Margaret E. Duncan
- Cancer Research top 2%
- Oncology top 5%
- Molecular Biology
- Immunology and Allergy top 2%
- Hematology top 10%
- Co-authors
- John E. FothergillWilliam T. MelvinGraeme I. MurrayP. BrookesJudith McKayG I MurrayJonathan P. RichardsonNuala A. Booth
- Topics
- Protease and Inhibitor Mechanisms (6 papers)Carcinogens and Genotoxicity Assessment (5 papers)Peptidase Inhibition and Analysis (5 papers)
- Partner nations
- United KingdomUnited StatesGermany
In The Last Decade
Margaret E. Duncan
18 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 91
- Cancer Research 834
- Oncology 631
- Molecular Biology 513
- Immunology and Allergy 240
- Hematology 138
Countries citing papers authored by Margaret E. Duncan
This map shows the geographic impact of Margaret E. Duncan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Margaret E. Duncan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Margaret E. Duncan more than expected).
Fields of papers citing papers by Margaret E. Duncan
This network shows the impact of papers produced by Margaret E. Duncan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Margaret E. Duncan. The network helps show where Margaret E. Duncan may publish in the future.
Co-authorship network of co-authors of Margaret E. Duncan
This figure shows the co-authorship network connecting the top 25 collaborators of Margaret E. Duncan. A scholar is included among the top collaborators of Margaret E. Duncan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Margaret E. Duncan. Margaret E. Duncan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 3 | |
| 3 | 18 | |
| 4 | 1 | |
| 5 | 49 | |
| 6 | 252 | |
| 7 | 24 | |
| 8 | 388 | |
| 9 | 2 | |
| 10 | 297 | |
| 11 | 17 | |
| 12 | 2 | |
| 13 | 18 | |
| 14 | 56 | |
| 15 | The induction of azaguanine-resistant mutants in cultured Chinese hamster cells by reactive derivatives of carcinogenic hydrocarbons. | 67 |
| 16 | 16 | |
| 17 | 43 | |
| 18 | 40 |
About Margaret E. Duncan
Margaret E. Duncan is a scholar working on Cancer Research, Immunology and Allergy and Emergency Medical Services, having authored 18 papers that have together received 1.3k indexed citations. Recurring topics across this work include Protease and Inhibitor Mechanisms (6 papers), Carcinogens and Genotoxicity Assessment (5 papers) and Peptidase Inhibition and Analysis (5 papers). The work is most often cited by research in Cancer Research (834 citations), Immunology and Allergy (240 citations) and Oncology (631 citations). Margaret E. Duncan has collaborated with scholars based in United Kingdom, United States and Germany. Frequent co-authors include John E. Fothergill, William T. Melvin, Graeme I. Murray, P. Brookes, Judith McKay, G I Murray, Jonathan P. Richardson, Nuala A. Booth, Sybil M. McAleese and Rainald Fischer. Their work appears in journals such as Nature, Nature Medicine and European Journal of Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.