Markus P. Kummer

18.2k total citations · 4 hit papers
52 papers, 8.7k citations indexed

About

Markus P. Kummer is a scholar working on Physiology, Neurology and Molecular Biology. According to data from OpenAlex, Markus P. Kummer has authored 52 papers receiving a total of 8.7k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Physiology, 27 papers in Neurology and 18 papers in Molecular Biology. Recurrent topics in Markus P. Kummer's work include Alzheimer's disease research and treatments (32 papers), Neuroinflammation and Neurodegeneration Mechanisms (27 papers) and Tryptophan and brain disorders (10 papers). Markus P. Kummer is often cited by papers focused on Alzheimer's disease research and treatments (32 papers), Neuroinflammation and Neurodegeneration Mechanisms (27 papers) and Tryptophan and brain disorders (10 papers). Markus P. Kummer collaborates with scholars based in Germany, United States and Spain. Markus P. Kummer's co-authors include Michael T. Heneka, Eicke Latz, Dick Terwel, Angelika Griep, Stephanie Schwartz, Ana Vieira‐Saecker, Ellen Gelpí, Douglas T. Golenbock, Martin Körte and Annett Halle and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Markus P. Kummer

52 papers receiving 8.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

NLRP3 is activated in Alzheimer’s disease and contributes to pathology in APP/PS1 mice

2012 2.2k citations Michael T. Heneka, Markus P. Kummer et al. Nature profile →
Innate immune activation in neurodegenerative disease

2014 1.1k citations Michael T. Heneka, Markus P. Kummer et al. profile →
Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease

2017 708 citations Carmen Venegas, Sathish Kumar et al. Nature profile →
Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease

2021 207 citations Emma Gerrits, Nieske Brouwer et al. Acta Neuropathologica profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Markus P. Kummer Germany	37	3.9k	3.9k	3.4k	1.6k	1.4k	52	8.7k
Robert Veerhuis Netherlands	49	3.5k 0.9×	2.8k 0.7×	2.3k 0.7×	712 0.5×	928 0.7×	126	7.0k
Colin K. Combs United States	46	3.0k 0.8×	2.8k 0.7×	2.5k 0.7×	646 0.4×	862 0.6×	101	7.4k
Steven W. Barger United States	45	3.3k 0.8×	2.4k 0.6×	3.9k 1.2×	746 0.5×	977 0.7×	111	8.5k
Donna M. Wilcock United States	50	5.5k 1.4×	3.9k 1.0×	2.4k 0.7×	853 0.5×	766 0.5×	158	9.2k
Lih‐Fen Lue United States	44	3.4k 0.9×	2.8k 0.7×	1.8k 0.5×	582 0.4×	883 0.6×	79	6.7k
Magdalena Sastre United Kingdom	51	4.2k 1.1×	2.6k 0.7×	3.8k 1.1×	786 0.5×	576 0.4×	99	9.1k
Delphine Boche United Kingdom	43	3.5k 0.9×	3.5k 0.9×	1.9k 0.6×	878 0.6×	950 0.7×	98	7.0k
Alberto Serrano‐Pozo United States	35	5.4k 1.4×	2.7k 0.7×	2.5k 0.8×	756 0.5×	476 0.3×	73	8.2k
W. Sue T. Griffin United States	59	6.5k 1.7×	5.9k 1.5×	5.2k 1.5×	1.8k 1.2×	1.7k 1.2×	126	13.8k
Douglas G. Walker United States	64	6.4k 1.6×	5.1k 1.3×	3.9k 1.1×	977 0.6×	1.6k 1.2×	173	13.7k

Countries citing papers authored by Markus P. Kummer

Since Specialization

Citations

This map shows the geographic impact of Markus P. Kummer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Markus P. Kummer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Markus P. Kummer more than expected).

Fields of papers citing papers by Markus P. Kummer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Markus P. Kummer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Markus P. Kummer. The network helps show where Markus P. Kummer may publish in the future.

Co-authorship network of co-authors of Markus P. Kummer

This figure shows the co-authorship network connecting the top 25 collaborators of Markus P. Kummer. A scholar is included among the top collaborators of Markus P. Kummer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Markus P. Kummer. Markus P. Kummer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kummer, Markus P., Christina Ising, Christiane Kummer, et al.. (2021). Microglial PD‐1 stimulation by astrocytic PD‐L1 suppresses neuroinflammation and Alzheimer’s disease pathology. The EMBO Journal. 40(24). e108662–e108662. 68 indexed citations

Rueda‐Carrasco, Javier, María Jesús Martín‐Bermejo, Aldo Borroto, et al.. (2021). SFRP1 modulates astrocyte‐to‐microglia crosstalk in acute and chronic neuroinflammation. EMBO Reports. 22(11). e51696–e51696. 51 indexed citations

