Lothar Rössig

6.1k total citations · 2 hit papers
39 papers, 5.1k citations indexed

About

Lothar Rössig is a scholar working on Molecular Biology, Cancer Research and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Lothar Rössig has authored 39 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 9 papers in Cancer Research and 7 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Lothar Rössig's work include Angiogenesis and VEGF in Cancer (14 papers), Histone Deacetylase Inhibitors Research (5 papers) and Nitric Oxide and Endothelin Effects (4 papers). Lothar Rössig is often cited by papers focused on Angiogenesis and VEGF in Cancer (14 papers), Histone Deacetylase Inhibitors Research (5 papers) and Nitric Oxide and Endothelin Effects (4 papers). Lothar Rössig collaborates with scholars based in Germany, United States and United Kingdom. Lothar Rössig's co-authors include Stefanie Dimmeler, Andreas M. Zeiher, Judith Haendeler, Carmen Urbich, Mariuca Vasa, Stephan Fichtlscherer, Caroline Schmidt‐Lucke, Alexandra Aicher, Martina Britten and Cornel Badorff and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Lothar Rössig

39 papers receiving 5.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Reduced Number of Circulating Endothelial Progenitor Cells Predicts Future Cardiovascular Events

2005 866 citations Caroline Schmidt‐Lucke, Lothar Rössig et al. profile →
SIRT1 controls endothelial angiogenic functions during vascular growth

2007 508 citations Michael Potente, Danila Baldessari et al. Genes & Development profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Lothar Rössig Germany	26	3.1k	829	825	820	738	39	5.1k
Goutam Ghosh Choudhury United States	47	3.9k 1.2×	1.2k 1.5×	784 1.0×	791 1.0×	768 1.0×	145	6.3k
Nichola Figg United Kingdom	36	2.6k 0.8×	643 0.8×	407 0.5×	727 0.9×	697 0.9×	67	5.6k
Toshihiro Ichiki Japan	46	3.4k 1.1×	565 0.7×	414 0.5×	1.1k 1.3×	842 1.1×	111	7.4k
Tetsuo Shioi Japan	40	3.8k 1.2×	473 0.6×	480 0.6×	867 1.1×	961 1.3×	73	7.0k
Yves Gorin United States	41	2.4k 0.8×	462 0.6×	361 0.4×	1.3k 1.6×	527 0.7×	61	5.8k
Hou‐Zao Chen China	42	2.7k 0.8×	905 1.1×	415 0.5×	1.2k 1.4×	451 0.6×	109	5.7k
Seijiro Mori Japan	31	2.5k 0.8×	319 0.4×	567 0.7×	810 1.0×	540 0.7×	137	4.5k
Haruhiro Toko Japan	38	3.5k 1.1×	473 0.6×	525 0.6×	795 1.0×	1.3k 1.8×	71	6.0k
Toshiyuki Sasaguri Japan	39	2.6k 0.8×	423 0.5×	644 0.8×	485 0.6×	414 0.6×	141	4.6k
Guy Eelen Belgium	38	2.4k 0.8×	1.2k 1.4×	571 0.7×	569 0.7×	370 0.5×	64	5.1k

Countries citing papers authored by Lothar Rössig

Since Specialization

Citations

This map shows the geographic impact of Lothar Rössig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lothar Rössig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lothar Rössig more than expected).

Fields of papers citing papers by Lothar Rössig

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lothar Rössig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lothar Rössig. The network helps show where Lothar Rössig may publish in the future.

Co-authorship network of co-authors of Lothar Rössig

This figure shows the co-authorship network connecting the top 25 collaborators of Lothar Rössig. A scholar is included among the top collaborators of Lothar Rössig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lothar Rössig. Lothar Rössig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Rao, Vishal N., Derek D. Cyr, Lisa Wruck, et al.. (2023). Electronic health record characterization and outcomes of heart failure with preserved ejection fraction. American Heart Journal. 263. 1–14. 1 indexed citations

Kaluza, David, Jens Krøll, Sabine Gesierich, et al.. (2011). Class IIb HDAC6 regulates endothelial cell migration and angiogenesis by deacetylation of cortactin. The EMBO Journal. 30(20). 4142–4156. 166 indexed citations

Schmidt‐Lucke, Caroline, et al.. (2010). Improvement of endothelial damage and regeneration indexes in patients with coronary artery disease after 4 weeks of statin therapy. Atherosclerosis. 211(1). 249–254. 39 indexed citations

Urbich, Carmen, Ayesha I. De Souza, Lothar Rössig, et al.. (2010). Proteomic characterization of human early pro-angiogenic cells. Journal of Molecular and Cellular Cardiology. 50(2). 333–336. 32 indexed citations

