Davide Marangon

461 total citations
18 papers, 341 citations indexed

About

Davide Marangon is a scholar working on Molecular Biology, Developmental Neuroscience and Cancer Research. According to data from OpenAlex, Davide Marangon has authored 18 papers receiving a total of 341 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Developmental Neuroscience and 7 papers in Cancer Research. Recurrent topics in Davide Marangon's work include Neurogenesis and neuroplasticity mechanisms (10 papers), MicroRNA in disease regulation (7 papers) and Extracellular vesicles in disease (4 papers). Davide Marangon is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (10 papers), MicroRNA in disease regulation (7 papers) and Extracellular vesicles in disease (4 papers). Davide Marangon collaborates with scholars based in Italy, Denmark and Spain. Davide Marangon's co-authors include Davide Lecca, Maria P. Abbracchio, Marta Fumagalli, Marta Boccazzi, G. Coppolino, Roberto Furlan, Stefano Raffaele, Camilla Negri, Annamaria Finardi and Vittorio Martinelli and has published in prestigious journals such as PLoS ONE, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Davide Marangon

17 papers receiving 338 citations

Peers

Davide Marangon
Suyog Pol United States
Jiashu Li China
Roey Baror United Kingdom
Terra J. Frederick United States
Suyog Pol United States
Davide Marangon
Citations per year, relative to Davide Marangon Davide Marangon (= 1×) peers Suyog Pol

Countries citing papers authored by Davide Marangon

Since Specialization
Citations

This map shows the geographic impact of Davide Marangon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Davide Marangon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Davide Marangon more than expected).

Fields of papers citing papers by Davide Marangon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Davide Marangon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Davide Marangon. The network helps show where Davide Marangon may publish in the future.

Co-authorship network of co-authors of Davide Marangon

This figure shows the co-authorship network connecting the top 25 collaborators of Davide Marangon. A scholar is included among the top collaborators of Davide Marangon based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Davide Marangon. Davide Marangon is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Raffaele, Stefano, Marta Lombardi, Davide Marangon, et al.. (2025). Microglia-Derived Extracellular Vesicles Enhance Oligodendrocyte Maturation by Transcriptionally Regulating Mitochondrial Molecular Pathways. Cellular and Molecular Neurobiology. 45(1). 89–89.
2.
Raffaele, Stefano, Bettina Hjelm Clausen, Martin Weiss Nielsen, et al.. (2024). Characterisation of GPR17 ‐expressing oligodendrocyte precursors in human ischaemic lesions and correlation with reactive glial responses. The Journal of Pathology. 265(2). 226–243. 1 indexed citations
3.
Marangon, Davide, et al.. (2024). Oligodendrocyte Progenitors in Glial Scar: A Bet on Remyelination. Cells. 13(12). 1024–1024. 8 indexed citations
4.
Marangon, Davide & Davide Lecca. (2023). Exosomal non-coding RNAs in glioma progression: insights into tumor microenvironment dynamics and therapeutic implications. Frontiers in Cell and Developmental Biology. 11. 1275755–1275755. 5 indexed citations
5.
Boccazzi, Marta, Giulia Macchiarulo, Sophie Lebon, et al.. (2023). G protein-coupled receptor 17 is regulated by WNT pathway during oligodendrocyte precursor cell differentiation. Neurobiology of Disease. 187. 106315–106315. 9 indexed citations
6.
Marangon, Davide, et al.. (2022). Neuronal and Glial Communication via Non-Coding RNAs: Messages in Extracellular Vesicles. International Journal of Molecular Sciences. 24(1). 470–470. 9 indexed citations
7.
Marangon, Davide, Matteo Audano, Silvia Pedretti, et al.. (2022). Rewiring of Glucose and Lipid Metabolism Induced by G Protein-Coupled Receptor 17 Silencing Enables the Transition of Oligodendrocyte Progenitors to Myelinating Cells. Cells. 11(15). 2369–2369. 10 indexed citations
8.
Angelini, Jacopo, Davide Marangon, Stefano Raffaele, Davide Lecca, & Maria P. Abbracchio. (2021). The Distribution of GPR17-Expressing Cells Correlates with White Matter Inflammation Status in Brain Tissues of Multiple Sclerosis Patients. International Journal of Molecular Sciences. 22(9). 4574–4574. 19 indexed citations
9.
Marangon, Davide, Nicolò Caporale, Marta Boccazzi, et al.. (2021). Novel in vitro Experimental Approaches to Study Myelination and Remyelination in the Central Nervous System. Frontiers in Cellular Neuroscience. 15. 22 indexed citations
10.
Marangon, Davide, Marta Boccazzi, Davide Lecca, & Marta Fumagalli. (2020). Regulation of Oligodendrocyte Functions: Targeting Lipid Metabolism and Extracellular Matrix for Myelin Repair. Journal of Clinical Medicine. 9(2). 470–470. 50 indexed citations
11.
Paladini, Maria Serena, Davide Marangon, A.C. Rossetti, et al.. (2020). Prenatal Stress Impairs Spinal Cord Oligodendrocyte Maturation via BDNF Signaling in the Experimental Autoimmune Encephalomyelitis Model of Multiple Sclerosis. Cellular and Molecular Neurobiology. 42(4). 1225–1240. 9 indexed citations
12.
Marangon, Davide, Maria P. Abbracchio, & Davide Lecca. (2020). Pathway-Focused Profiling of Oligodendrocytes Over-Expressing miR-125a-3p Reveals Alteration of Wnt and Cell-to-Cell Signaling. Cellular and Molecular Neurobiology. 41(1). 105–114. 10 indexed citations
13.
Marangon, Davide, Enrica Boda, Roberta Parolisi, et al.. (2020). In vivo silencing of miR‐125a‐3p promotes myelin repair in models of white matter demyelination. Glia. 68(10). 2001–2014. 35 indexed citations
14.
Parravicini, Chiara, Davide Lecca, Davide Marangon, et al.. (2020). Development of the first in vivo GPR17 ligand through an iterative drug discovery pipeline: A novel disease-modifying strategy for multiple sclerosis. PLoS ONE. 15(4). e0231483–e0231483. 19 indexed citations
15.
Marangon, Davide, Stefano Raffaele, Marta Fumagalli, & Davide Lecca. (2019). MicroRNAs change the games in central nervous system pharmacology. Biochemical Pharmacology. 168. 162–172. 20 indexed citations
16.
Coppolino, G., Davide Marangon, Camilla Negri, et al.. (2018). Differential local tissue permissiveness influences the final fate of GPR17‐expressing oligodendrocyte precursors in two distinct models of demyelination. Glia. 66(5). 1118–1130. 40 indexed citations
17.
Boccazzi, Marta, Davide Lecca, Davide Marangon, et al.. (2016). A new role for the P2Y-like GPR17 receptor in the modulation of multipotency of oligodendrocyte precursor cells in vitro. Purinergic Signalling. 12(4). 661–672. 17 indexed citations
18.
Lecca, Davide, Davide Marangon, G. Coppolino, et al.. (2016). MiR-125a-3p timely inhibits oligodendroglial maturation and is pathologically up-regulated in human multiple sclerosis. Scientific Reports. 6(1). 34503–34503. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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