M. T. Vanier
- Physiology top 1%
- Molecular Biology top 10%
- Cell Biology top 2%
- Organic Chemistry top 5%
- Epidemiology top 10%
- Co-authors
- Lars SvennerholmJ.‐E. MånssonPeter G. PentchevHoward S. KruthMarcella ComlyEugene D. CarsteaJ D ButlerClaire Rodriguez‐Lafrasse
- Topics
- Lysosomal Storage Disorders Research (39 papers)Cellular transport and secretion (9 papers)Autoimmune and Inflammatory Disorders Research (8 papers)
- Cited by
- PhysiologyCell Biology
- Partner nations
- FranceUnited StatesCanada
In The Last Decade
M. T. Vanier
57 papers receiving 2.4k citations
Peers
Comparison fields: 5 of 105
- Physiology 1.8k
- Molecular Biology 931
- Cell Biology 610
- Organic Chemistry 572
- Epidemiology 373
Countries citing papers authored by M. T. Vanier
This map shows the geographic impact of M. T. Vanier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. T. Vanier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. T. Vanier more than expected).
Fields of papers citing papers by M. T. Vanier
This network shows the impact of papers produced by M. T. Vanier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. T. Vanier. The network helps show where M. T. Vanier may publish in the future.
Co-authorship network of co-authors of M. T. Vanier
This figure shows the co-authorship network connecting the top 25 collaborators of M. T. Vanier. A scholar is included among the top collaborators of M. T. Vanier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. T. Vanier. M. T. Vanier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 15 | |
| 2 | 8 | |
| 3 | 20 | |
| 4 | 69 | |
| 5 | 10 | |
| 6 | 43 | |
| 7 | 17 | |
| 8 | 5 | |
| 9 | 38 | |
| 10 | 38 | |
| 11 | 17 | |
| 12 | 49 | |
| 13 | Niemann-Pick disease type C: Genetic heterogeneity | 1 |
| 14 | 103 | |
| 15 | 12 | |
| 16 | 18 | |
| 17 | Prenatal diagnosis of Niemann-Pick type C disease: current strategy from an experience of 37 pregnancies at risk. | 45 |
| 18 | 20 | |
| 19 | 2 | |
| 20 | 56 |
About M. T. Vanier
M. T. Vanier is a scholar working on Physiology, Hematology and Cell Biology, having authored 57 papers that have together received 2.5k indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (39 papers), Cellular transport and secretion (9 papers) and Autoimmune and Inflammatory Disorders Research (8 papers). The work is most often cited by research in Physiology (1.8k citations), Physiology (265 citations) and Cell Biology (610 citations). M. T. Vanier has collaborated with scholars based in France, United States and Canada. Frequent co-authors include Lars Svennerholm, J.‐E. Månsson, Peter G. Pentchev, Howard S. Kruth, Marcella Comly, Eugene D. Carstea, J D Butler, Claire Rodriguez‐Lafrasse, Sylvie Duthel and David A. Wenger. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Gut.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.