Maria Savvaki

455 total citations
17 papers, 334 citations indexed

About

Maria Savvaki is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Maria Savvaki has authored 17 papers receiving a total of 334 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Developmental Neuroscience, 9 papers in Cellular and Molecular Neuroscience and 7 papers in Molecular Biology. Recurrent topics in Maria Savvaki's work include Neurogenesis and neuroplasticity mechanisms (10 papers), Neuroscience and Neuropharmacology Research (5 papers) and Cellular Mechanics and Interactions (4 papers). Maria Savvaki is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (10 papers), Neuroscience and Neuropharmacology Research (5 papers) and Cellular Mechanics and Interactions (4 papers). Maria Savvaki collaborates with scholars based in Greece, France and Japan. Maria Savvaki's co-authors include Domna Karagogeos, Lida Zoupi, Fotini Stylianopoulou, Antonios Stamatakis, Kazutada Watanabe, Kyriaki Thermos, Foteini Kiagiadaki, Kyriacos Kyriacou, Theofanis I. Panagiotaropoulos and Kleopas A. Kleopa and has published in prestigious journals such as Journal of Neuroscience, SHILAP Revista de lepidopterología and Journal of Cell Science.

In The Last Decade

Maria Savvaki

17 papers receiving 333 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Maria Savvaki Greece 11 160 131 80 66 49 17 334
Thomas Ostendorf Germany 5 168 1.1× 150 1.1× 64 0.8× 40 0.6× 52 1.1× 5 380
Erika Lorenzetto Italy 13 159 1.0× 149 1.1× 57 0.7× 46 0.7× 17 0.3× 16 362
Bianca Mages Germany 10 103 0.6× 70 0.5× 35 0.4× 34 0.5× 43 0.9× 16 419
Johanne Bertrand Canada 7 267 1.7× 317 2.4× 76 0.9× 45 0.7× 32 0.7× 10 528
Anna J. Khalaj United States 10 116 0.7× 110 0.8× 96 1.2× 16 0.2× 32 0.7× 11 392
Ana Luisa Piña Germany 11 243 1.5× 77 0.6× 28 0.3× 26 0.4× 37 0.8× 20 412
Luba Kojic Canada 8 188 1.2× 158 1.2× 15 0.2× 39 0.6× 32 0.7× 9 346
Seung‐Pil Yang Canada 8 200 1.3× 73 0.6× 15 0.2× 33 0.5× 88 1.8× 10 396
Jimena Baleriola Spain 10 291 1.8× 140 1.1× 47 0.6× 137 2.1× 45 0.9× 18 551

Countries citing papers authored by Maria Savvaki

Since Specialization
Citations

This map shows the geographic impact of Maria Savvaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maria Savvaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maria Savvaki more than expected).

Fields of papers citing papers by Maria Savvaki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maria Savvaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maria Savvaki. The network helps show where Maria Savvaki may publish in the future.

Co-authorship network of co-authors of Maria Savvaki

This figure shows the co-authorship network connecting the top 25 collaborators of Maria Savvaki. A scholar is included among the top collaborators of Maria Savvaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maria Savvaki. Maria Savvaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Nikoletopoulou, Vassiliki, et al.. (2025). Disruption of Oligodendroglial Autophagy Leads to Myelin Morphological Deficits, Neuronal Apoptosis, and Cognitive Decline in Aged Mice. Glia. 73(7). 1383–1397. 1 indexed citations
2.
Spanaki, Cleanthe, et al.. (2024). Glutamate-specific gene linked to human brain evolution enhances synaptic plasticity and cognitive processes. iScience. 27(2). 108821–108821. 3 indexed citations
3.
Savvaki, Maria, et al.. (2023). Nicotinamide enhances myelin production after demyelination through reduction of astrogliosis and microgliosis. Frontiers in Cellular Neuroscience. 17. 1201317–1201317. 5 indexed citations
4.
Kolotuev, Irina, et al.. (2022). Autophagic degradation of CNS myelin maintains axon integrity. SHILAP Revista de lepidopterología. 6(12). 93–107. 12 indexed citations
5.
Savvaki, Maria, et al.. (2021). Neuronal, but not glial, Contactin 2 negatively regulates axon regeneration in the injured adult optic nerve. European Journal of Neuroscience. 53(6). 1705–1721. 8 indexed citations
6.
Savvaki, Maria, et al.. (2021). Implication of Contactins in Demyelinating Pathologies. Life. 11(1). 51–51. 9 indexed citations
7.
Θεοδοσίου, Θεοδόσιος, Maria Savvaki, Aristides G. Eliopoulos, et al.. (2020). UniProt-Related Documents (UniReD): assisting wet lab biologists in their quest on finding novel counterparts in a protein network. NAR Genomics and Bioinformatics. 2(1). lqaa005–lqaa005. 11 indexed citations
8.
Kastriti, Maria Eleni, Maria Savvaki, Marina Vidaki, et al.. (2019). Ablation of CNTN2+ Pyramidal Neurons During Development Results in Defects in Neocortical Size and Axonal Tract Formation. Frontiers in Cellular Neuroscience. 13. 454–454. 11 indexed citations
9.
Karagogeos, Domna, et al.. (2019). Models and treatments for traumatic optic neuropathy and demyelinating optic neuritis. Developmental Neurobiology. 79(8). 819–836. 24 indexed citations
10.
Hivert, Bruno, Margaux Saint‐Martin, Nelly Noraz, et al.. (2017). The Kv1-associated molecules TAG-1 and Caspr2 are selectively targeted to the axon initial segment in hippocampal neurons. Journal of Cell Science. 130(13). 2209–2220. 26 indexed citations
11.
Zoupi, Lida, et al.. (2017). The function of contactin‐2/TAG‐1 in oligodendrocytes in health and demyelinating pathology. Glia. 66(3). 576–591. 26 indexed citations
12.
Zoupi, Lida, Maria Savvaki, & Domna Karagogeos. (2011). Axons and myelinating glia: An intimate contact. IUBMB Life. 63(9). n/a–n/a. 12 indexed citations
13.
Savvaki, Maria, Lida Zoupi, Antonios Stamatakis, et al.. (2010). The Expression of TAG-1 in Glial Cells Is Sufficient for the Formation of the Juxtaparanodal Complex and the Phenotypic Rescue of Tag-1 Homozygous Mutants in the CNS. Journal of Neuroscience. 30(42). 13943–13954. 52 indexed citations
14.
Alpantaki, Kalliopi, Maria Savvaki, & Domna Karagogeos. (2010). Expression of cell adhesion molecule L1 in the long head of biceps tendon.. PubMed. 56 Suppl. OL1286–9. 3 indexed citations
15.
Kiagiadaki, Foteini, Maria Savvaki, & Kyriaki Thermos. (2009). Activation of somatostatin receptor (sst5) protects the rat retina from AMPA-induced neurotoxicity. Neuropharmacology. 58(1). 297–303. 29 indexed citations
16.
Savvaki, Maria, Theofanis I. Panagiotaropoulos, Antonios Stamatakis, et al.. (2008). Impairment of learning and memory in TAG-1 deficient mice associated with shorter CNS internodes and disrupted juxtaparanodes. Molecular and Cellular Neuroscience. 39(3). 478–490. 62 indexed citations
17.
Miguez, Andrés, Maria Savvaki, Aude Muzerelle, et al.. (2008). Structural Requirement of TAG-1 for Retinal Ganglion Cell Axons and Myelin in the Mouse Optic Nerve. Journal of Neuroscience. 28(30). 7624–7636. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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