Małgorzata Piechota

1.2k total citations
16 papers, 923 citations indexed

About

Małgorzata Piechota is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Małgorzata Piechota has authored 16 papers receiving a total of 923 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Physiology and 3 papers in Cellular and Molecular Neuroscience. Recurrent topics in Małgorzata Piechota's work include Mitochondrial Function and Pathology (5 papers), Telomeres, Telomerase, and Senescence (4 papers) and Ubiquitin and proteasome pathways (3 papers). Małgorzata Piechota is often cited by papers focused on Mitochondrial Function and Pathology (5 papers), Telomeres, Telomerase, and Senescence (4 papers) and Ubiquitin and proteasome pathways (3 papers). Małgorzata Piechota collaborates with scholars based in Poland, United Kingdom and United States. Małgorzata Piechota's co-authors include Marcela Votruba, Vanessa Davies, Kathryn White, Ewa Sikora, Andrew J. Hollins, Jennifer R. Davies, Michael E. Boulton, Małgorzata Alicja Śliwińska, Piotr Sunderland and Kasia Radwańska and has published in prestigious journals such as Nucleic Acids Research, Brain and Human Molecular Genetics.

In The Last Decade

Małgorzata Piechota

16 papers receiving 920 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Małgorzata Piechota Poland 12 701 208 173 111 100 16 923
Marcel V. Alavi Germany 16 995 1.4× 159 0.8× 167 1.0× 143 1.3× 98 1.0× 25 1.2k
Jean‐Michel Griffoin France 6 1.4k 2.0× 135 0.6× 444 2.6× 143 1.3× 122 1.2× 7 1.5k
Bernard Arnaud France 14 1.5k 2.1× 132 0.6× 370 2.1× 390 3.5× 138 1.4× 23 1.8k
Sébastien Augustin France 21 778 1.1× 87 0.4× 184 1.1× 307 2.8× 50 0.5× 33 1.1k
Marta Zaninello Italy 7 555 0.8× 122 0.6× 95 0.5× 16 0.1× 152 1.5× 10 742
Kristina Klupsch United Kingdom 11 837 1.2× 228 1.1× 77 0.4× 22 0.2× 306 3.1× 14 1.3k
Jeffrey Adijanto United States 8 738 1.1× 114 0.5× 56 0.3× 205 1.8× 45 0.5× 8 887
Peter M. J. Quinn United States 16 751 1.1× 100 0.5× 21 0.1× 245 2.2× 125 1.3× 46 1.0k
Praveena Gupta United States 14 495 0.7× 648 3.1× 38 0.2× 166 1.5× 128 1.3× 43 1.1k
Vasiliki Panagiotakopoulou Germany 7 278 0.4× 130 0.6× 20 0.1× 49 0.4× 67 0.7× 7 558

Countries citing papers authored by Małgorzata Piechota

Since Specialization
Citations

This map shows the geographic impact of Małgorzata Piechota's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Małgorzata Piechota with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Małgorzata Piechota more than expected).

Fields of papers citing papers by Małgorzata Piechota

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Małgorzata Piechota. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Małgorzata Piechota. The network helps show where Małgorzata Piechota may publish in the future.

