Mahel Zeghouf

3.0k citations
38 papers · 2.3k indexed · 1 hit paper · h-index 23

Mahel Zeghouf

38 papers receiving 2.2k citations

Hit Papers

Regulation of Small GTPases by GEFs, GAPs, and GDIs9182013202620172021250500750

Peers

Mahel Zeghouf
Comparison fields: 5 of 103
  • Cell Biology 778
  • Molecular Biology 1.7k
  • Physiology 107
  • Oncology 323
  • Endocrinology 58
Replace Darerca Owen with:
Darerca Owen United Kingdom
J. Christopher Fromme United States
Martin D. Snider United States
Kay Oliver Schink Norway
Michel Dominguez Canada
Jeffrey H. Stack United States
Jennifer M. Kavran United States
Hideki Shibata Japan
Grant W. Booker Australia
Petra Beli Germany
Mahel Zeghouf relative to Darerca Owen United Kingdom Darerca Owen's profile →
Citations per field
00.5×1.5×2.3×
Darerca Owen · 1×
Citations per year

Countries citing papers authored by Mahel Zeghouf

Since Specialization
Citations

This map shows the geographic impact of Mahel Zeghouf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mahel Zeghouf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mahel Zeghouf more than expected).

Fields of papers citing papers by Mahel Zeghouf

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mahel Zeghouf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mahel Zeghouf. The network helps show where Mahel Zeghouf may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mahel Zeghouf, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mahel Zeghouf Line = papers co-authored together Mahel Zeghouf links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20238
2 202313
3 201924
4 201911
5 201725
6 201552
7 201428
8 201314
9 201339
10 201143
11 201037
12 201012
13 200917
14 20086
15 200782
16 200671
17 2003179
18 200031
19 19995
20 19989

About Mahel Zeghouf

Mahel Zeghouf is a scholar working on Cell Biology, Molecular Biology and Physiology, having authored 38 papers that have together received 2.3k indexed citations. Recurring topics across this work include Cellular transport and secretion (21 papers), Protein Kinase Regulation and GTPase Signaling (18 papers), Retinal Development and Disorders (6 papers), Amino Acid Enzymes and Metabolism (3 papers), Lipid Membrane Structure and Behavior (3 papers), Receptor Mechanisms and Signaling (3 papers), Enzyme Structure and Function (3 papers) and Drug Transport and Resistance Mechanisms (2 papers). The work is most often cited by research in Cell Biology (778 citations), Molecular Biology (1.7k citations) and Physiology (107 citations). Mahel Zeghouf has collaborated with scholars based in France, Canada and United States. Frequent co-authors include Jacqueline Cherfils, Jack Greenblatt, Jacques Covès, Marc Fontecave, Lori Frappier, Hong Wu, Vicki Athanasopoulos, Judy Tellam, Jean‐Christophe Zeeh and Veronica Canadien. Their work appears in journals such as Biochemical Society Transactions, Journal of Biological Chemistry, Biochemistry, Nature Communications and Small GTPases.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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