Marie‐Christine Lecomte
- Physiology top 2%
- Erythrocyte Function and Pathophysiology 48
- Cell Biology top 5%
- Caveolin-1 and cellular processes 4
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- Blood properties and coagulation 33
- Hematology top 10%
- Blood groups and transfusion 13
- Genetics top 10%
- Diabetes and associated disorders 11
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- Pancreatic function and diabetes 8
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- Ion channel regulation and function 4
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- Neonatal Health and Biochemistry 3
- Journals
- Journal of Biological Chemistry (5 papers)Journal of Clinical Investigation (4 papers)Blood (3 papers)
- Partner nations
- FranceUnited StatesPoland
In The Last Decade
Marie‐Christine Lecomte
61 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 100
- Physiology 894
- Cell Biology 344
- Pulmonary and Respiratory Medicine 586
- Hematology 143
- Genetics 129
Countries citing papers authored by Marie‐Christine Lecomte
This map shows the geographic impact of Marie‐Christine Lecomte's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marie‐Christine Lecomte with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marie‐Christine Lecomte more than expected).
Fields of papers citing papers by Marie‐Christine Lecomte
This network shows the impact of papers produced by Marie‐Christine Lecomte. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marie‐Christine Lecomte. The network helps show where Marie‐Christine Lecomte may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Marie‐Christine Lecomte, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 6 | |
| 2 | 2017 | 13 | |
| 3 | 2014 | 10 | |
| 4 | 2012 | 2 | |
| 5 | 2007 | 18 | |
| 6 | 2004 | 35 | |
| 7 | 2003 | 16 | |
| 8 | 2003 | 12 | |
| 9 | 2002 | 80 | |
| 10 | 1999 | 18 | |
| 11 | 1997 | 12 | |
| 12 | 1996 | 54 | |
| 13 | 1993 | 13 | |
| 14 | 1992 | 34 | |
| 15 | 1992 | 13 | |
| 16 | 1991 | 30 | |
| 17 | 1990 | 90 | |
| 18 | 1990 | 8 | |
| 19 | 1987 | 8 | |
| 20 | 1987 | 5 |
About Marie‐Christine Lecomte
Marie‐Christine Lecomte is a scholar working on Physiology, Hematology and Pulmonary and Respiratory Medicine, having authored 61 papers that have together received 1.6k indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (48 papers), Blood properties and coagulation (33 papers), Blood groups and transfusion (13 papers), Diabetes and associated disorders (11 papers), Pancreatic function and diabetes (8 papers), Ion channel regulation and function (4 papers), Caveolin-1 and cellular processes (4 papers) and Neonatal Health and Biochemistry (3 papers). The work is most often cited by research in Physiology (894 citations), Cell Biology (344 citations) and Pulmonary and Respiratory Medicine (586 citations). Marie‐Christine Lecomte has collaborated with scholars based in France, United States and Poland. Frequent co-authors include D Dhermy, Pierre Boivin, M Garbarz, H Gautero, Gaël Nicolas, Odile Bournier, Beata Machnicka, C Galand, Walter Gratzer and Aleksander F. Sikorski. Their work appears in journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.