Co-authorship network of co-authors of Małgorzata Piechota

This figure shows the co-authorship network connecting the top 25 collaborators of Małgorzata Piechota. A scholar is included among the top collaborators of Małgorzata Piechota based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Małgorzata Piechota. Małgorzata Piechota is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Chikhale, Rupesh V., Małgorzata Piechota, Pradeep N. Gandhale, et al.. (2024). SARS-CoV-2 inhibitory potential of fish oil-derived 2-pyrone compounds by acquiring linoleic acid binding site on the spike protein. International Journal of Biological Macromolecules. 275(Pt 1). 133634–133634. 1 indexed citations
2.
Stefaniak, Filip, Małgorzata Piechota, Andrea Cappannini, et al.. (2024). DEGRONOPEDIA: a web server for proteome-wide inspection of degrons. Nucleic Acids Research. 52(W1). W221–W232. 12 indexed citations
3.
Piechota, Małgorzata, et al.. (2023). Lysine deserts and cullin-RING ligase receptors: Navigating untrodden paths in proteostasis. iScience. 26(11). 108344–108344. 3 indexed citations
4.
Turek, Michał, Małgorzata Piechota, Matylda Macias, et al.. (2021). Muscle‐derived exophers promote reproductive fitness. EMBO Reports. 22(8). e52071–e52071. 32 indexed citations
5.
Beroun, Anna, et al.. (2018). Generation of silent synapses in dentate gyrus correlates with development of alcohol addiction. Neuropsychopharmacology. 43(10). 1989–1999. 23 indexed citations
6.
Błasiak, Janusz, et al.. (2017). Cellular Senescence in Age‐Related Macular Degeneration: Can Autophagy and DNA Damage Response Play a Role?. Oxidative Medicine and Cellular Longevity. 2017(1). 5293258–5293258. 78 indexed citations
7.
Sunderland, Piotr, Luigi Carlessi, Alessandro Corti, et al.. (2017). Deficiency of ATM in neural cells induces markers of senescence through oxidative stress. Experimental Gerontology. 94. 114–114. 1 indexed citations
8.
Piechota, Małgorzata, Piotr Sunderland, Adrianna Wysocka, et al.. (2016). Is senescence-associated β-galactosidase a marker of neuronal senescence?. Oncotarget. 7(49). 81099–81109. 109 indexed citations
9.
Mosieniak, Grażyna, Małgorzata Alicja Śliwińska, Olga Alster, et al.. (2015). Polyploidy Formation in Doxorubicin-Treated Cancer Cells Can Favor Escape from Senescence. Neoplasia. 17(12). 882–893. 120 indexed citations
10.
Mikuła‐Pietrasik, Justyna, Patrycja Sosińska, Marek Murias, et al.. (2014). Resveratrol Derivative, 3,3′,4,4′-Tetrahydroxy-trans-Stilbene, Retards Senescence of Mesothelial Cells via Hormetic-Like Prooxidative Mechanism. The Journals of Gerontology Series A. 70(10). 1169–1180. 16 indexed citations
11.
Piechota, Małgorzata & Piotr Sunderland. (2014). [Neuronal ageing].. PubMed. 60(2). 177–86. 1 indexed citations
12.
Williams, Pete A., et al.. (2012). Opa1 is essential for retinal ganglion cell synaptic architecture and connectivity. Brain. 135(2). 493–505. 71 indexed citations
13.
White, Kathryn, Vanessa Davies, Vanessa Hogan, et al.. (2009). OPA1 Deficiency Associated with Increased Autophagy in Retinal Ganglion Cells in a Murine Model of Dominant Optic Atrophy. Investigative Ophthalmology & Visual Science. 50(6). 2567–2567. 71 indexed citations
14.
Yu‐Wai‐Man, Patrick, Vanessa Davies, Małgorzata Piechota, et al.. (2009). Secondary mtDNA Defects Do Not Cause Optic Nerve Dysfunction in a Mouse Model of Dominant Optic Atrophy. Investigative Ophthalmology & Visual Science. 50(10). 4561–4561. 17 indexed citations
15.
Powell, Kate, Kathryn White, W C Yip, et al.. (2008). A missense mutation in the murine Opa3 gene models human Costeff syndrome. Brain. 131(2). 368–380. 29 indexed citations
16.
Davies, Vanessa, Andrew J. Hollins, Małgorzata Piechota, et al.. (2007). Opa1 deficiency in a mouse model of autosomal dominant optic atrophy impairs mitochondrial morphology, optic nerve structure and visual function. Human Molecular Genetics. 16(11). 1307–1318. 339 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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