Anna J. Khalaj

502 total citations
11 papers, 392 citations indexed

About

Anna J. Khalaj is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Immunology. According to data from OpenAlex, Anna J. Khalaj has authored 11 papers receiving a total of 392 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Pathology and Forensic Medicine, 4 papers in Molecular Biology and 4 papers in Immunology. Recurrent topics in Anna J. Khalaj's work include Multiple Sclerosis Research Studies (7 papers), Receptor Mechanisms and Signaling (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). Anna J. Khalaj is often cited by papers focused on Multiple Sclerosis Research Studies (7 papers), Receptor Mechanisms and Signaling (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). Anna J. Khalaj collaborates with scholars based in United States, Israel and Germany. Anna J. Khalaj's co-authors include Seema K. Tiwari‐Woodruff, Spencer M. Moore, Jonathan Hasselmann, Shalini Kumar, John F. Marshall, Subir Ghosh, Jenny I. Szu, Norio Yasui, John A. Katzenellenbogen and Devin K. Binder and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Cell Biology and Neurology.

In The Last Decade

Anna J. Khalaj

11 papers receiving 391 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Anna J. Khalaj United States	10	136	116	110	96	86	11	392
Spencer M. Moore United States	13	146 1.1×	159 1.4×	78 0.7×	135 1.4×	100 1.2×	21	529
Mario Mangiardi United States	6	200 1.5×	149 1.3×	168 1.5×	188 2.0×	122 1.4×	6	535
Laura A. Greco Italy	6	205 1.5×	84 0.7×	157 1.4×	108 1.1×	98 1.1×	8	371
Eve E. Kelland United States	11	74 0.5×	133 1.1×	195 1.8×	182 1.9×	107 1.2×	15	459
Jordi Tomàs-Roig Spain	10	69 0.5×	152 1.3×	90 0.8×	49 0.5×	48 0.6×	15	369
Óscar G. Vidaurre Spain	10	89 0.7×	290 2.5×	161 1.5×	61 0.6×	105 1.2×	12	506
Marina Ziehn United States	8	241 1.8×	55 0.5×	52 0.5×	80 0.8×	150 1.7×	16	397
Antonia M. H. Piergies United States	6	62 0.5×	149 1.3×	59 0.5×	20 0.2×	137 1.6×	11	499
Sharon W. Way United States	8	52 0.4×	358 3.1×	76 0.7×	55 0.6×	45 0.5×	9	627
Masae Yaguchi Japan	9	41 0.3×	119 1.0×	151 1.4×	33 0.3×	48 0.6×	14	421

Countries citing papers authored by Anna J. Khalaj

Since Specialization

Citations

This map shows the geographic impact of Anna J. Khalaj's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna J. Khalaj with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna J. Khalaj more than expected).

Fields of papers citing papers by Anna J. Khalaj

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anna J. Khalaj. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna J. Khalaj. The network helps show where Anna J. Khalaj may publish in the future.

Co-authorship network of co-authors of Anna J. Khalaj

This figure shows the co-authorship network connecting the top 25 collaborators of Anna J. Khalaj. A scholar is included among the top collaborators of Anna J. Khalaj based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anna J. Khalaj. Anna J. Khalaj is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

Khalaj, Anna J., Fredrik Sterky, Alessandra Sclip, et al.. (2020). Deorphanizing FAM19A proteins as pan-neurexin ligands with an unusual biosynthetic binding mechanism. The Journal of Cell Biology. 219(9). 27 indexed citations

Lapato, Andrew, Jenny I. Szu, Jonathan Hasselmann, et al.. (2017). Chronic demyelination-induced seizures. Neuroscience. 346. 409–422. 47 indexed citations

Hasselmann, Jonathan, et al.. (2017). Consistent induction of chronic experimental autoimmune encephalomyelitis in C57BL/6 mice for the longitudinal study of pathology and repair. Journal of Neuroscience Methods. 284. 71–84. 50 indexed citations

Khalaj, Anna J., et al.. (2016). Nudging oligodendrocyte intrinsic signaling to remyelinate and repair: Estrogen receptor ligand effects. The Journal of Steroid Biochemistry and Molecular Biology. 160. 43–52. 34 indexed citations

Alaghband, Yasaman, et al.. (2014). Retrieval-induced NMDA receptor-dependent Arc expression in two models of cocaine-cue memory. Neurobiology of Learning and Memory. 116. 79–89. 21 indexed citations

Moore, Spencer M., Anna J. Khalaj, Shalini Kumar, et al.. (2014). Multiple functional therapeutic effects of the estrogen receptor β agonist indazole-Cl in a mouse model of multiple sclerosis. Proceedings of the National Academy of Sciences. 111(50). 18061–18066. 75 indexed citations

Moore, Spencer M., et al.. (2014). Restoration of axon conduction and motor deficits by therapeutic treatment with glatiramer acetate. Journal of Neuroscience Research. 92(12). 1621–1636. 16 indexed citations

Moore, Spencer M., et al.. (2013). Therapeutic Laquinimod Treatment Restores Axon Myelination, Callosal Conduction and Motor Deficit in a Chronic Mouse Model of Multiple Sclerosis (P05.197). Neurology. 80(7_supplement). 1 indexed citations

Moore, Spencer M., Anna J. Khalaj, Rhusheet Patel, et al.. (2013). Therapeutic laquinimod treatment decreases inflammation, initiates axon remyelination, and improves motor deficit in a mouse model of multiple sclerosis. Brain and Behavior. 3(6). 664–682. 38 indexed citations

10.

Khalaj, Anna J., et al.. (2013). Estrogen receptor (ER) β expression in oligodendrocytes is required for attenuation of clinical disease by an ERβ ligand. Proceedings of the National Academy of Sciences. 110(47). 19125–19130. 64 indexed citations

11.

Khalaj, Anna J., et al.. (2011). Dopamine D1 receptor antagonism impairs extinction of cocaine-cue memories. Behavioural Brain Research. 226(1). 357–360. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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