Martin N. Andersen
- Molecular Biology
- Cardiology and Cardiovascular Medicine top 5%
- Cellular and Molecular Neuroscience
- Surgery
- Cell Biology
- Co-authors
- Hanne B. RasmussenSøren‐Peter OlesenNicole SchmittThomas JespersenJesper Hastrup SvendsenAnnette Buur SteffensenMorten S. OlesenStig Haunsø
- Topics
- Cardiac electrophysiology and arrhythmias (14 papers)Ion channel regulation and function (12 papers)Receptor Mechanisms and Signaling (4 papers)
- Partner nations
- DenmarkUnited StatesAustralia
In The Last Decade
Martin N. Andersen
19 papers receiving 658 citations
Peers
Comparison fields: 5 of 69
- Molecular Biology 476
- Cardiology and Cardiovascular Medicine 357
- Cellular and Molecular Neuroscience 93
- Surgery 44
- Cell Biology 41
Countries citing papers authored by Martin N. Andersen
This map shows the geographic impact of Martin N. Andersen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin N. Andersen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin N. Andersen more than expected).
Fields of papers citing papers by Martin N. Andersen
This network shows the impact of papers produced by Martin N. Andersen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin N. Andersen. The network helps show where Martin N. Andersen may publish in the future.
Co-authorship network of co-authors of Martin N. Andersen
This figure shows the co-authorship network connecting the top 25 collaborators of Martin N. Andersen. A scholar is included among the top collaborators of Martin N. Andersen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin N. Andersen. Martin N. Andersen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 24 | |
| 2 | 3 | |
| 3 | 20 | |
| 4 | 9 | |
| 5 | 26 | |
| 6 | 7 | |
| 7 | 27 | |
| 8 | 59 | |
| 9 | 25 | |
| 10 | 2 | |
| 11 | 35 | |
| 12 | 30 | |
| 13 | 27 | |
| 14 | 11 | |
| 15 | 148 | |
| 16 | 118 | |
| 17 | 37 | |
| 18 | 36 | |
| 19 | 21 |
About Martin N. Andersen
Martin N. Andersen is a scholar working on Cardiology and Cardiovascular Medicine, Hepatology and Physiology, having authored 19 papers that have together received 665 indexed citations. Recurring topics across this work include Cardiac electrophysiology and arrhythmias (14 papers), Ion channel regulation and function (12 papers) and Receptor Mechanisms and Signaling (4 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (357 citations), Molecular Biology (476 citations) and Physiology (27 citations). Martin N. Andersen has collaborated with scholars based in Denmark, United States and Australia. Frequent co-authors include Hanne B. Rasmussen, Søren‐Peter Olesen, Nicole Schmitt, Thomas Jespersen, Jesper Hastrup Svendsen, Annette Buur Steffensen, Morten S. Olesen, Stig Haunsø, Alicia Lundby and Jesper V. Olsen. Their work appears in journals such as Journal of Biological Chemistry, Scientific Reports and European Heart Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.