Martin Christmann

527 total citations
18 papers, 368 citations indexed

About

Martin Christmann is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Martin Christmann has authored 18 papers receiving a total of 368 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 6 papers in Immunology and 3 papers in Oncology. Recurrent topics in Martin Christmann's work include Immune Response and Inflammation (5 papers), Ubiquitin and proteasome pathways (5 papers) and Fungal and yeast genetics research (3 papers). Martin Christmann is often cited by papers focused on Immune Response and Inflammation (5 papers), Ubiquitin and proteasome pathways (5 papers) and Fungal and yeast genetics research (3 papers). Martin Christmann collaborates with scholars based in Germany, Switzerland and Australia. Martin Christmann's co-authors include Gerhard H. Braus, Bernd Kaina, Dorthe Aasland, Maja Tomičić, René Huber, Korbinian Brand, Rolf Bikker, Christian Birr, Viktor Mutt and J. Metz and has published in prestigious journals such as PLoS ONE, PEDIATRICS and International Journal of Molecular Sciences.

In The Last Decade

Martin Christmann

18 papers receiving 355 citations

Peers

Countries citing papers authored by Martin Christmann

Since Specialization

Citations

This map shows the geographic impact of Martin Christmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Christmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Christmann more than expected).

Fields of papers citing papers by Martin Christmann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Christmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Christmann. The network helps show where Martin Christmann may publish in the future.

Co-authorship network of co-authors of Martin Christmann

This figure shows the co-authorship network connecting the top 25 collaborators of Martin Christmann. A scholar is included among the top collaborators of Martin Christmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Christmann. Martin Christmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

#	Work	Indexed citations
1	S100A9 is indispensable for survival of pneumococcal pneumonia in mice 2023 ·PLoS Pathogens ·(unknown), Benjamin Seeliger, Sascha David, (unknown), Regina Maus, (unknown), Martin Christmann, Konstantin Neumann, Korbinian Brand, (unknown), Frank Bühling, James C. Paton, Johannes Roth, Thomas Vogl, Dorothee Viemann, Tobias Welte, Ulrich A. Maus	5
2	Activation of GSK3 Prevents Termination of TNF-Induced Signaling 2021 ·Journal of Inflammation Research ·(unknown), Rolf Bikker, (unknown), (unknown), Martin Christmann, Korbinian Brand, René Huber	4
3	Implementing LOINC – Current Status and Ongoing Work at a Medical University 2019 ·Studies in health technology and informatics ·(unknown), Matthias Gietzelt, (unknown), Martin Christmann, (unknown), H. Laser, (unknown), (unknown), (unknown), (unknown)	4
4	Implementing LOINC: Current Status and Ongoing Work at the Hannover Medical School 2019 ·Studies in health technology and informatics ·(unknown), Matthias Gietzelt, (unknown), Martin Christmann, (unknown), H. Laser, (unknown), (unknown), (unknown), (unknown)	1
5	Proteome and Phosphoproteome Analysis in TNF Long Term-Exposed Primary Human Monocytes 2019 ·International Journal of Molecular Sciences ·(unknown), Rolf Bikker, (unknown), Martin Christmann, Konstantin Neumann, (unknown), (unknown), (unknown), Andreas Pich, René Huber, Korbinian Brand	10
6	TNF phase III signalling in tolerant cells is tightly controlled by A20 and CYLD 2017 ·Cellular Signalling ·Rolf Bikker, Martin Christmann, (unknown), (unknown), Judith Friesenhagen, Oliver Dittrich‐Breiholz, René Huber, Korbinian Brand	16
7	TNF Tolerance in Monocytes and Macrophages: Characteristics and Molecular Mechanisms 2017 ·Journal of Immunology Research ·René Huber, Rolf Bikker, (unknown), Martin Christmann, Korbinian Brand	28
8	The DenA/DEN1 Interacting Phosphatase DipA Controls Septa Positioning and Phosphorylation-Dependent Stability of Cytoplasmatic DenA/DEN1 during Fungal Development 2016 ·PLoS Genetics ·(unknown), (unknown), Martin Christmann, Oliver Valerius, Sina K. Stumpf, (unknown), Gerhard H. Braus	13
9	C/EBPβ-LAP/LAP Expression Is Mediated by RSK/eIF4B-Dependent Signalling and Boosted by Increased Protein Stability in Models of Monocytic Differentiation 2015 ·PLoS ONE* ·René Huber, (unknown), (unknown), Martin Christmann, Judith Friesenhagen, (unknown), Daniel Pietsch, Korbinian Brand	13
10	Integration of the catalytic subunit activates deneddylase activity in vivo as final step in fungal COP9 signalosome assembly 2015 ·Molecular Microbiology ·(unknown), (unknown), (unknown), Martin Christmann, Oliver Draht, (unknown), Oliver Valerius, Gerhard H. Braus	15
11	Eculizumab as First-Line Therapy for Atypical Hemolytic Uremic Syndrome 2014 ·PEDIATRICS ·Martin Christmann, Matthias Hansen, Carsten Bergmann, Dirk Schwabe, (unknown), W. Schneider	19
12	Control of Multicellular Development by the Physically Interacting Deneddylases DEN1/DenA and COP9 Signalosome 2013 ·PLoS Genetics ·Martin Christmann, (unknown), Colin Gordon, Xiaohua Huang, Özgür Bayram, (unknown), Sina K. Stumpf, Wolfgang Dubiel, Gerhard H. Braus	37
13	The COP9 signalosome counteracts the accumulation of cullin SCF ubiquitin E3 RING ligases during fungal development 2012 ·Molecular Microbiology ·Márcia Regina von Zeska Kress, Rebekka Harting, Özgür Bayram, Martin Christmann, Henriette Irmer, Oliver Valerius, (unknown), Gustavo H. Goldman, Gerhard H. Braus	30
14	Recruitment of the inhibitor Cand1 to the cullin substrate adaptor site mediates interaction to the neddylation site 2010 ·Molecular Biology of the Cell ·Kerstin Helmstaedt, (unknown), Martin Christmann, (unknown), (unknown), Rebekka Harting, Stephanie Grond, Silke Busch, Gerhard H. Braus	23
15	Growth hormone supplementation increased latency to tumourigenesis inAtm-deficient mice 2009 ·Growth Factors ·Ralf Schubert, (unknown), (unknown), (unknown), (unknown), (unknown), Martin Christmann, Stefan Zielen	10
16	A role for UV-light-induced c-Fos: stimulation of nucleotide excision repair and protection against sustained JNK activation and apoptosis 2006 ·Carcinogenesis ·Martin Christmann, Maja Tomičić, Dorthe Aasland, Bernd Kaina	46
17	Samenanatomische Untersuchungen an den GattungenPolyalthiaBl. undGreenwayodendron(Engl. et Diels) Verdc. (Annonaceae) 1987 ·Feddes Repertorium ·Martin Christmann	3
18	The auricular myocardiocytes of the heart constitute an endocrine organ characterization of a porcine cardiac peptide hormone, cardiodilatin-126 1984 ·Cell and Tissue Research ·W. G. Forssmann, Christian Birr, Mats Carlquist, Martin Christmann, R. Finke, Andreas Henschen, D. Hock, H. R. Kirchheim, V. A. W. Kreye, F. Lottspeich, J. Metz, Viktor Mutt, M. Reinecke	91

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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