Robert W. Maul

2.6k total citations
50 papers, 1.8k citations indexed

About

Robert W. Maul is a scholar working on Immunology, Molecular Biology and Genetics. According to data from OpenAlex, Robert W. Maul has authored 50 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Immunology, 23 papers in Molecular Biology and 10 papers in Genetics. Recurrent topics in Robert W. Maul's work include Immune Cell Function and Interaction (25 papers), T-cell and B-cell Immunology (24 papers) and DNA Repair Mechanisms (16 papers). Robert W. Maul is often cited by papers focused on Immune Cell Function and Interaction (25 papers), T-cell and B-cell Immunology (24 papers) and DNA Repair Mechanisms (16 papers). Robert W. Maul collaborates with scholars based in United States, France and Poland. Robert W. Maul's co-authors include Patricia J. Gearhart, Marc K. Jenkins, Kathryn A. Pape, Justin J. Taylor, Mark D. Sutton, Huseyin Saribasak, William Yang, Rahul M. Kohli, James T. Stivers and Shaun R. Abrams and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Robert W. Maul

49 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert W. Maul United States 23 972 781 257 229 180 50 1.8k
Ronda Bransteitter United States 12 887 0.9× 1.1k 1.4× 212 0.8× 383 1.7× 148 0.8× 14 2.0k
Mani Larijani Canada 22 656 0.7× 575 0.7× 125 0.5× 149 0.7× 139 0.8× 48 1.2k
Vera Chan Hong Kong 20 1.4k 1.5× 540 0.7× 143 0.6× 140 0.6× 318 1.8× 40 2.2k
Marilyn Diaz United States 25 1.2k 1.3× 966 1.2× 223 0.9× 165 0.7× 165 0.9× 48 2.2k
Maria A. Turchaninova Russia 13 1.3k 1.3× 553 0.7× 106 0.4× 176 0.8× 469 2.6× 21 1.8k
Annie Yang United States 14 655 0.7× 462 0.6× 168 0.7× 278 1.2× 317 1.8× 40 1.3k
Ahmad Faili France 18 915 0.9× 843 1.1× 178 0.7× 150 0.7× 181 1.0× 39 1.8k
Heather Hinton Switzerland 19 781 0.8× 721 0.9× 81 0.3× 214 0.9× 316 1.8× 33 1.6k
Maria D. Iglesias-Ussel United States 15 691 0.7× 766 1.0× 79 0.3× 172 0.8× 114 0.6× 25 1.4k
Carol E. Schrader United States 23 1.5k 1.6× 1.3k 1.7× 135 0.5× 413 1.8× 355 2.0× 48 2.5k

Countries citing papers authored by Robert W. Maul

Since Specialization
Citations

This map shows the geographic impact of Robert W. Maul's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert W. Maul with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert W. Maul more than expected).

Fields of papers citing papers by Robert W. Maul

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert W. Maul. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert W. Maul. The network helps show where Robert W. Maul may publish in the future.

Co-authorship network of co-authors of Robert W. Maul

This figure shows the co-authorship network connecting the top 25 collaborators of Robert W. Maul. A scholar is included among the top collaborators of Robert W. Maul based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert W. Maul. Robert W. Maul is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Maul, Robert W., et al.. (2025). Optimized CUT&RUN protocol for activated primary mouse B cells. PLoS ONE. 20(4). e0322139–e0322139.
2.
Dhakal, Santosh, Han-Sol Park, Kumba Seddu, et al.. (2024). Estradiol mediates greater germinal center responses to influenza vaccination in female than male mice. mBio. 15(4). e0032624–e0032624. 14 indexed citations
3.
Thomas, Morgane, Sébastien Bender, Claire Carrion, et al.. (2023). The IgH Eµ-MAR regions promote UNG-dependent error-prone repair to optimize somatic hypermutation. Frontiers in Immunology. 14. 1030813–1030813. 2 indexed citations
4.
Maul, Robert W., et al.. (2022). Promoter Proximity Defines Mutation Window for VH and VΚ Genes Rearranged to Different J Genes. The Journal of Immunology. 208(9). 2220–2226. 2 indexed citations
5.
Park, Han-Sol, Kimberly J. Zanotti, Li Zhang, et al.. (2021). Auto-Antibody Production During Experimental Atherosclerosis in ApoE-/- Mice. Frontiers in Immunology. 12. 695220–695220. 18 indexed citations
6.
Jones, Bart G., Robert E. Sealy, Rhiannon R. Penkert, et al.. (2020). From Influenza Virus Infections to Lupus: Synchronous Estrogen Receptor α and RNA Polymerase II Binding Within the Immunoglobulin Heavy Chain Locus. Viral Immunology. 33(4). 307–315. 7 indexed citations
7.
Ragonnaud, Emeline, Kanako Moritoh, Monica Bodogai, et al.. (2019). Tumor-Derived Thymic Stromal Lymphopoietin Expands Bone Marrow B-cell Precursors in Circulation to Support Metastasis. Cancer Research. 79(22). 5826–5838. 22 indexed citations
8.
Zanotti, Kimberly J., Robert W. Maul, William Yang, & Patricia J. Gearhart. (2019). DNA Breaks in Ig V Regions Are Predominantly Single Stranded and Are Generated by UNG and MSH6 DNA Repair Pathways. The Journal of Immunology. 202(5). 1573–1581. 3 indexed citations
9.
Park, Han-Sol, et al.. (2019). B cells from young and old mice switch isotypes with equal frequencies after ex vivo stimulation. Cellular Immunology. 345. 103966–103966. 9 indexed citations
10.
Pape, Kathryn A., et al.. (2018). Naive B Cells with High-Avidity Germline-Encoded Antigen Receptors Produce Persistent IgM+ and Transient IgG+ Memory B Cells. Immunity. 48(6). 1135–1143.e4. 60 indexed citations
11.
12.
Maul, Robert W., Thomas MacCarthy, Ekaterina G. Frank, et al.. (2016). DNA polymerase ι functions in the generation of tandem mutations during somatic hypermutation of antibody genes. The Journal of Experimental Medicine. 213(9). 1675–1683. 23 indexed citations
13.
14.
Hathcock, Karen S., Hesed Padilla‐Nash, Jordi Camps, et al.. (2015). ATM deficiency promotes development of murine B-cell lymphomas that resemble diffuse large B-cell lymphoma in humans. Blood. 126(20). 2291–2301. 14 indexed citations
15.
Maul, Robert W., Huseyin Saribasak, Zheng Cao, & Patricia J. Gearhart. (2015). Topoisomerase I deficiency causes RNA polymerase II accumulation and increases AID abundance in immunoglobulin variable genes. DNA repair. 30. 46–52. 9 indexed citations
16.
Vallabhaneni, Haritha, Zhou Fang, Robert W. Maul, et al.. (2015). Defective Repair of Uracil Causes Telomere Defects in Mouse Hematopoietic Cells. Journal of Biological Chemistry. 290(9). 5502–5511. 22 indexed citations
17.
Pape, Kathryn A., Justin J. Taylor, Robert W. Maul, Patricia J. Gearhart, & Marc K. Jenkins. (2011). Different B Cell Populations Mediate Early and Late Memory During an Endogenous Immune Response. Science. 331(6021). 1203–1207. 425 indexed citations
18.
Maul, Robert W. & Patricia J. Gearhart. (2010). Controlling somatic hypermutation in immunoglobulin variable and switch regions. Immunologic Research. 47(1-3). 113–122. 25 indexed citations
19.
Maul, Robert W. & Patricia J. Gearhart. (2010). AID and Somatic Hypermutation. Advances in immunology. 105. 159–191. 155 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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