Mahsa Motavaf
- Molecular Biology
- Epidemiology
- Hepatology top 10%
- Cancer Research
- Pharmacology top 10%
- Co-authors
- Seyed Moayed AlavianSaeid SafariFarnad ImaniXianhua PiaoMajid SadeghizadehMohammad JavanSadegh BabashahKazem Parivar
- Topics
- Hepatitis C virus research (5 papers)Neurogenesis and neuroplasticity mechanisms (4 papers)RNA Interference and Gene Delivery (3 papers)
In The Last Decade
Mahsa Motavaf
34 papers receiving 588 citations
Peers
Comparison fields: 5 of 100
- Molecular Biology 210
- Epidemiology 159
- Hepatology 113
- Cancer Research 88
- Pharmacology 54
Countries citing papers authored by Mahsa Motavaf
This map shows the geographic impact of Mahsa Motavaf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mahsa Motavaf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mahsa Motavaf more than expected).
Fields of papers citing papers by Mahsa Motavaf
This network shows the impact of papers produced by Mahsa Motavaf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mahsa Motavaf. The network helps show where Mahsa Motavaf may publish in the future.
Co-authorship network of co-authors of Mahsa Motavaf
This figure shows the co-authorship network connecting the top 25 collaborators of Mahsa Motavaf. A scholar is included among the top collaborators of Mahsa Motavaf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mahsa Motavaf. Mahsa Motavaf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 1 | |
| 3 | 7 | |
| 4 | 2 | |
| 5 | 6 | |
| 6 | 37 | |
| 7 | 8 | |
| 8 | 69 | |
| 9 | 20 | |
| 10 | 16 | |
| 11 | Mesenchymal stem cell-derived exosomes: A novel potential therapeutic avenue for cardiac regeneration. | 22 |
| 12 | 46 | |
| 13 | Therapeutic application of mesenchymal stem cell-derived exosomes: A promising cell-free therapeutic strategy in regenerative medicine. | 25 |
| 14 | 26 | |
| 15 | 50 | |
| 16 | 80 | |
| 17 | 12 | |
| 18 | 5 | |
| 19 | 3 | |
| 20 | 1 |
About Mahsa Motavaf
Mahsa Motavaf is a scholar working on Developmental Neuroscience, General Dentistry and Hepatology, having authored 34 papers that have together received 603 indexed citations. Recurring topics across this work include Hepatitis C virus research (5 papers), Neurogenesis and neuroplasticity mechanisms (4 papers) and RNA Interference and Gene Delivery (3 papers). The work is most often cited by research in Hepatology (113 citations), Developmental Neuroscience (50 citations) and Anesthesiology and Pain Medicine (36 citations). Mahsa Motavaf has collaborated with scholars based in Iran, Singapore and Hungary. Frequent co-authors include Seyed Moayed Alavian, Saeid Safari, Farnad Imani, Xianhua Piao, Majid Sadeghizadeh, Mohammad Javan, Sadegh Babashah, Kazem Parivar, Majid Sadeghizadeh and Shuo Deng. Their work appears in journals such as Photochemistry and Photobiology, Journal of Cellular and Molecular Medicine and Frontiers in Cellular Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.