María J. Guerra

4.2k total citations
75 papers, 3.5k citations indexed

About

María J. Guerra is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, María J. Guerra has authored 75 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Cellular and Molecular Neuroscience, 31 papers in Molecular Biology and 24 papers in Neurology. Recurrent topics in María J. Guerra's work include Parkinson's Disease Mechanisms and Treatments (21 papers), Nerve injury and regeneration (19 papers) and Receptor Mechanisms and Signaling (19 papers). María J. Guerra is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (21 papers), Nerve injury and regeneration (19 papers) and Receptor Mechanisms and Signaling (19 papers). María J. Guerra collaborates with scholars based in Spain, Sweden and Germany. María J. Guerra's co-authors include José L. Labandeira‐García, Ana I. Rodríguez‐Pérez, Jannette Rodrı́guez-Pallares, G. Rozas, Ana Muñoz, Begoña Villar‐Cheda, P. Rey, Pablo Garrido‐Gil, Juan A. Parga and Antonio Dominguez‐Meijide and has published in prestigious journals such as Brain, The Journal of Comparative Neurology and Brain Research.

In The Last Decade

María J. Guerra

74 papers receiving 3.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
María J. Guerra Spain	36	1.7k	1.2k	1.1k	763	705	75	3.5k
Jannette Rodrı́guez-Pallares Spain	23	809 0.5×	768 0.6×	426 0.4×	417 0.5×	499 0.7×	49	1.8k
Andrzej Członkowski Poland	32	1.8k 1.0×	887 0.7×	900 0.8×	941 1.2×	112 0.2×	89	3.4k
Pablo Garrido‐Gil Spain	26	595 0.3×	633 0.5×	486 0.4×	398 0.5×	476 0.7×	45	1.7k
Catherine Widmann France	29	1.3k 0.7×	1.5k 1.2×	416 0.4×	748 1.0×	253 0.4×	47	3.7k
Yutaka Kōyama Japan	32	1.2k 0.7×	1.4k 1.2×	419 0.4×	784 1.0×	100 0.1×	125	3.3k
Rajesh N. Kalaria United States	35	870 0.5×	1.3k 1.1×	576 0.5×	1.1k 1.5×	167 0.2×	65	3.9k
Małgorzata Chalimoniuk Poland	28	729 0.4×	850 0.7×	529 0.5×	573 0.8×	95 0.1×	100	2.9k
Karen Horsburgh United Kingdom	41	983 0.6×	1.6k 1.3×	836 0.7×	1.7k 2.2×	175 0.2×	88	4.6k
Francesca Boscia Italy	35	1.5k 0.8×	1.5k 1.2×	241 0.2×	421 0.6×	229 0.3×	80	3.0k
Laibaik Park United States	30	421 0.2×	809 0.7×	663 0.6×	1.8k 2.3×	362 0.5×	39	3.6k

Countries citing papers authored by María J. Guerra

Since Specialization

Citations

This map shows the geographic impact of María J. Guerra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by María J. Guerra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites María J. Guerra more than expected).

Fields of papers citing papers by María J. Guerra

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by María J. Guerra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by María J. Guerra. The network helps show where María J. Guerra may publish in the future.

Co-authorship network of co-authors of María J. Guerra

This figure shows the co-authorship network connecting the top 25 collaborators of María J. Guerra. A scholar is included among the top collaborators of María J. Guerra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with María J. Guerra. María J. Guerra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Fernández‐Espejo, Emilio, et al.. (2025). Association between serum myeloperoxidase enzyme activity and Parkinson’s disease status. npj Parkinson s Disease. 11(1). 94–94. 2 indexed citations

Rodríguez‐Pérez, Ana I., Maria A. Costa-Besada, Andrea López-López, et al.. (2024). Modulation of Mitochondrial Dynamics by the Angiotensin System in Dopaminergic Neurons and Microglia. Aging and Disease. 16(5). 3180–3203. 4 indexed citations

Borrajo, Ana, Ana I. Rodríguez‐Pérez, Begoña Villar‐Cheda, María J. Guerra, & José L. Labandeira‐García. (2014). Inhibition of the microglial response is essential for the neuroprotective effects of Rho-kinase inhibitors on MPTP-induced dopaminergic cell death. Neuropharmacology. 85. 1–8. 65 indexed citations

Dominguez‐Meijide, Antonio, et al.. (2013). Effect of chronic treatment with angiotensin type 1 receptor antagonists on striatal dopamine levels in normal rats and in a rat model of Parkinson's disease treated with l-DOPA. Neuropharmacology. 76. 156–168. 38 indexed citations

Garrido‐Gil, Pablo, Belen Joglar, Ana I. Rodríguez‐Pérez, María J. Guerra, & José L. Labandeira‐García. (2012). Involvement of PPAR-γ in the neuroprotective and anti-inflammatory effects of angiotensin type 1 receptor inhibition: effects of the receptor antagonist telmisartan and receptor deletion in a mouse MPTP model of Parkinson's disease. Journal of Neuroinflammation. 9(1). 38–38. 99 indexed citations

