Martin Kann

2.0k total citations
28 papers, 1.1k citations indexed

About

Martin Kann is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Martin Kann has authored 28 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 9 papers in Cellular and Molecular Neuroscience and 9 papers in Neurology. Recurrent topics in Martin Kann's work include Parkinson's Disease Mechanisms and Treatments (7 papers), Renal Diseases and Glomerulopathies (7 papers) and Neurological disorders and treatments (6 papers). Martin Kann is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (7 papers), Renal Diseases and Glomerulopathies (7 papers) and Neurological disorders and treatments (6 papers). Martin Kann collaborates with scholars based in Germany, United States and Canada. Martin Kann's co-authors include Peter P. Pramstaller, Christine Klein, Thomas Benzing, Bernhard Schermer, P. Vieregge, Laurie J. Ozelius, Katja Hedrich, E. Schwinger, Xandra O. Breakefield and Markus M. Rinschen and has published in prestigious journals such as Development, Neurology and Annals of Neurology.

In The Last Decade

Martin Kann

27 papers receiving 1.1k citations

Peers

Martin Kann
Martin Kann
Citations per year, relative to Martin Kann Martin Kann (= 1×) peers Elide Mantuano

Countries citing papers authored by Martin Kann

Since Specialization
Citations

This map shows the geographic impact of Martin Kann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Kann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Kann more than expected).

Fields of papers citing papers by Martin Kann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Kann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Kann. The network helps show where Martin Kann may publish in the future.

Co-authorship network of co-authors of Martin Kann

This figure shows the co-authorship network connecting the top 25 collaborators of Martin Kann. A scholar is included among the top collaborators of Martin Kann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Kann. Martin Kann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fabretti, Francesca, Paul T. Brinkkoetter, Thomas Benzing, et al.. (2025). Transcriptional Regulators YAP and TAZ Have Distinct Abilities to Compensate for One Another in Podocytes. Journal of the American Society of Nephrology. 36(8). 1520–1534. 1 indexed citations
2.
Wagner, Tristan, Nils Große Hokamp, Katarzyna Bożek, et al.. (2024). First experiences with machine learning predictions of accelerated declining eGFR slope of living kidney donors 3 years after donation. Journal of Nephrology. 37(6). 1631–1642. 1 indexed citations
3.
Chen, He, et al.. (2024). Gene regulatory networks in disease and ageing. Nature Reviews Nephrology. 20(9). 616–633. 11 indexed citations
4.
Grundmann, Franziska, Martin Kann, Felix C. Koehler, et al.. (2024). Five‐year single‐center analysis of cytomegalovirus viremia in kidney transplant recipients and possible implication for novel prophylactic therapy approaches. Transplant Infectious Disease. 26(1). e14233–e14233. 2 indexed citations
5.
Ignarski, Michael, Lucas Kühne, Linus Butt, et al.. (2021). CALINCA—A Novel Pipeline for the Identification of lncRNAs in Podocyte Disease. Cells. 10(3). 692–692. 4 indexed citations
6.
Kann, Martin & Thomas Benzing. (2021). Update 2021: COVID-19 aus Sicht der Nephrologie. DMW - Deutsche Medizinische Wochenschrift. 146(13/14). 915–917.
7.
Cristanziano, Veronica Di, Eva Heger, Elena Knops, et al.. (2021). Combined Therapy with Intravenous Immunoglobulins, Letermovir and (Val-)Ganciclovir in Complicated Courses of CMV-Infection in Transplant Recipients. Microorganisms. 9(8). 1666–1666. 8 indexed citations
9.
Schroeter, Christina B., Sybille Koehler, Martin Kann, et al.. (2018). Protein half‐life determines expression of proteostatic networks in podocyte differentiation. The FASEB Journal. 32(9). 4696–4713. 13 indexed citations
10.
Rinschen, Markus M., Florian Grahammer, Priyanka Kohli, et al.. (2017). YAP-mediated mechanotransduction determines the podocyte’s response to damage. Science Signaling. 10(474). 66 indexed citations
11.
Rinschen, Markus M., Christina B. Schroeter, Sybille Koehler, et al.. (2016). Quantitative deep mapping of the cultured podocyte proteome uncovers shifts in proteostatic mechanisms during differentiation. American Journal of Physiology-Cell Physiology. 311(3). C404–C417. 25 indexed citations
12.
Ising, Christina, Sybille Koehler, Sebastian Brähler, et al.. (2015). Inhibition of insulin/ IGF ‐1 receptor signaling protects from mitochondria‐mediated kidney failure. EMBO Molecular Medicine. 7(3). 275–287. 54 indexed citations
13.
Kann, Martin, Youngsook L. Jung, Maximilian Lenz, et al.. (2015). Genome-Wide Analysis of Wilms’ Tumor 1-Controlled Gene Expression in Podocytes Reveals Key Regulatory Mechanisms. Journal of the American Society of Nephrology. 26(9). 2097–2104. 88 indexed citations
14.
McMahon, Gearoid M., Dipak Datta, Sarah Bruneau, et al.. (2012). Constitutive activation of the mTOR signaling pathway within the normal glomerulus. Biochemical and Biophysical Research Communications. 425(2). 244–249. 8 indexed citations
15.
Kis, Bernhard, Katja Hedrich, Martin Kann, et al.. (2005). Oculogyric dystonic states in early-onset parkinsonism with basal ganglia calcifications. Neurology. 65(5). 761–761. 4 indexed citations
16.
Hedrich, Katja, Beth Wilmot, Karen Marder, et al.. (2004). Distribution, type, and origin of Parkin mutations: Review and case studies. Movement Disorders. 19(10). 1146–1157. 165 indexed citations
17.
Kann, Martin, Helfried Jacobs, Katja Hedrich, et al.. (2002). Role of parkin mutations in 111 community‐based patients with early‐onset parkinsonism. Annals of Neurology. 51(5). 621–625. 110 indexed citations
18.
Hedrich, Katja, Karen Marder, Juliette Harris, et al.. (2002). Evaluation of 50 probands with early-onset Parkinson’s disease for Parkin mutations. Neurology. 58(8). 1239–1246. 118 indexed citations
19.
Kann, Martin, Bernhard Kis, Peter P. Pramstaller, et al.. (2000). Genetik der Dystonien. Der Nervenarzt. 71(6). 431–441. 4 indexed citations
20.
Klein, Christine, Peter P. Pramstaller, Bernhard Kis, et al.. (2000). Parkin deletions in a family with adult-onset, tremor-dominant parkinsonism: Expanding the phenotype. Annals of Neurology. 48(1). 65–71. 178 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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