Margaret Colden‐Stanfield
- Rheumatology top 2%
- Molecular Biology
- Hematology top 5%
- Immunology top 10%
- Physiology top 10%
- Co-authors
- Silvia S. PierangeliXiaowei LiuWilliam P. SchillingDiana L. KunzeEdward N. HarrisRicardo EspinolaJohn H. BarkerGary L. Anderson
- Topics
- Ion channel regulation and function (6 papers)Receptor Mechanisms and Signaling (5 papers)Systemic Lupus Erythematosus Research (5 papers)
- Partner nations
- United StatesFranceChina
In The Last Decade
Margaret Colden‐Stanfield
16 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 87
- Rheumatology 530
- Molecular Biology 322
- Hematology 295
- Immunology 227
- Physiology 202
Countries citing papers authored by Margaret Colden‐Stanfield
This map shows the geographic impact of Margaret Colden‐Stanfield's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Margaret Colden‐Stanfield with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Margaret Colden‐Stanfield more than expected).
Fields of papers citing papers by Margaret Colden‐Stanfield
This network shows the impact of papers produced by Margaret Colden‐Stanfield. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Margaret Colden‐Stanfield. The network helps show where Margaret Colden‐Stanfield may publish in the future.
Co-authorship network of co-authors of Margaret Colden‐Stanfield
This figure shows the co-authorship network connecting the top 25 collaborators of Margaret Colden‐Stanfield. A scholar is included among the top collaborators of Margaret Colden‐Stanfield based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Margaret Colden‐Stanfield. Margaret Colden‐Stanfield is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 59 | |
| 3 | 8 | |
| 4 | 73 | |
| 5 | 134 | |
| 6 | 17 | |
| 7 | 13 | |
| 8 | 75 | |
| 9 | 255 | |
| 10 | 17 | |
| 11 | 13 | |
| 12 | 24 | |
| 13 | 55 | |
| 14 | 63 | |
| 15 | Morphine induces an intracellular alkalinization in bovine aortic endothelial cells (BAECs). | 3 |
| 16 | 252 |
About Margaret Colden‐Stanfield
Margaret Colden‐Stanfield is a scholar working on Rheumatology, Immunology and Allergy and Hematology, having authored 16 papers that have together received 1.1k indexed citations. Recurring topics across this work include Ion channel regulation and function (6 papers), Receptor Mechanisms and Signaling (5 papers) and Systemic Lupus Erythematosus Research (5 papers). The work is most often cited by research in Rheumatology (530 citations), Immunology and Allergy (168 citations) and Hematology (295 citations). Margaret Colden‐Stanfield has collaborated with scholars based in United States, France and China. Frequent co-authors include Silvia S. Pierangeli, Xiaowei Liu, William P. Schilling, Diana L. Kunze, Edward N. Harris, Ricardo Espinola, John H. Barker, Gary L. Anderson, L. T. Navarro and A K Ritchie. Their work appears in journals such as Circulation, The Journal of Immunology and Circulation Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.