M. Wick

1.0k citations
12 papers · 857 indexed · h-index 10

M. Wick

12 papers receiving 831 citations

Peers

M. Wick
Comparison fields: 5 of 81
  • Immunology 234
  • Cancer Research 159
  • Immunology and Allergy 64
  • Oncology 243
  • Aging 15
Replace Anke Sparmann with:
Anke Sparmann Germany
Y. Jeffrey Chiang United States
Ettore Meccia Italy
K Mafune Japan
Shunsuke Kon Japan
Tommaso Zanocco‐Marani Italy
Naoko Ohtani‐Fujita Japan
Paloma García United Kingdom
Kerstin Enquist Sweden
H W Sharma United States
M. Wick relative to Anke Sparmann Germany Anke Sparmann's profile →
Citations per field
00.5×1.5×
Anke Sparmann · 1×
Citations per year

Countries citing papers authored by M. Wick

Since Specialization
Citations

This map shows the geographic impact of M. Wick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Wick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Wick more than expected).

Fields of papers citing papers by M. Wick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. Wick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Wick. The network helps show where M. Wick may publish in the future.

Co-authorship network

The 25 scholars most cited alongside M. Wick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with M. Wick Line = papers co-authored together M. Wick links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1
TLK286-induced activation of the stress response apoptotic signaling pathway
20026
2 1999229
3 1997163
4
Transgenic human DAF-expressing porcine livers: their function during hemoperfusion with human blood.
19963
5 199635
6 199556
7 199554
8 199440
9 199466
10 1994109
11 199471
12
Inhibition of Fos- and Ras-induced transformation by mutant Fos proteins with structural alterations in functionally different domains.
199225

About M. Wick

M. Wick is a scholar working on Immunology, Oncology and Cancer Research, having authored 12 papers that have together received 857 indexed citations. Recurring topics across this work include CAR-T cell therapy research (2 papers), Ubiquitin and proteasome pathways (2 papers), Immunotherapy and Immune Responses (2 papers), Protease and Inhibitor Mechanisms (2 papers), Cancer-related Molecular Pathways (2 papers), Telomeres, Telomerase, and Senescence (2 papers), Nuclear Receptors and Signaling (2 papers) and Nuclear Structure and Function (1 paper). The work is most often cited by research in Immunology (234 citations), Cancer Research (159 citations) and Immunology and Allergy (64 citations). M. Wick has collaborated with scholars based in Germany and United States. Frequent co-authors include Rolf Müller, Gretchen Hagen, Christiane Bürger, Sabine Brüsselbach, Frances C. Lucibello, Hans Schreiber, Claudia Bürger, Hartmut Koeppen, Jeffrey A. Bluestone and Patrick E. Fields. Their work appears in journals such as Journal of Cell Science, Gene, Biochemical Journal, Experimental Cell Research and Seminars in Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026