Martin Bachman
- Molecular Biology top 5%
- Genetics top 10%
- Cancer Research top 10%
- Pediatrics, Perinatology and Child Health top 10%
- Virology top 10%
- Co-authors
- Shankar BalasubramanianAdele MurrellSantiago Uribe‐LewisXiaoping YangWolf ReikMichael J. WilliamsMichael J. BoothMario Iurlaro
- Topics
- Epigenetics and DNA Methylation (15 papers)RNA modifications and cancer (13 papers)Cancer-related gene regulation (7 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of the American Chemical SocietyPLoS ONE
- Partner nations
- United KingdomGermanyBelgium
In The Last Decade
Martin Bachman
22 papers receiving 1.8k citations
Peers
Comparison fields: 5 of 99
- Molecular Biology 1.6k
- Genetics 226
- Cancer Research 193
- Pediatrics, Perinatology and Child Health 106
- Virology 73
Countries citing papers authored by Martin Bachman
This map shows the geographic impact of Martin Bachman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Bachman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Bachman more than expected).
Fields of papers citing papers by Martin Bachman
This network shows the impact of papers produced by Martin Bachman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Bachman. The network helps show where Martin Bachman may publish in the future.
Co-authorship network of co-authors of Martin Bachman
This figure shows the co-authorship network connecting the top 25 collaborators of Martin Bachman. A scholar is included among the top collaborators of Martin Bachman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Bachman. Martin Bachman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 7 | |
| 3 | 3 | |
| 4 | 9 | |
| 5 | 4 | |
| 6 | 38 | |
| 7 | 128 | |
| 8 | 52 | |
| 9 | 43 | |
| 10 | 154 | |
| 11 | 40 | |
| 12 | 196 | |
| 13 | 72 | |
| 14 | 48 | |
| 15 | 60 | |
| 16 | 403 | |
| 17 | 1 | |
| 18 | 173 | |
| 19 | 66 | |
| 20 | 17 |
About Martin Bachman
Martin Bachman is a scholar working on Molecular Biology, Biochemistry and Pediatrics, Perinatology and Child Health, having authored 22 papers that have together received 1.8k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (15 papers), RNA modifications and cancer (13 papers) and Cancer-related gene regulation (7 papers). The work is most often cited by research in Molecular Biology (1.6k citations), Virology (73 citations) and Cancer Research (193 citations). Martin Bachman has collaborated with scholars based in United Kingdom, Germany and Belgium. Frequent co-authors include Shankar Balasubramanian, Adele Murrell, Santiago Uribe‐Lewis, Xiaoping Yang, Wolf Reik, Michael J. Williams, Michael J. Booth, Mario Iurlaro, Dario Beraldi and Gabriella Ficz. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.