Lovise Mæhle

6.2k total citations
61 papers, 1.6k citations indexed

About

Lovise Mæhle is a scholar working on Genetics, Cancer Research and Pathology and Forensic Medicine. According to data from OpenAlex, Lovise Mæhle has authored 61 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Genetics, 24 papers in Cancer Research and 14 papers in Pathology and Forensic Medicine. Recurrent topics in Lovise Mæhle's work include BRCA gene mutations in cancer (43 papers), Cancer Genomics and Diagnostics (22 papers) and Genomic variations and chromosomal abnormalities (19 papers). Lovise Mæhle is often cited by papers focused on BRCA gene mutations in cancer (43 papers), Cancer Genomics and Diagnostics (22 papers) and Genomic variations and chromosomal abnormalities (19 papers). Lovise Mæhle collaborates with scholars based in Norway, United Kingdom and Netherlands. Lovise Mæhle's co-authors include Pål Møller, Jaran Apold, Ketil Heimdal, Anne Irene Hagen, Neal Clark, Eli Marie Grindedal, Anita Vabø, Astrid Stormorken, D. Gareth Evans and Eivind Hovig and has published in prestigious journals such as PLoS ONE, Clinical Cancer Research and Environmental Health Perspectives.

In The Last Decade

Lovise Mæhle

59 papers receiving 1.6k citations

Peers

Lovise Mæhle

GENET

ONCOL

PFM

Rita K. Schmutzler Germany

L. Phuong United States

Orland Dı́ez Spain

Barbara Weber United States

Mary B. Daly United States

Leigha Senter United States

E. von Schoultz Sweden

Joanne L. Blum United States

Nora Wong Canada

Anne Irene Hagen Norway

Rita K. Schmutzler Germany

Lovise Mæhle

1.2k ×1.3

GENET

626 ×1.2

438 ×0.9

ONCOL

493 ×1.1

PFM

523 ×1.2

Citations per year, relative to Lovise Mæhle Lovise Mæhle (= 1×) peers Rita K. Schmutzler

Countries citing papers authored by Lovise Mæhle

Since Specialization

Citations

This map shows the geographic impact of Lovise Mæhle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lovise Mæhle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lovise Mæhle more than expected).

Fields of papers citing papers by Lovise Mæhle

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lovise Mæhle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lovise Mæhle. The network helps show where Lovise Mæhle may publish in the future.

Co-authorship network of co-authors of Lovise Mæhle

This figure shows the co-authorship network connecting the top 25 collaborators of Lovise Mæhle. A scholar is included among the top collaborators of Lovise Mæhle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lovise Mæhle. Lovise Mæhle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wangensteen, Teresia, et al.. (2018). BRCA1 and BRCA2 mutation spectrum – an update on mutation distribution in a large cancer genetics clinic in Norway. Hereditary Cancer in Clinical Practice. 16(1). 3–3. 28 indexed citations

Akslen, Lars A., Ellen Schlichting, Turid Aas, et al.. (2018). Trends in Diagnostics, Surgical Treatment, and Prognostic Factors for Outcomes in Medullary Thyroid Carcinoma in Norway: A Nationwide Population-Based Study. European Thyroid Journal. 8(1). 31–40. 32 indexed citations

Stensland, Hilde Monica Frostad Riise, et al.. (2016). Characterization of BRCA1 and BRCA2 variants found in a Norwegian breast or ovarian cancer cohort. Familial Cancer. 16(1). 1–16. 11 indexed citations

Ekstrøm, Per Olaf, et al.. (2015). Ten modifiers of BRCA1 penetrance validated in a Norwegian series. Hereditary Cancer in Clinical Practice. 13(1). 14–14. 3 indexed citations

Møller, Pål, et al.. (2012). Age‐specific incidence rates for breast cancer in carriers of BRCA1 mutations from Norway. Clinical Genetics. 83(1). 88–91. 13 indexed citations

Møller, Pål, Neal Clark, & Lovise Mæhle. (2011). A simplified method for segregation analysis (SISA) to determine penetrance and expression of a genetic variant in a family. Human Mutation. 32(5). 568–571. 14 indexed citations