Gerrits, Emma, Nieske Brouwer, Susanne M. Kooistra, et al.. (2021). Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease. Acta Neuropathologica. 141(5). 681–696. 207 indexed citations breakdown →

Brosseron, Frederic, Andreas Traschütz, Catherine Widmann, et al.. (2018). Characterization and clinical use of inflammatory cerebrospinal fluid protein markers in Alzheimer’s disease. Alzheimer s Research & Therapy. 10(1). 25–25. 76 indexed citations

Sylvester, Marc, Nahal Brocke‐Ahmadinejad, Markus P. Kummer, et al.. (2017). Proteome profiling of s-nitrosylated synaptosomal proteins by isobaric mass tags. Journal of Neuroscience Methods. 291. 95–100. 6 indexed citations

Venegas, Carmen, Sathish Kumar, Bernardo S. Franklin, et al.. (2017). Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease. Nature. 552(7685). 355–361. 708 indexed citations breakdown →

Kummer, Markus P., Thomas Hammerschmidt, Alfredo Martı́nez, et al.. (2014). Ear2 Deletion Causes Early Memory and Learning Deficits in APP/PS1 Mice. Journal of Neuroscience. 34(26). 8845–8854. 50 indexed citations

Brosseron, Frederic, Marius Krauthausen, Markus P. Kummer, & Michael T. Heneka. (2014). Body Fluid Cytokine Levels in Mild Cognitive Impairment and Alzheimer’s Disease: a Comparative Overview. Molecular Neurobiology. 50(2). 534–544. 342 indexed citations

Stoffels, Burkhard, Susanne A. Snoek, Sjoerd van Bree, et al.. (2013). Postoperative Ileus Involves Interleukin-1 Receptor Signaling in Enteric Glia. Gastroenterology. 146(1). 176–187.e1. 117 indexed citations

10.

Yamanaka, Mitsugu, et al.. (2012). PPARγ/RXRα-Induced and CD36-Mediated Microglial Amyloid-β Phagocytosis Results in Cognitive Improvement in Amyloid Precursor Protein/Presenilin 1 Mice. Journal of Neuroscience. 32(48). 17321–17331. 280 indexed citations

11.

Kummer, Markus P., et al.. (2012). Nitric Oxide Decreases the Enzymatic Activity of Insulin Degrading Enzyme in APP/PS1 Mice. Journal of Neuroimmune Pharmacology. 7(1). 165–172. 23 indexed citations

12.

Backes, Heiko, Thomas Viel, Markus P. Kummer, et al.. (2012). Imaging microglial activation and glucose consumption in a mouse model of Alzheimer's disease. Neurobiology of Aging. 34(1). 351–354. 36 indexed citations

13.

Heneka, Michael T., Markus P. Kummer, Sascha Weggen, et al.. (2011). Molecular Mechanisms and Therapeutic Application of NSAIDs and Derived Compounds in Alzheimers Disease. Current Alzheimer Research. 8(2). 115–131. 40 indexed citations

14.

Mildner, Alexander, Bernhard Schlevogt, Katrin Kierdorf, et al.. (2011). Distinct and Non-Redundant Roles of Microglia and Myeloid Subsets in Mouse Models of Alzheimer's Disease. Journal of Neuroscience. 31(31). 11159–11171. 277 indexed citations

15.

Heneka, Michael T., M. Kerry O’Banion, Dick Terwel, & Markus P. Kummer. (2010). Neuroinflammatory processes in Alzheimer’s disease. Journal of Neural Transmission. 117(8). 919–947. 360 indexed citations

16.

Kummer, Markus P., et al.. (2008). Formation of Pmel17 Amyloid Is Regulated by Juxtamembrane Metalloproteinase Cleavage, and the Resulting C-terminal Fragment Is a Substrate for γ-Secretase. Journal of Biological Chemistry. 284(4). 2296–2306. 50 indexed citations

17.

Leuchtenberger, Stefanie, Markus P. Kummer, Thomas Kukar, et al.. (2005). Inhibitors of Rho‐kinase modulate amyloid‐β (Aβ) secretion but lack selectivity for Aβ42. Journal of Neurochemistry. 96(2). 355–365. 26 indexed citations

18.

Kanzler, Holger, et al.. (2000). Thyroglobulin type‐I‐like domains in invariant chain fusion proteins mediate resistance to cathepsin L digestion. FEBS Letters. 485(1). 67–70. 7 indexed citations

19.

Kappy, Michael S., et al.. (1999). Pathological Case of the Month. Archives of Pediatrics and Adolescent Medicine. 153(4). 427–427. 2 indexed citations

20.

Gadner, Helmut, et al.. (1985). [Cooperative studies in the treatment of acute lymphoblastic leukemia in children in Austria--report of 10 years' experience].. PubMed. 97(3). 140–8. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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