determination of copper speciation in highway stormwater runoff using competitive ligand exchange – Adsorptive cathodic stripping voltammetry. Water Research. 46(17). 5788–5798. 13 indexed citations

12.

Kemper, Kristel, Martin R. Sprick, Alessandro Scopelliti, et al.. (2010). The AC133 Epitope, but not the CD133 Protein, Is Lost upon Cancer Stem Cell Differentiation. Cancer Research. 70(2). 719–729. 288 indexed citations

13.

Vermeulen, Louis, Felipe de Sousa e Melo, Maartje van der Heijden, et al.. (2010). Wnt activity defines colon cancer stem cells and is regulated by the microenvironment. Nature Cell Biology. 12(5). 468–476. 1433 indexed citations breakdown →

14.

Vermeulen, Louis, Matilde Todaro, Martin R. Sprick, et al.. (2008). Single-cell cloning of colon cancer stem cells reveals a multi-lineage differentiation capacity. Proceedings of the National Academy of Sciences. 105(36). 13427–13432. 597 indexed citations breakdown →

15.

Koschny, Ronald, Tom M. Ganten, Jaromir Sykora, et al.. (2007). TRAIL/bortezomib cotreatment is potentially hepatotoxic but induces cancer-specific apoptosis within a therapeutic window. Hepatology. 45(3). 649–658. 102 indexed citations

16.

Sprick, Martin R., Andreas Kerstan, Kirsty McPherson, et al.. (2004). cFLIPL Inhibits Tumor Necrosis Factor-related Apoptosis-inducing Ligand-mediated NF-κB Activation at the Death-inducing Signaling Complex in Human Keratinocytes. Journal of Biological Chemistry. 279(51). 52824–52834. 46 indexed citations

17.

Leverkus, Martin, Martin R. Sprick, Thilo Mengling, et al.. (2003). Proteasome Inhibition Results in TRAIL Sensitization of Primary Keratinocytes by Removing the Resistance-Mediating Block of Effector Caspase Maturation. Molecular and Cellular Biology. 23(9). 3375–3375. 3 indexed citations

18.

Weigand, Markus A., Anne Große-Wilde, Markus Janke, et al.. (2003). TNF-Related Apoptosis-Inducing Ligand Mediates Tumoricidal Activity of Human Monocytes Stimulated by Newcastle Disease Virus. The Journal of Immunology. 170(4). 1814–1821. 93 indexed citations

19.

Sprick, Martin R. & Henning Walczak. (2003). The interplay between the Bcl-2 family and death receptor-mediated apoptosis. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1644(2-3). 125–132. 178 indexed citations

20.

Walczak, Henning & Martin R. Sprick. (2001). Biochemistry and function of the DISC. Trends in Biochemical Sciences. 26(7). 452–453. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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