Marta Boccazzi

882 total citations
17 papers, 501 citations indexed

About

Marta Boccazzi is a scholar working on Developmental Neuroscience, Molecular Biology and Genetics. According to data from OpenAlex, Marta Boccazzi has authored 17 papers receiving a total of 501 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Developmental Neuroscience, 6 papers in Molecular Biology and 6 papers in Genetics. Recurrent topics in Marta Boccazzi's work include Neurogenesis and neuroplasticity mechanisms (8 papers), Genetics and Neurodevelopmental Disorders (5 papers) and Adenosine and Purinergic Signaling (4 papers). Marta Boccazzi is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (8 papers), Genetics and Neurodevelopmental Disorders (5 papers) and Adenosine and Purinergic Signaling (4 papers). Marta Boccazzi collaborates with scholars based in Italy, France and Australia. Marta Boccazzi's co-authors include Marta Fumagalli, Maria P. Abbracchio, Stefano Raffaele, Stefania Ceruti, Davide Lecca, Davide Marangon, Giulia Magni, Annalisa Buffo, Francesca Viganò and Valeria Vezzoli and has published in prestigious journals such as British Journal of Pharmacology, Biochemical Pharmacology and Glia.

In The Last Decade

Marta Boccazzi

17 papers receiving 498 citations

Peers

Marta Boccazzi

NEURO

CMN

PHYSI

Yosuke M. Morizawa Japan

Yuri Hirayama Japan

Przemyslaw Swiatkowski United States

Katharine J. Liang United States

Paul Hubbard United Kingdom

Xu-Fei Du China

Vilma Spreuer United States

Vitali Belzer Israel

Ming Sum Ruby Chiang United States

Henglin Yan United States

Yosuke M. Morizawa Japan

Marta Boccazzi

167 ×0.9

357 ×2.0

NEURO

89 ×0.7

175 ×2.2

CMN

116 ×1.7

PHYSI

Citations per year, relative to Marta Boccazzi Marta Boccazzi (= 1×) peers Yosuke M. Morizawa

Countries citing papers authored by Marta Boccazzi

Since Specialization

Citations

This map shows the geographic impact of Marta Boccazzi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marta Boccazzi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marta Boccazzi more than expected).

Fields of papers citing papers by Marta Boccazzi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marta Boccazzi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marta Boccazzi. The network helps show where Marta Boccazzi may publish in the future.

Co-authorship network of co-authors of Marta Boccazzi

This figure shows the co-authorship network connecting the top 25 collaborators of Marta Boccazzi. A scholar is included among the top collaborators of Marta Boccazzi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marta Boccazzi. Marta Boccazzi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Pedretti, Silvia, Marta Boccazzi, Lorenzo Da Dalt, et al.. (2025). D-lactate and glycerol as potential biomarkers of sorafenib activity in hepatocellular carcinoma. Signal Transduction and Targeted Therapy. 10(1). 200–200. 2 indexed citations

Raffaele, Stefano, Nhung Nguyen, Marco Milanese, et al.. (2024). Montelukast improves disease outcome in SOD1^G93A female mice by counteracting oligodendrocyte dysfunction and aberrant glial reactivity. British Journal of Pharmacology. 181(18). 3303–3326. 5 indexed citations

Boccazzi, Marta, et al.. (2023). Altered Purinergic Signaling in Neurodevelopmental Disorders: Focus on P2 Receptors. Biomolecules. 13(5). 856–856. 6 indexed citations

Boccazzi, Marta, Giulia Macchiarulo, Sophie Lebon, et al.. (2023). G protein-coupled receptor 17 is regulated by WNT pathway during oligodendrocyte precursor cell differentiation. Neurobiology of Disease. 187. 106315–106315. 9 indexed citations

Fumagalli, Marta, Marta Boccazzi, & Stefano Raffaele. (2022). Not only myelination: the immune-inflammatory functions of oligodendrocytes. Neural Regeneration Research. 17(12). 2661–2661. 26 indexed citations

Passera, Sofia, Marta Boccazzi, Cindy Bokobza, et al.. (2021). Therapeutic potential of stem cells for preterm infant brain damage: Can we move from the heterogeneity of preclinical and clinical studies to established therapeutics?. Biochemical Pharmacology. 186. 114461–114461. 16 indexed citations

Raffaele, Stefano, Marta Boccazzi, & Marta Fumagalli. (2021). Oligodendrocyte Dysfunction in Amyotrophic Lateral Sclerosis: Mechanisms and Therapeutic Perspectives. Cells. 10(3). 565–565. 52 indexed citations

Boccazzi, Marta, Juliette Van Steenwinckel, Anne‐Laure Schang, et al.. (2021). The immune-inflammatory response of oligodendrocytes in a murine model of preterm white matter injury: the role of TLR3 activation. Cell Death and Disease. 12(2). 166–166. 43 indexed citations

Marangon, Davide, Nicolò Caporale, Marta Boccazzi, et al.. (2021). Novel in vitro Experimental Approaches to Study Myelination and Remyelination in the Central Nervous System. Frontiers in Cellular Neuroscience. 15. 22 indexed citations

10.

Marangon, Davide, Marta Boccazzi, Davide Lecca, & Marta Fumagalli. (2020). Regulation of Oligodendrocyte Functions: Targeting Lipid Metabolism and Extracellular Matrix for Myelin Repair. Journal of Clinical Medicine. 9(2). 470–470. 50 indexed citations

11.

Magni, Giulia, Marta Boccazzi, Antonella Bodini, et al.. (2019). Basal astrocyte and microglia activation in the central nervous system of Familial Hemiplegic Migraine Type I mice. Cephalalgia. 39(14). 1809–1817. 26 indexed citations

12.

Boccazzi, Marta, Davide Lecca, Davide Marangon, et al.. (2016). A new role for the P2Y-like GPR17 receptor in the modulation of multipotency of oligodendrocyte precursor cells in vitro. Purinergic Signalling. 12(4). 661–672. 17 indexed citations

13.

Boccazzi, Marta, Maria Passafaro, Davide Lecca, et al.. (2016). SNX27, a protein involved in down syndrome, regulates GPR17 trafficking and oligodendrocyte differentiation. Glia. 64(8). 1437–1460. 18 indexed citations

14.

Boccazzi, Marta, Chiara Rolando, Maria P. Abbracchio, Annalisa Buffo, & Stefania Ceruti. (2013). Purines regulate adult brain subventricular zone cell functions: Contribution of reactive astrocytes. Glia. 62(3). 428–439. 33 indexed citations

15.

Ceruti, Stefania, Giulia Magni, Francesca Viganò, et al.. (2011). Oxygen–glucose deprivation increases the enzymatic activity and the microvesicle-mediated release of ectonucleotidases in the cells composing the blood–brain barrier. Neurochemistry International. 59(2). 259–271. 42 indexed citations

16.

Ceruti, Stefania, Francesca Viganò, Enrica Boda, et al.. (2010). Expression of the new P2Y-like receptor GPR17 during oligodendrocyte precursor cell maturation regulates sensitivity to ATP-induced death. Glia. 59(3). 363–378. 54 indexed citations

17.

Dossena, Silvia, Simona Rodighiero, Valeria Vezzoli, et al.. (2009). Functional characterization of wild-type and mutated pendrin (SLC26A4), the anion transporter involved in Pendred syndrome. Journal of Molecular Endocrinology. 43(3). 93–103. 80 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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