Lucie Dupuis

2.6k citations
28 papers · 554 indexed · h-index 13
Topics
Genomic variations and chromosomal abnormalities (7 papers)Genetic Syndromes and Imprinting (5 papers)Connective tissue disorders research (5 papers)

In The Last Decade

Lucie Dupuis

28 papers receiving 548 citations

Peers

Lucie Dupuis
Comparison fields: 5 of 69
  • Genetics 335
  • Molecular Biology 243
  • Cell Biology 92
  • Rheumatology 88
  • Cancer Research 51
Replace Meena Balasubramanian with:
Meena Balasubramanian United Kingdom
Pelin Özlem Şimşek‐Kiper Türkiye
Yasutsugu Chinen Japan
Nisha Patel Saudi Arabia
Fulya Taylan Sweden
Luitgard Graul‐Neumann Germany
Véronique Geoffroy France
Muneera Alshammari Saudi Arabia
Hyon J. Kim South Korea
Pauline Terhal Netherlands
Lucie Dupuis relative to Meena Balasubramanian United Kingdom Meena Balasubramanian's profile →
Citations per field
00.5×1.7×
Meena Balasubramanian · 1×
Citations per year

Countries citing papers authored by Lucie Dupuis

Since Specialization
Citations

This map shows the geographic impact of Lucie Dupuis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lucie Dupuis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lucie Dupuis more than expected).

Fields of papers citing papers by Lucie Dupuis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lucie Dupuis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lucie Dupuis. The network helps show where Lucie Dupuis may publish in the future.

Co-authorship network of co-authors of Lucie Dupuis

This figure shows the co-authorship network connecting the top 25 collaborators of Lucie Dupuis. A scholar is included among the top collaborators of Lucie Dupuis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lucie Dupuis. Lucie Dupuis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 7
2 4
3 1
4 2
5 65
6 95
7 14
8 1
9 62
10 19
11 33
12 42
13 10
14 25
15 19
16 27
17 16
18 3
19 9
20 47

About Lucie Dupuis

Lucie Dupuis is a scholar working on Genetics, Cell Biology and Molecular Biology, having authored 28 papers that have together received 554 indexed citations. Recurring topics across this work include Genomic variations and chromosomal abnormalities (7 papers), Genetic Syndromes and Imprinting (5 papers) and Connective tissue disorders research (5 papers). The work is most often cited by research in Genetics (335 citations), Rheumatology (88 citations) and Cell Biology (92 citations). Lucie Dupuis has collaborated with scholars based in Canada, United States and Australia. Frequent co-authors include Roberto Mendoza‐Londono, Stephen W. Scherer, Christian R. Marshall, Dimitri J. Stavropoulos, Pekka Kannus, Tracy Stockley, Anath C. Lionel, Rebekah Jobling, Andrew Howard and Abdul Noor. Their work appears in journals such as Neurology, The American Journal of Human Genetics and Human Molecular Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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