Marsha DeSmet

794 total citations
24 papers, 630 citations indexed

About

Marsha DeSmet is a scholar working on Genetics, Molecular Biology and Epidemiology. According to data from OpenAlex, Marsha DeSmet has authored 24 papers receiving a total of 630 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Genetics, 11 papers in Molecular Biology and 10 papers in Epidemiology. Recurrent topics in Marsha DeSmet's work include Cervical Cancer and HPV Research (8 papers), Virus-based gene therapy research (7 papers) and Vitamin D Research Studies (5 papers). Marsha DeSmet is often cited by papers focused on Cervical Cancer and HPV Research (8 papers), Virus-based gene therapy research (7 papers) and Vitamin D Research Studies (5 papers). Marsha DeSmet collaborates with scholars based in United States, China and Philippines. Marsha DeSmet's co-authors include James C. Fleet, Robert L. Johnson, Yan Li, Elliot J. Androphy, Leny Jose, Paul W. Snyder, Sriramana Kanginakudru, Yingben Xue, Keke C. Fairfax and Fang Xie and has published in prestigious journals such as PLoS ONE, Journal of Virology and Biochemical Journal.

In The Last Decade

Marsha DeSmet

23 papers receiving 619 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Marsha DeSmet United States 11 323 185 173 156 149 24 630
Trine B. Levring Denmark 8 308 1.0× 125 0.7× 60 0.3× 63 0.4× 77 0.5× 11 621
P J Malloy United States 13 497 1.5× 239 1.3× 237 1.4× 54 0.3× 65 0.4× 15 832
Aouatef Ismail Chouchane Qatar 8 199 0.6× 107 0.6× 36 0.2× 46 0.3× 48 0.3× 11 478
Carsten Kriebitzsch Belgium 7 148 0.5× 182 1.0× 104 0.6× 66 0.4× 20 0.1× 9 408
Frank G. Hustmyer United States 11 375 1.2× 204 1.1× 149 0.9× 123 0.8× 22 0.1× 14 692
Kazuya Takahama Japan 16 61 0.2× 294 1.6× 214 1.2× 54 0.3× 80 0.5× 38 683
Kristen L. Williams United States 7 30 0.1× 116 0.6× 213 1.2× 96 0.6× 133 0.9× 31 499
John Sterpka United States 7 30 0.1× 254 1.4× 142 0.8× 110 0.7× 40 0.3× 8 551
Ali Murat Ceyhan Türkiye 12 55 0.2× 90 0.5× 28 0.2× 76 0.5× 84 0.6× 32 487
Abdullateef A. Alzolibani Saudi Arabia 21 144 0.4× 159 0.9× 88 0.5× 43 0.3× 131 0.9× 57 976

Countries citing papers authored by Marsha DeSmet

Since Specialization
Citations

This map shows the geographic impact of Marsha DeSmet's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marsha DeSmet with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marsha DeSmet more than expected).

Fields of papers citing papers by Marsha DeSmet

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marsha DeSmet. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marsha DeSmet. The network helps show where Marsha DeSmet may publish in the future.

