Mani Larijani

1.6k total citations
48 papers, 1.2k citations indexed

About

Mani Larijani is a scholar working on Immunology, Molecular Biology and Virology. According to data from OpenAlex, Mani Larijani has authored 48 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Immunology, 24 papers in Molecular Biology and 9 papers in Virology. Recurrent topics in Mani Larijani's work include T-cell and B-cell Immunology (22 papers), Immune Cell Function and Interaction (21 papers) and CRISPR and Genetic Engineering (9 papers). Mani Larijani is often cited by papers focused on T-cell and B-cell Immunology (22 papers), Immune Cell Function and Interaction (21 papers) and CRISPR and Genetic Engineering (9 papers). Mani Larijani collaborates with scholars based in Canada, United States and Sweden. Mani Larijani's co-authors include Alberto Martín, Justin J. King, Michael D. Grant, Maribel Berrú, Alexander P. Petrov, Sergey N. Krylov, Hala Abdouni, Calvin Yu, Ronda Bransteitter and Siegfried Hapfelmeier and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Mani Larijani

47 papers receiving 1.2k citations

Peers

Mani Larijani
Robert W. Maul United States
Joyce Hu United States
J J O'Shea United States
Shari M. Kaiser United States
Carol Dahlberg United States
Laurence Chatel Switzerland
Andrew Pincetic United States
Lissa R. Herron United States
Karen Duus United States
Robert W. Maul United States
Mani Larijani
Citations per year, relative to Mani Larijani Mani Larijani (= 1×) peers Robert W. Maul

Countries citing papers authored by Mani Larijani

Since Specialization
Citations

This map shows the geographic impact of Mani Larijani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mani Larijani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mani Larijani more than expected).

Fields of papers citing papers by Mani Larijani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mani Larijani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mani Larijani. The network helps show where Mani Larijani may publish in the future.

Co-authorship network of co-authors of Mani Larijani

This figure shows the co-authorship network connecting the top 25 collaborators of Mani Larijani. A scholar is included among the top collaborators of Mani Larijani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mani Larijani. Mani Larijani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Dávalos, Liliana M., et al.. (2023). Evolutionary potential of the monkeypox genome arising from interactions with human APOBEC3 enzymes. Virus Evolution. 9(2). vead047–vead047. 10 indexed citations
2.
Solbakken, Monica Hongrø, Khalil Eslamloo, Cassandra Collins, et al.. (2022). Ancestral reconstruction reveals catalytic inactivation of activation-induced cytidine deaminase concomitant with cold water adaption in the Gadiformes bony fish. BMC Biology. 20(1). 293–293. 8 indexed citations
3.
Grant, Michael D., et al.. (2019). APOBEC3G Regulation of the Evolutionary Race Between Adaptive Immunity and Viral Immune Escape Is Deeply Imprinted in the HIV Genome. Frontiers in Immunology. 9. 3032–3032. 3 indexed citations
4.
Eslamloo, Khalil, et al.. (2019). Characterization and Transcript Expression Analyses of Atlantic Cod Viperin. Frontiers in Immunology. 10. 311–311. 20 indexed citations
5.
Methot, Stephen P., Ludivine C. Litzler, Anne‐Marie Patenaude, et al.. (2018). A licensing step links AID to transcription elongation for mutagenesis in B cells. Nature Communications. 9(1). 1248–1248. 32 indexed citations
6.
Chelakkot, Vipin Shankar, María Licursi, Justin J. King, et al.. (2018). Enhancement of Cancer-Specific Protoporphyrin IX Fluorescence by Targeting Oncogenic Ras/MEK Pathway. Theranostics. 8(8). 2134–2146. 50 indexed citations
7.
King, Justin J., et al.. (2015). Genes and Environment as Predisposing Factors in Autoimmunity: Acceleration of Spontaneous Thyroiditis by Dietary Iodide in NOD.H2h4Mice. International Reviews of Immunology. 34(6). 542–556. 22 indexed citations
8.
King, Justin J., et al.. (2015). Catalytic Pocket Inaccessibility of Activation-Induced Cytidine Deaminase Is a Safeguard against Excessive Mutagenic Activity. Structure. 23(4). 615–627. 33 indexed citations
9.
Zahn, Astrid, Anne‐Marie Patenaude, Stephen P. Methot, et al.. (2014). Activation induced deaminase C-terminal domain links DNA breaks to end protection and repair during class switch recombination. Proceedings of the National Academy of Sciences. 111(11). E988–97. 41 indexed citations
10.
Gallant, Maureen, et al.. (2014). Positioning of APOBEC3G/F Mutational Hotspots in the Human Immunodeficiency Virus Genome Favors Reduced Recognition by CD8+ T Cells. PLoS ONE. 9(4). e93428–e93428. 41 indexed citations
11.
Doucette, Lance P., Susan Moore, Kathy Hodgkinson, et al.. (2013). Identification of a novel in-frame deletion in KCNQ4 (DFNA2A) and evidence of multiple phenocopies of unknown origin in a family with ADSNHL. European Journal of Human Genetics. 21(10). 1112–1119. 9 indexed citations
12.
Grant, Michael D., et al.. (2012). Emerging complexities of APOBEC3G action on immunity and viral fitness during HIV infection and treatment. Retrovirology. 9(1). 35–35. 30 indexed citations
13.
Larijani, Mani & Alberto Martín. (2012). The biochemistry of activation-induced deaminase and its physiological functions. Seminars in Immunology. 24(4). 255–263. 28 indexed citations
14.
Larijani, Mani, et al.. (2006). Antibody Diversification: Mutational Mechanisms and Oncogenesis. Immunologic Research. 35(1-2). 75–88. 12 indexed citations
15.
Larijani, Mani, et al.. (2005). The mutation spectrum of purified AID is similar to the mutability index in Ramos cells and in ung?/?msh2?/? mice. Immunogenetics. 56(11). 840–845. 52 indexed citations
16.
Larijani, Mani. (2005). Lack of MSH2 involvement differentiates V(D)J recombination from other non-homologous end joining events. Nucleic Acids Research. 33(21). 6733–6742. 9 indexed citations
17.
Larijani, Mani, et al.. (2005). Coding Joint Diversity in Mature and Immature B‐Cell Lines. Scandinavian Journal of Immunology. 62(s1). 114–118. 1 indexed citations
18.
Larijani, Mani, Timothy M. Sonbuchner, Ronda Bransteitter, et al.. (2004). Methylation protects cytidines from AID-mediated deamination. Molecular Immunology. 42(5). 599–604. 62 indexed citations
19.
Montalbano, Alina P., Kisani M. Ogwaro, Adam G. W. Matthews, et al.. (2003). V(D)J Recombination Frequencies Can Be Profoundly Affected by Changes in the Spacer Sequence. The Journal of Immunology. 171(10). 5296–5304. 29 indexed citations
20.
Larijani, Mani, et al.. (1999). The role of components of recombination signal sequences in immunoglobulin gene segment usage: a V81x model. Nucleic Acids Research. 27(11). 2304–2309. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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