M. Eppenberger

641 total citations
9 papers, 517 citations indexed

About

M. Eppenberger is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cell Biology. According to data from OpenAlex, M. Eppenberger has authored 9 papers receiving a total of 517 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Cardiology and Cardiovascular Medicine and 3 papers in Cell Biology. Recurrent topics in M. Eppenberger's work include Cardiomyopathy and Myosin Studies (4 papers), Muscle Physiology and Disorders (3 papers) and Hemoglobin structure and function (2 papers). M. Eppenberger is often cited by papers focused on Cardiomyopathy and Myosin Studies (4 papers), Muscle Physiology and Disorders (3 papers) and Hemoglobin structure and function (2 papers). M. Eppenberger collaborates with scholars based in Switzerland, United States and France. M. Eppenberger's co-authors include Hans M. Eppenberger, R. Richterich, H. Aebi, Colleen Nixon, F. Homburger, J. Baker, Nathan O. Kaplan, David M. Dawson, Claude A. Dechesne and Thomas Baechi and has published in prestigious journals such as Nature, Annals of the New York Academy of Sciences and Developmental Biology.

In The Last Decade

M. Eppenberger

9 papers receiving 467 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
M. Eppenberger Switzerland	7	298	172	143	73	62	9	517
D Pette Germany	10	198 0.7×	80 0.5×	60 0.4×	55 0.8×	83 1.3×	19	410
Claus Heilmann Germany	10	568 1.9×	127 0.7×	450 3.1×	104 1.4×	73 1.2×	13	841
Y.H. Lau United States	10	458 1.5×	149 0.9×	113 0.8×	139 1.9×	129 2.1×	11	586
Natalie Cambon United States	8	258 0.9×	71 0.4×	173 1.2×	37 0.5×	38 0.6×	11	433
H. Misawa Japan	14	369 1.2×	98 0.6×	81 0.6×	96 1.3×	45 0.7×	17	617
Monika Eppenberger-Eberhardt Switzerland	11	531 1.8×	110 0.6×	364 2.5×	49 0.7×	88 1.4×	13	811
Petra Wetzel Germany	14	412 1.4×	84 0.5×	73 0.5×	55 0.8×	119 1.9×	25	538
J A Lash United States	5	220 0.7×	113 0.7×	129 0.9×	44 0.6×	55 0.9×	5	346
Joan Gannon Ireland	15	550 1.8×	310 1.8×	91 0.6×	63 0.9×	219 3.5×	20	762
K Iizuka Japan	7	519 1.7×	131 0.8×	132 0.9×	68 0.9×	195 3.1×	13	718

Countries citing papers authored by M. Eppenberger

Since Specialization

Citations

This map shows the geographic impact of M. Eppenberger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Eppenberger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Eppenberger more than expected).

Fields of papers citing papers by M. Eppenberger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. Eppenberger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Eppenberger. The network helps show where M. Eppenberger may publish in the future.

Co-authorship network of co-authors of M. Eppenberger

This figure shows the co-authorship network connecting the top 25 collaborators of M. Eppenberger. A scholar is included among the top collaborators of M. Eppenberger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. Eppenberger. M. Eppenberger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Eppenberger, M., et al.. (1988). Immunocytochemical analysis of the regeneration of myofibrils in long-term cultures of adult cardiomyocytes of the rat. Developmental Biology. 130(1). 1–15. 81 indexed citations

2.

Eppenberger, M., et al.. (1987). Myofibril formation in longterm-cultures of adult rat heart cells.. PubMed. 46(8-9). S640–5. 6 indexed citations

3.

Eppenberger, M., et al.. (1984). Myofibrillar M‐line structure in normal and dystrophic hamster muscle. Muscle & Nerve. 7(4). 304–311. 4 indexed citations

4.

Dawson, David M., Hans M. Eppenberger, & M. Eppenberger. (1968). MULTIPLE MOLECULAR FORMS OF CREATINE KINASES*. Annals of the New York Academy of Sciences. 151(1). 616–626. 48 indexed citations

5.

Eppenberger, M., Hans M. Eppenberger, & Nathan O. Kaplan. (1967). Evolution of Creatine Kinase. Nature. 214(5085). 239–241. 54 indexed citations

6.

Homburger, F., Colleen Nixon, M. Eppenberger, & J. Baker. (1966). HEREDITARY MYOPATHY IN THE SYRIAN HAMSTER: STUDIES ON PATHOGENESIS*. Annals of the New York Academy of Sciences. 138(1). 14–27. 70 indexed citations

7.

Homburger, F., et al.. (1965). Susceptibility of certain inbred strains of hamsters to teratogenic effects of thalidomide. Toxicology and Applied Pharmacology. 7(5). 686–693. 18 indexed citations

8.

Eppenberger, M., Colleen Nixon, J. Baker, & F. Homburger. (1964). Serum Phosphocreatine Kinase in Hereditary Muscular Dystrophy and Cardiac Necrosis of Syrian Golden Hamsters.. Experimental Biology and Medicine. 117(2). 465–468. 13 indexed citations

9.

Eppenberger, Hans M., M. Eppenberger, R. Richterich, & H. Aebi. (1964). The ontogeny of creatine kinase isozymes. Developmental Biology. 10(1). 1–16. 223 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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