Makoto Ema

2.7k total citations
74 papers, 2.0k citations indexed

About

Makoto Ema is a scholar working on Health, Toxicology and Mutagenesis, Materials Chemistry and Cancer Research. According to data from OpenAlex, Makoto Ema has authored 74 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Health, Toxicology and Mutagenesis, 21 papers in Materials Chemistry and 11 papers in Cancer Research. Recurrent topics in Makoto Ema's work include Nanoparticles: synthesis and applications (20 papers), Effects and risks of endocrine disrupting chemicals (20 papers) and Carcinogens and Genotoxicity Assessment (11 papers). Makoto Ema is often cited by papers focused on Nanoparticles: synthesis and applications (20 papers), Effects and risks of endocrine disrupting chemicals (20 papers) and Carcinogens and Genotoxicity Assessment (11 papers). Makoto Ema collaborates with scholars based in Japan, United States and Sri Lanka. Makoto Ema's co-authors include Kazumasa Honda, Masashi Gamo, Masato Naya, Junko Nakanishi, Norihiro Kobayashi, Emiko Miyawaki, Akira Harazono, Atsuo Kishimoto, Karin Sørig Hougaard and Hirokazu Okuda and has published in prestigious journals such as Toxicology and Applied Pharmacology, Toxicology and Risk Analysis.

In The Last Decade

Makoto Ema

68 papers receiving 1.9k citations

Peers

Makoto Ema

HTM

POLLU

B. van Ravenzwaay Germany

Akio Ogata Japan

Michael G. Wolfarth United States

Tiancheng Wang China

Ali Kermanizadeh United Kingdom

Jürgen Pauluhn Germany

Lu Kong China

Brian A. Wong United States

Paul M. Hinderliter United States

Ruth Magaye Australia

B. van Ravenzwaay Germany

Makoto Ema

631 ×0.7

628 ×0.8

HTM

263 ×0.5

189 ×0.9

POLLU

164 ×0.9

Citations per year, relative to Makoto Ema Makoto Ema (= 1×) peers B. van Ravenzwaay

Countries citing papers authored by Makoto Ema

Since Specialization

Citations

This map shows the geographic impact of Makoto Ema's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Makoto Ema with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Makoto Ema more than expected).

Fields of papers citing papers by Makoto Ema

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Makoto Ema. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Makoto Ema. The network helps show where Makoto Ema may publish in the future.

Co-authorship network of co-authors of Makoto Ema

This figure shows the co-authorship network connecting the top 25 collaborators of Makoto Ema. A scholar is included among the top collaborators of Makoto Ema based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Makoto Ema. Makoto Ema is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ema, Makoto, Hirokazu Okuda, Masashi Gamo, & Kazumasa Honda. (2017). A review of reproductive and developmental toxicity of silver nanoparticles in laboratory animals. Reproductive Toxicology. 67. 149–164. 138 indexed citations

Ema, Makoto, Masashi Gamo, & Kazumasa Honda. (2015). A review of toxicity studies of single-walled carbon nanotubes in laboratory animals. Regulatory Toxicology and Pharmacology. 74. 42–63. 82 indexed citations

Ema, Makoto, Masashi Gamo, & Kazumasa Honda. (2015). Developmental toxicity of engineered nanomaterials in rodents. Toxicology and Applied Pharmacology. 299. 47–52. 37 indexed citations

Naya, Masato, Norihiro Kobayashi, Makoto Ema, et al.. (2011). In vivo genotoxicity study of titanium dioxide nanoparticles using comet assay following intratracheal instillation in rats. Regulatory Toxicology and Pharmacology. 62(1). 1–6. 51 indexed citations

Makris, Susan L., Howard M. Solomon, Kohei Shiota, et al.. (2009). Terminology of developmental abnormalities in common laboratory mammals (version 2)☆☆☆. Reproductive Toxicology. 28(3). 371–434. 55 indexed citations

Ema, Makoto, et al.. (2007). Evaluation of reproductive and developmental toxicity of the rubber accelerator N,N‐dicyclohexyl‐2‐benzothiazolesulfenamide in rats. Congenital Anomalies. 47(4). 149–155. 3 indexed citations

Ema, Makoto, et al.. (2006). Evaluation of Developmental Toxicity of Ultraviolet Absorber 2-(3′,5′-Di-tert-Butyl-2′-hydroxyphenyl)-5-Chlorobenzotriazole in Rats. Drug and Chemical Toxicology. 29(2). 215–225. 10 indexed citations

Hayashi, Makoto, Eiichi Kamata, Akihiko Hirose, et al.. (2005). In silico assessment of chemical mutagenesis in comparison with results of Salmonella microsome assay on 909 chemicals. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 588(2). 129–135. 28 indexed citations

Hirata‐Koizumi, Mutsuko, Yoshihiko Ito, Yumi Wako, et al.. (2005). Elevated susceptibility of newborn as compared with young rats to 2‐tert‐butylphenol and 2,4‐di‐tert‐butylphenol toxicity. Congenital Anomalies. 45(4). 146–153. 31 indexed citations

10.

Harazono, Akira & Makoto Ema. (2001). P11-20 Suppression of Decidual Cell Response following Administration of Dibutyltin Dichloride in Pseudopregnant Rats.. The Journal of Toxicological Sciences. 26(4). 264. 1 indexed citations

11.

Hirose, Akihiko, Eiichi Kamata, Akiyoshi Nishikawa, et al.. (2001). Toxicity Evaluation of Formaldehyde and Its Risk Assessment for Drinking Water Via Oral and Inhalation Exposure.. Journal of Japan Society on Water Environment. 24(5). 308–316. 1 indexed citations

12.

Ema, Makoto & Akira Harazono. (2000). Developmental and reproductive toxicity of tributyltin and its metabolite, dibutyltin, in rats. Congenital Anomalies. 40. 4 indexed citations

13.

Harazono, Akira & Makoto Ema. (2000). Suppression of decidual cell response induced by tributyltin chloride in pseudopregnant rats: a cause of early embryonic loss. Archives of Toxicology. 74(10). 632–637. 16 indexed citations

14.

Ema, Makoto. (1997). Effects of triphenyltin chloride on implantation and pregnancy in rats. Reproductive Toxicology. 11(2-3). 201–206. 31 indexed citations

15.

Ema, Makoto, Emiko Miyawaki, Akira Harazono, & Yoshiyuki Ogawa. (1996). "P-45 Change of Developmental Toxicity Induced by Monobenzyl Phthalate with Developmental Stages at Time of Administration.. The Journal of Toxicological Sciences. 21(5). 389. 1 indexed citations

16.

Ema, Makoto, Reiko Kurosaka, Hiro Amano, & Yoshiyuki Ogawa. (1995). Comparison of the developmental toxicity of monobutyltin, dibutyltin and tributyltin in rats. The Journal of Toxicological Sciences. 20(4). 523. 1 indexed citations

17.

Ema, Makoto, et al.. (1991). Teratogenicity of butyl benzyl phthalate in rats : Abstracts of Papers Presented at the Thirty-First Annual Meeting of the Japanese Teratology Society Izumo, Japan, July 11-12, 1991. Congenital Anomalies. 31(3). 230–231.

18.

Ema, Makoto, et al.. (1984). . Folia Pharmacologica Japonica. 83(5). 433–440. 1 indexed citations

19.

Ema, Makoto, et al.. (1983). . Folia Pharmacologica Japonica. 81(6). 469–480.

20.

Ema, Makoto, et al.. (1982). . Folia Pharmacologica Japonica. 79(5). 369–381. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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