Rössig, Lothar, Sabine Genth‐Zotz, Matthias Rau, et al.. (2010). Argatroban for elective percutaneous coronary intervention: The ARG-E04 multi-center study. International Journal of Cardiology. 148(2). 214–219. 19 indexed citations

Potente, Michael, Danila Baldessari, Raúl Mostoslavsky, et al.. (2007). SIRT1 controls endothelial angiogenic functions during vascular growth. Genes & Development. 21(20). 2644–2658. 508 indexed citations breakdown →

Fichtlscherer, Stephan, Caroline Schmidt‐Lucke, Lothar Rössig, et al.. (2006). Differential effects of short-term lipid lowering with ezetimibe and statins on endothelial function in patients with CAD: clinical evidence for ‘pleiotropic’ functions of statin therapy. European Heart Journal. 27(10). 1182–1190. 115 indexed citations

Rössig, Lothar, Carmen Urbich, Thomas Brühl, et al.. (2005). Histone deacetylase activity is essential for the expression of HoxA9 and for endothelial commitment of progenitor cells. The Journal of Experimental Medicine. 201(11). 1825–1835. 140 indexed citations

Rössig, Lothar, Stephan Fichtlscherer, Christopher Heeschen, et al.. (2004). The pro-apoptotic serum activity is an independent mortality predictor of patients with heart failure. European Heart Journal. 25(18). 1620–1625. 16 indexed citations

10.

Heeschen, Christopher, Alexandra Aicher, Judith Haendeler, et al.. (2004). p21 ^Cip1 Levels Differentially Regulate Turnover of Mature Endothelial Cells, Endothelial Progenitor Cells, and In Vivo Neovascularization. Circulation Research. 94(5). 686–692. 33 indexed citations

11.

Rössig, Lothar, Corinna Hermann, Judith Haendeler, et al.. (2002). Angiotensin II-induced upregulation of MAP kinase phosphatase-3 mRNA levels mediates endothelial cell apotosis. Basic Research in Cardiology. 97(1). 1–8. 25 indexed citations

12.

Rössig, Lothar, Huige Li, Beate Fißlthaler, et al.. (2002). Inhibitors of Histone Deacetylation Downregulate the Expression of Endothelial Nitric Oxide Synthase and Compromise Endothelial Cell Function in Vasorelaxation and Angiogenesis. Circulation Research. 91(9). 837–844. 173 indexed citations

13.

Rössig, Lothar, Stefanie Dimmeler, & Andreas M. Zeiher. (2001). Apoptosis in the vascular wall and atherosclerosis. Basic Research in Cardiology. 96(1). 11–22. 129 indexed citations

14.

Rössig, Lothar, Judith Haendeler, Ziad Mallat, et al.. (2000). Congestive heart failure induces endothelial cell apoptosis: protective role of carvedilol. Journal of the American College of Cardiology. 36(7). 2081–2089. 118 indexed citations

15.

Rössig, Lothar, Birgit Fichtlscherer, Kristin Breitschopf, et al.. (1999). Nitric Oxide Inhibits Caspase-3 by S-Nitrosationin Vivo. Journal of Biological Chemistry. 274(11). 6823–6826. 363 indexed citations

16.

Schöfl, Christof, et al.. (1996). CYCLIC adenosine 3',5'-monophosphate potentiates Ca2+ signaling and insulin secretion by phospholipase C-linked hormones in HIT cells.. Endocrinology. 137(7). 3026–3032. 9 indexed citations

17.

Schöfl, Christof, et al.. (1996). Generation of repetitive Ca2+ transients by bombesin requires intracellular release and influx of Ca2+ through voltage-dependent and voltage independent channels in single HIT cells. Cell Calcium. 19(6). 485–493. 19 indexed citations

18.

Schöfl, Christof, et al.. (1995). Extracellular ATP and UTP Increase Cytosolic Free Calcium by Activating a Common P2U-Receptor in Single Human Thyrocytes. Biochemical and Biophysical Research Communications. 213(3). 928–934. 22 indexed citations

19.

Schöfl, Christof, et al.. (1995). Extracellular Nucleotides Increase Cytosolic Free Calcium by Activating P2u-Receptors in Single Human Gastric Mucous Cells. Biochemical and Biophysical Research Communications. 216(2). 636–641. 8 indexed citations

20.

Schwaninger, Markus, Christof Schöfl, R Blume, Lothar Rössig, & W Knepel. (1995). Inhibition by antidepressant drugs of cyclic AMP response element-binding protein/cyclic AMP response element-directed gene transcription.. Molecular Pharmacology. 47(6). 1112–1118. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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