Sjursen, Wenche, Bjørn Ivar Haukanes, Eli Marie Grindedal, et al.. (2010). Current clinical criteria for Lynch syndrome are not sensitive enough to identify MSH6 mutation carriers. Journal of Medical Genetics. 47(9). 579–585. 60 indexed citations

Hagen, Anne Irene, et al.. (2009). Survival in Norwegian BRCA1 mutation carriers with breast cancer. Hereditary Cancer in Clinical Practice. 7(1). 7–7. 4 indexed citations

Grindedal, Eli Marie, Laura Renkonen‐Sinisalo, Hans F. A. Vasen, et al.. (2009). Survival in women with MMR mutations and ovarian cancer: a multicentre study in Lynch syndrome kindreds. Journal of Medical Genetics. 47(2). 99–102. 57 indexed citations

10.

Grindedal, Eli Marie, Ignacio Blanco, Astrid Stormorken, et al.. (2008). High risk of endometrial cancer in colorectal cancer kindred is pathognomonic for MMR-mutation carriers. Familial Cancer. 8(2). 145–151. 11 indexed citations

11.

Rustad, Cecilie F., Merete Bjørnslett, Ketil Heimdal, et al.. (2006). Germline PTEN mutations are rare and highly penetrant. Hereditary Cancer in Clinical Practice. 4(4). 177–177. 12 indexed citations

12.

Stormorken, Astrid, Neal Clark, Eli Marie Grindedal, Lovise Mæhle, & Pål Møller. (2006). Prevention of colorectal cancer by colonoscopic surveillance in families with hereditary colorectal cancer. Scandinavian Journal of Gastroenterology. 42(5). 611–617. 11 indexed citations

13.

Geirdal, Amy Østertun, Jon Gerhard Reichelt, Alv A. Dahl, et al.. (2005). Psychological distress in women at risk of hereditary breast/ovarian or HNPCC cancers in the absence of demonstrated mutations. Familial Cancer. 4(2). 121–126. 35 indexed citations

14.

Heimdal, Ketil, Lovise Mæhle, Jaran Apold, Jan Pedersen, & Pål Møller. (2003). The Norwegian founder mutations in BRCA1: high penetrance confirmed in an incident cancer series and differences observed in the risk of ovarian cancer. European Journal of Cancer. 39(15). 2205–2213. 33 indexed citations

15.

Møller, Pål, Åke Borg, D. Gareth Evans, et al.. (2002). Survival in prospectively ascertained familial breast cancer: Analysis of a series stratified by tumour characteristics, BRCA mutations and oophorectomy. International Journal of Cancer. 101(6). 555–559. 88 indexed citations

16.

Møller, Pål, Åke Borg, Ketil Heimdal, et al.. (2001). The BRCA1 syndrome and other inherited breast or breast–ovarian cancers in a Norwegian prospective series. European Journal of Cancer. 37(8). 1027–1032. 24 indexed citations

17.

Møller, Pål, Jaran Apold, Lovise Mæhle, & Ketil Heimdal. (2000). [European guidelines for health care in hereditary breast cancer].. PubMed. 120(6). 726–7.

18.

Heimdal, Ketil, Lovise Mæhle, & Pål Møller. (1999). Costs and Benefits of Diagnosing Familial Breast Cancer. Disease Markers. 15(1-3). 167–173. 22 indexed citations

19.

Dørum, Anne, Pål Møller, Erik-Jan Kamsteeg, et al.. (1997). A BRCA1 founder mutation, identified with haplotype analysis, allowing genotype/phenotype determination and predictive testing. European Journal of Cancer. 33(14). 2390–2392. 21 indexed citations

20.

Haugen, Aage, Lovise Mæhle, Sarah Mollerup, Edgar Rivedal, & David Ryberg. (1994). Nickel-induced alterations in human renal epithelial cells.. Environmental Health Perspectives. 102(suppl 3). 117–118. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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