Co-authorship network of co-authors of Marsha DeSmet

This figure shows the co-authorship network connecting the top 25 collaborators of Marsha DeSmet. A scholar is included among the top collaborators of Marsha DeSmet based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marsha DeSmet. Marsha DeSmet is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
González, Jéssica, Marsha DeSmet, & Elliot J. Androphy. (2025). A Conserved Di-Lysine Motif in the E2 Transactivation Domain Regulates MmuPV1 Replication and Disease Progression. Pathogens. 14(1). 84–84.
2.
DeSmet, Marsha, et al.. (2024). A conserved cysteine in the DNA-binding domain of MmuPV1 E2 is required for replication in vivo. Journal of Virology. 99(1). e0142324–e0142324. 2 indexed citations
3.
DeSmet, Marsha, et al.. (2024). Regulation of R-Loops in DNA Tumor Viruses. Pathogens. 13(10). 863–863. 3 indexed citations
4.
Jose, Leny, et al.. (2024). Senataxin mediates R-loop resolution on HPV episomes. Journal of Virology. 98(8). e0100324–e0100324. 4 indexed citations
5.
Jose, Leny, et al.. (2023). Focal Adhesion Kinase Binds to the HPV E2 Protein to Regulate Initial Replication after Infection. Pathogens. 12(10). 1203–1203. 2 indexed citations
6.
Jose, Leny, et al.. (2021). Regulation of the Human Papillomavirus Lifecyle through Post-Translational Modifications of the Viral E2 Protein. Pathogens. 10(7). 793–793. 8 indexed citations
7.
Jose, Leny, Elliot J. Androphy, & Marsha DeSmet. (2020). Phosphorylation of the Human Papillomavirus E2 Protein at Tyrosine 138 Regulates Episomal Replication. Journal of Virology. 94(14). 10 indexed citations
8.
Xie, Fang, et al.. (2020). Human Papillomavirus 31 Tyrosine 102 Regulates Interaction with E2 Binding Partners and Episomal Maintenance. Journal of Virology. 94(16). 6 indexed citations
9.
DeSmet, Marsha, et al.. (2019). Phosphorylation of a Conserved Tyrosine in the Papillomavirus E2 Protein Regulates Brd4 Binding and Viral Replication. Journal of Virology. 93(10). 15 indexed citations
10.
DeSmet, Marsha, et al.. (2018). Papillomavirus E2 protein is regulated by specific fibroblast growth factor receptors. Virology. 521. 62–68. 9 indexed citations
11.
DeSmet, Marsha & James C. Fleet. (2017). Constitutively active RAS signaling reduces 1,25 dihydroxyvitamin D-mediated gene transcription in intestinal epithelial cells by reducing vitamin D receptor expression. The Journal of Steroid Biochemistry and Molecular Biology. 173. 194–201. 19 indexed citations
12.
Johnson, Robert L., et al.. (2017). Vitamin D Receptor–Dependent Signaling Protects Mice From Dextran Sulfate Sodium-Induced Colitis. Endocrinology. 158(6). 1951–1963. 36 indexed citations
13.
DeSmet, Marsha, et al.. (2016). The Replicative Consequences of Papillomavirus E2 Protein Binding to the Origin Replication Factor ORC2. PLoS Pathogens. 12(10). e1005934–e1005934. 22 indexed citations
14.
DeSmet, Marsha, et al.. (2016). Molecular Probing of the HPV-16 E6 Protein Alpha Helix Binding Groove with Small Molecule Inhibitors. PLoS ONE. 11(2). e0149845–e0149845. 26 indexed citations
15.
Johnson, Robert L., et al.. (2015). An Inducible, Large-Intestine-Specific Transgenic Mouse Model for Colitis and Colitis-Induced Colon Cancer Research. Digestive Diseases and Sciences. 61(4). 1069–1079. 5 indexed citations
16.
Kanginakudru, Sriramana, et al.. (2015). Levels of the E2 interacting protein TopBP1 modulate papillomavirus maintenance stage replication. Virology. 478. 129–136. 24 indexed citations
17.
Fleet, James C., Marsha DeSmet, Robert L. Johnson, & Yan Li. (2011). Vitamin D and cancer: a review of molecular mechanisms. Biochemical Journal. 441(1). 61–76. 323 indexed citations
18.
Xue, Yingben, Robert L. Johnson, Marsha DeSmet, Paul W. Snyder, & James C. Fleet. (2010). Generation of a Transgenic Mouse for Colorectal Cancer Research with Intestinal Cre Expression Limited to the Large Intestine. Molecular Cancer Research. 8(8). 1095–1104. 47 indexed citations
19.
Zhang, Zhentao, et al.. (2010). Constitutive activation of the mitogen‐activated protein kinase pathway impairs vitamin D signaling in human prostate epithelial cells. Journal of Cellular Physiology. 224(2). 433–442. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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