Marcin Ufnal
- Molecular Biology top 5%
- Physiology top 2%
- Public Health, Environmental and Occupational Health top 5%
- Cardiology and Cardiovascular Medicine top 5%
- Biochemistry top 1%
- Co-authors
- Piotr KonopelskiRyszard OstaszewskiKinga JaworskaAdrian DrapałaLenka TomášováAnna Żądło‐DobrowolskaMariusz SikoraJanusz Skrzypecki
- Topics
- Gut microbiota and health (30 papers)Diet and metabolism studies (28 papers)Sulfur Compounds in Biology (20 papers)
- Partner nations
- PolandSlovakiaUnited States
In The Last Decade
Marcin Ufnal
112 papers receiving 3.1k citations
Peers
Comparison fields: 5 of 128
- Molecular Biology 1.6k
- Physiology 1.2k
- Public Health, Environmental and Occupational Health 431
- Cardiology and Cardiovascular Medicine 406
- Biochemistry 393
Countries citing papers authored by Marcin Ufnal
This map shows the geographic impact of Marcin Ufnal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marcin Ufnal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marcin Ufnal more than expected).
Fields of papers citing papers by Marcin Ufnal
This network shows the impact of papers produced by Marcin Ufnal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marcin Ufnal. The network helps show where Marcin Ufnal may publish in the future.
Co-authorship network of co-authors of Marcin Ufnal
This figure shows the co-authorship network connecting the top 25 collaborators of Marcin Ufnal. A scholar is included among the top collaborators of Marcin Ufnal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marcin Ufnal. Marcin Ufnal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 8 | |
| 3 | 2 | |
| 4 | 4 | |
| 5 | 7 | |
| 6 | 7 | |
| 7 | 2 | |
| 8 | 17 | |
| 9 | 4 | |
| 10 | 26 | |
| 11 | 63 | |
| 12 | 101 | |
| 13 | 31 | |
| 14 | 31 | |
| 15 | 3 | |
| 16 | 86 | |
| 17 | 51 | |
| 18 | 36 | |
| 19 | 40 | |
| 20 | [The role of nitric oxide, hydrogen sulfide and carbon monoxide in the regulation of the circulatory system and their pharmacotherapeutic potential]. | 9 |
About Marcin Ufnal
Marcin Ufnal is a scholar working on Biological Psychiatry, Biochemistry and Behavioral Neuroscience, having authored 115 papers that have together received 3.2k indexed citations. Recurring topics across this work include Gut microbiota and health (30 papers), Diet and metabolism studies (28 papers) and Sulfur Compounds in Biology (20 papers). The work is most often cited by research in Biological Psychiatry (341 citations), Biochemistry (393 citations) and Physiology (1.2k citations). Marcin Ufnal has collaborated with scholars based in Poland, Slovakia and United States. Frequent co-authors include Piotr Konopelski, Ryszard Ostaszewski, Kinga Jaworska, Adrian Drapała, Lenka Tomášová, Anna Żądło‐Dobrowolska, Mariusz Sikora, Janusz Skrzypecki, Tomasz Huć and Tymoteusz Żera. Their work appears in journals such as PLoS ONE, Scientific Reports and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.