Martin Chada

725 citations
17 papers · 275 · h-index 11

Impact in

Papers in

Martin Chada

16 papers receiving 264 citations

Peers

Martin Chada
Comparison fields: 5 of 70
  • Endocrine and Autonomic Systems 64
  • Reproductive Medicine 32
  • Experimental and Cognitive Psychology 32
  • Pulmonary and Respiratory Medicine 75
  • Cognitive Neuroscience 44
Replace Rina Perlman with:
Rina Perlman Israel
Masahide Takii Japan
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Countries citing papers authored by Martin Chada

Since Specialization
Citations

This map shows the geographic impact of Martin Chada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Chada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Chada more than expected).

Fields of papers citing papers by Martin Chada

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin Chada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Chada. The network helps show where Martin Chada may publish in the future.

Co-authors

The 25 scholars most cited alongside Martin Chada, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Martin Chada Line = papers co-authored together Martin Chada links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 200346
2 201037
3 200736
4 200324
5 200320
6 200220
7
Serum alpha-glutathione S-transferase as a sensitive marker of hepatocellular damage in patients with cystic fibrosis.
200317
8 200415
9 200414
10 200812
11 200312
12
[Amylin--its physiological role in humans].
20029
13 20164
14 20164
15 20093
16 20252
17 20160

About Martin Chada

Martin Chada is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine, Public Health, Environmental and Occupational Health, Cellular and Molecular Neuroscience and Endocrine and Autonomic Systems, having authored 17 papers that have together received 275 indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (3 papers), Pulmonary Hypertension Research and Treatments (3 papers), TGF-β signaling in diseases (2 papers), Glutathione Transferases and Polymorphisms (2 papers), Respiratory Support and Mechanisms (2 papers), Sleep and related disorders (2 papers), Immune Response and Inflammation (2 papers) and Reproductive Biology and Fertility (2 papers). The work is most often cited by research in Endocrine and Autonomic Systems (64 citations), Reproductive Medicine (32 citations), Experimental and Cognitive Psychology (32 citations), Pulmonary and Respiratory Medicine (75 citations) and Cognitive Neuroscience (44 citations). Martin Chada has collaborated with scholars based in Germany, Czechia and United States. Frequent co-authors include Jiří Bronský, Richard Průša, Karel Kotaška, L Lisá, Katharina von der Hardt, Wolfgang Rascher, Michael A. Kandler, J Nevoral, J Nedvı́dková and Ellen Schoof. Their work appears in journals such as Physiological Research, International Journal of Eating Disorders, Prostaglandins & Other Lipid Mediators, Pediatric Pulmonology and Clinical Microbiology and Infection.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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