Labandeira‐García, José L., Jannette Rodrı́guez-Pallares, Ana I. Rodríguez‐Pérez, et al.. (2012). Brain angiotensin and dopaminergic degeneration: relevance to Parkinson's disease.. PubMed. 1(3). 226–44. 30 indexed citations

Villar‐Cheda, Begoña, Antonio Dominguez‐Meijide, Belen Joglar, et al.. (2012). Involvement of microglial RhoA/Rho-Kinase pathway activation in the dopaminergic neuron death. Role of angiotensin via angiotensin type 1 receptors. Neurobiology of Disease. 47(2). 268–279. 91 indexed citations

Rodríguez‐Pérez, Ana I., Rita Valenzuela, Begoña Villar‐Cheda, María J. Guerra, & José L. Labandeira‐García. (2011). Dopaminergic neuroprotection of hormonal replacement therapy in young and aged menopausal rats: role of the brain angiotensin system. Brain. 135(1). 124–138. 56 indexed citations

Villar‐Cheda, Begoña, Rita Valenzuela, Ana I. Rodríguez‐Pérez, María J. Guerra, & José L. Labandeira‐García. (2010). Aging-related changes in the nigral angiotensin system enhances proinflammatory and pro-oxidative markers and 6-OHDA-induced dopaminergic degeneration. Neurobiology of Aging. 33(1). 204.e1–204.e11. 81 indexed citations

10.

Parga, Juan A., et al.. (2008). Different effects of anti‐sonic hedgehog antibodies and the hedgehog pathway inhibitor cyclopamine on generation of dopaminergic neurons from neurospheres of mesencephalic precursors. Developmental Dynamics. 237(4). 909–917. 5 indexed citations

11.

Rodrı́guez-Pallares, Jannette, Juan A. Parga, Ana Muñoz, et al.. (2007). Mechanism of 6‐hydroxydopamine neurotoxicity: the role of NADPH oxidase and microglial activation in 6‐hydroxydopamine‐induced degeneration of dopaminergic neurons. Journal of Neurochemistry. 103(1). 145–156. 183 indexed citations

12.

Muñoz, Ana, Ana López, Héctor J. Caruncho, María J. Guerra, & José L. Labandeira‐García. (2001). Long-Term Cortical Atrophy after Excitotoxic Striatal Lesion: Effects of Intrastriatal Fetal-Striatum Grafts and Implications for Huntington Disease. Journal of Neuropathology & Experimental Neurology. 60(8). 786–797. 5 indexed citations

13.

Caruncho, Héctor J., Jannette Rodrı́guez-Pallares, María J. Guerra, & José L. Labandeira‐García. (1998). Mature intrastriatal striatal grafts revert the changes in the expression of pallidal and thalamic α1, α2 and β2/3 GABAA receptor subunit induced by ibotenic acid lesions in the rat striatum. Molecular Brain Research. 57(2). 301–309. 4 indexed citations

14.

Rozas, G., María Elena López-Martín, María J. Guerra, & José L. Labandeira‐García. (1998). The overall rod performance test in the MPTP-treated-mouse model of Parkinsonism. Journal of Neuroscience Methods. 83(2). 165–175. 201 indexed citations

15.

Caruncho, Héctor J., Isabel Liste, G. Rozas, et al.. (1997). Time course of striatal, pallidal and thalamic α1, α2 and β2/3 GABAA receptor subunit changes induced by unilateral 6-OHDA lesion of the nigrostriatal pathway. Molecular Brain Research. 48(2). 243–250. 17 indexed citations

16.

Guerra, María J., et al.. (1996). Previous dopaminergic innervation is not necessary for the development of dopamine supersensitivity in rat striatal neurons. Neuroscience Letters. 206(2-3). 137–140. 3 indexed citations

17.

Rozas, G., Isabel Liste, María Elena López-Martín, et al.. (1996). Intrathalamic Implants of GABA-Releasing Polymer Matrices Reduce Motor Impairments in Rats with Excitotoxically Lesioned Striata. Experimental Neurology. 142(2). 323–330. 4 indexed citations

18.

Labandeira‐García, José L., et al.. (1995). Intrathalamic striatal grafts survive and affect circling behaviour in adult rats with excitotoxically lesioned striatum. Neuroscience. 68(3). 737–749. 12 indexed citations

19.

Labandeira‐García, José L. & María J. Guerra. (1994). Cortical stimulation induces fos expression in intrastriatal striatal grafts. Brain Research. 652(1). 87–97. 22 indexed citations

20.

Labandeira‐García, José L., et al.. (1994). Comparison between normal developing striatum and developing striatal grafts using drug-induced fos expression and neuron-specific enolase immunohistochemistry. Neuroscience. 60(2). 399–415. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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