M Brydson

606 total citations
12 papers, 481 citations indexed

About

M Brydson is a scholar working on Molecular Biology, Neurology and Cell Biology. According to data from OpenAlex, M Brydson has authored 12 papers receiving a total of 481 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Neurology and 4 papers in Cell Biology. Recurrent topics in M Brydson's work include Ion channel regulation and function (7 papers), Myasthenia Gravis and Thymoma (5 papers) and Nicotinic Acetylcholine Receptors Study (4 papers). M Brydson is often cited by papers focused on Ion channel regulation and function (7 papers), Myasthenia Gravis and Thymoma (5 papers) and Nicotinic Acetylcholine Receptors Study (4 papers). M Brydson collaborates with scholars based in United Kingdom, India and France. M Brydson's co-authors include David Beeson, John Newsom–Davis, Angela Vincent, Chris Shelley, C. J. Hatton, David Colquhoun, R Croxen, Richard Webster, Sharon Brownlow and Brian Neville and has published in prestigious journals such as Nucleic Acids Research, Journal of Clinical Investigation and Neurology.

In The Last Decade

M Brydson

12 papers receiving 469 citations

Peers

Countries citing papers authored by M Brydson

Since Specialization

Citations

This map shows the geographic impact of M Brydson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M Brydson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M Brydson more than expected).

Fields of papers citing papers by M Brydson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M Brydson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M Brydson. The network helps show where M Brydson may publish in the future.

Co-authorship network of co-authors of M Brydson

This figure shows the co-authorship network connecting the top 25 collaborators of M Brydson. A scholar is included among the top collaborators of M Brydson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M Brydson. M Brydson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

#	Work	Indexed citations
1	Mutation in the AChR ion channel gate underlies a fast channel congenital myasthenic syndrome 2004 ·Neurology ·Richard Webster, M Brydson, R Croxen, John Newsom–Davis, Angela Vincent, David Beeson	26
2	Properties of the human muscle nicotinic receptor, and of the slow-channel myasthenic syndrome mutant L221F, inferred from maximum likelihood fits 2003 ·The Journal of Physiology ·C. J. Hatton, Chris Shelley, M Brydson, David Beeson, David Colquhoun	62
3	Structural Abnormalities of the AChR Caused by Mutations Underlying Congenital Myasthenic Syndromes 2003 ·Annals of the New York Academy of Sciences ·David Beeson, Richard Webster, John Ealing, R Croxen, Sharon Brownlow, M Brydson, John Newsom–Davis, Clarke R. Slater, C. J. Hatton, Chris Shelley, David Colquhoun, Angela Vincent	9
4	Properties of the human muscle nicotinic receptor, and of the slow-channel myasthenic syndrome mutant epsilonL221F, inferred from maximum likelihood fits 2003 ·The Journal of Physiology ·C. J. Hatton, Chris Shelley, M Brydson, David Beeson, David Colquhoun	74
5	Recessive inheritance and variable penetrance of slow-channel congenital myasthenic syndromes 2002 ·Neurology ·R Croxen, C. J. Hatton, Chris Shelley, M Brydson, G Chauplannaz, Harry Oosterhuis, Angela Vincent, John Newsom–Davis, David Colquhoun, David Beeson	60
6	Acetylcholine receptor δ subunit mutations underlie a fast-channel myasthenic syndrome and arthrogryposis multiplex congenita 2001 ·Journal of Clinical Investigation ·Sharon Brownlow, Richard Webster, R Croxen, M Brydson, Brian Neville, Jean‐Pierre Lin, Angela Vincent, John Newsom–Davis, David Beeson	60
7	Acetylcholine receptor δ subunit mutations underlie a fast-channel myasthenic syndrome and arthrogryposis multiplex congenita 2001 ·Journal of Clinical Investigation ·Sharon Brownlow, Richard Webster, R Croxen, M Brydson, Brian Neville, Jean‐Pierre Lin, Angela Vincent, John Newsom–Davis, David Beeson	58
8	cDNA and Genomic Clones Encoding the Human Muscle Acetylcholine Receptor 1993 ·Annals of the New York Academy of Sciences ·David Beeson, Angela Vincent, (unknown), M Brydson, Leslie Jacobson, Fulvio Baggi, S.J. Jeremiah, Sue Povey, John Newsom–Davis	6
9	Primary structure of the human muscle acetylcholine receptor 1993 ·European Journal of Biochemistry ·David Beeson, M Brydson, Maria Betty, Steve Jeremiah, (unknown), Angela Vincent, John Newsom–Davis	69
10	PRIMARY STRUCTURE OF THE HUMAN MUSCLE ACETYLCHOLINE-RECEPTOR - CDNA CLONING OF THE GAMMA-SUBUNIT AND EPSILON-SUBUNIT 1993 ·UCL Discovery (University College London) ·David Beeson, M Brydson, Maria Betty, S.J. Jeremiah, S. Povey, Angela Vincent, John Newsom–Davis	6
11	Nucleotide sequence of human muscle acetylcholine receptor β-subunit 1989 ·Nucleic Acids Research ·David Beeson, M Brydson, John Newsom–Davis	34
12	Human muscle acetylcholine receptor: cloning and expression in Escherichia coli of cDNA for the α-subunit 1989 ·Biochemical Society Transactions ·(unknown), M Brydson, (unknown), Angela Vincent, John Newsom–Davis	17

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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