Hou‐Zao Chen

8.6k total citations · 1 hit paper
109 papers, 5.7k citations indexed

About

Hou‐Zao Chen is a scholar working on Molecular Biology, Geriatrics and Gerontology and Physiology. According to data from OpenAlex, Hou‐Zao Chen has authored 109 papers receiving a total of 5.7k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Molecular Biology, 37 papers in Geriatrics and Gerontology and 18 papers in Physiology. Recurrent topics in Hou‐Zao Chen's work include Sirtuins and Resveratrol in Medicine (37 papers), Adipose Tissue and Metabolism (11 papers) and Mitochondrial Function and Pathology (10 papers). Hou‐Zao Chen is often cited by papers focused on Sirtuins and Resveratrol in Medicine (37 papers), Adipose Tissue and Metabolism (11 papers) and Mitochondrial Function and Pathology (10 papers). Hou‐Zao Chen collaborates with scholars based in China, United States and Hong Kong. Hou‐Zao Chen's co-authors include De‐Pei Liu, Xiaoqiang Tang, Ran Zhang, Chih‐Chuan Liang, Zhu‐Qin Zhang, Shuang Zhou, De‐Long Hao, Hua Cai, Yuxuan Luo and Peng Gao and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Hou‐Zao Chen

106 papers receiving 5.7k citations

Hit Papers

Loss of Cardiac Ferritin ... 2020 2026 2022 2024 2020 100 200 300 400 500

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Hou‐Zao Chen 2.7k 1.3k 1.2k 905 803 109 5.7k
Keizo Kanasaki 2.4k 0.9× 648 0.5× 829 0.7× 747 0.8× 526 0.7× 143 6.6k
Yasuhiro Maejima 3.0k 1.1× 644 0.5× 934 0.8× 467 0.5× 1.3k 1.7× 121 6.2k
Ming‐Hui Zou 3.1k 1.2× 335 0.3× 1.8k 1.5× 633 0.7× 920 1.1× 82 6.2k
Toshiro Sugimoto 2.1k 0.8× 473 0.4× 843 0.7× 360 0.4× 607 0.8× 100 5.3k
Peiyong Zhai 5.5k 2.1× 1.3k 1.0× 1.8k 1.5× 672 0.7× 2.0k 2.5× 97 10.0k
Mahesh P. Gupta 4.0k 1.5× 2.7k 2.1× 1.8k 1.5× 500 0.6× 1.4k 1.7× 119 7.6k
Hiroyasu Yamamoto 3.0k 1.1× 1.6k 1.2× 2.0k 1.7× 503 0.6× 324 0.4× 61 7.0k
Nichola Figg 2.6k 1.0× 229 0.2× 727 0.6× 643 0.7× 628 0.8× 67 5.6k
Lorrie A. Kirshenbaum 4.4k 1.6× 325 0.3× 837 0.7× 989 1.1× 2.1k 2.6× 124 7.4k
Xiaoqiang Tang 1.5k 0.6× 527 0.4× 653 0.6× 300 0.3× 402 0.5× 53 3.1k

Countries citing papers authored by Hou‐Zao Chen

Since Specialization
Citations

This map shows the geographic impact of Hou‐Zao Chen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hou‐Zao Chen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hou‐Zao Chen more than expected).

Fields of papers citing papers by Hou‐Zao Chen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hou‐Zao Chen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hou‐Zao Chen. The network helps show where Hou‐Zao Chen may publish in the future.

Co-authorship network of co-authors of Hou‐Zao Chen

This figure shows the co-authorship network connecting the top 25 collaborators of Hou‐Zao Chen. A scholar is included among the top collaborators of Hou‐Zao Chen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hou‐Zao Chen. Hou‐Zao Chen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Yu, Hou‐Zao Chen, & Xiaoqiang Tang. (2025). Crotonyl-coenzyme A (crotonyl-CoA). Trends in Endocrinology and Metabolism. 36(12). 1154–1155.
2.
Lv, Cheng, Xiao Xiao, Fei Wang, et al.. (2025). Somatic NAP1L1 p.D349E promotes cardiac hypertrophy through cGAS-STING-IFN signaling. Nature Communications. 16(1). 3140–3140. 1 indexed citations
3.
5.
Zhang, Peng, Lele Liu, Xiang Zhao, et al.. (2024). Enhancing homology-directed repair efficiency with HDR-boosting modular ssDNA donor. Nature Communications. 15(1). 6843–6843. 11 indexed citations
6.
Zhang, Lei, Menglin Zhang, Jinlong Huang, et al.. (2024). Klf9 is essential for cardiac mitochondrial homeostasis. Nature Cardiovascular Research. 3(11). 1318–1336. 4 indexed citations
7.
Guo, Ziwei, Qing Wan, De‐Pei Liu, et al.. (2024). Disruption of PCSK9 Suppresses Inflammation and Attenuates Abdominal Aortic Aneurysm Formation. Arteriosclerosis Thrombosis and Vascular Biology. 45(1). e1–e14. 7 indexed citations
8.
Gong, Ke, Chao Xue, Mengyao Wang, et al.. (2024). Intestinal Nogo-B reduces GLP1 levels by binding to proglucagon on the endoplasmic reticulum to inhibit PCSK1 cleavage. Nature Communications. 15(1). 6845–6845. 2 indexed citations
9.
Cao, Jun, Ziyu Wei, Yu Nie, & Hou‐Zao Chen. (2024). Therapeutic potential of alternative splicing in cardiovascular diseases. EBioMedicine. 101. 104995–104995. 10 indexed citations
10.
Dong, Tingting, Weilong Zhang, Zhaohui Zhu, et al.. (2023). Mic60 is essential to maintain mitochondrial integrity and to prevent encephalomyopathy. Brain Pathology. 33(4). e13157–e13157. 6 indexed citations
11.
Zhang, Yang, Xiaoman Wang, Heping Wang, et al.. (2023). Sirtuin 2 deficiency aggravates ageing-induced vascular remodelling in humans and mice. European Heart Journal. 44(29). 2746–2759. 49 indexed citations
12.
Ding, Yang-Nan, Tingting Wang, Xiaoqiang Tang, et al.. (2023). SIRT6 is an epigenetic repressor of thoracic aortic aneurysms via inhibiting inflammation and senescence. Signal Transduction and Targeted Therapy. 8(1). 255–255. 24 indexed citations
13.
Song, Shen, Qianqian Yin, Bin Zhou, et al.. (2023). EZH2 controls epicardial cell migration during heart development. Life Science Alliance. 6(6). e202201765–e202201765. 8 indexed citations
14.
Fang, Xuexian, Zhaoxian Cai, Hao Wang, et al.. (2020). Loss of Cardiac Ferritin H Facilitates Cardiomyopathy via Slc7a11-Mediated Ferroptosis. Circulation Research. 127(4). 486–501. 569 indexed citations breakdown →
15.
Pei, Jian-Fei, Wenqi Li, Qian Gao, et al.. (2019). Diurnal oscillations of endogenous H2O2 sustained by p66Shc regulate circadian clocks. Nature Cell Biology. 21(12). 1553–1564. 89 indexed citations
16.
Tang, Xiaoqiang, Xiaofeng Chen, Xiaoman Wang, et al.. (2017). SIRT2 Acts as a Cardioprotective Deacetylase in Pathological Cardiac Hypertrophy. Circulation. 136(21). 2051–2067. 256 indexed citations
17.
Zhang, Dandan, Jian Xiong, Mulin Jun Li, et al.. (2017). Long noncoding RNA LINC00305 promotes inflammation by activating the AHRR-NF-κB pathway in human monocytes. Scientific Reports. 7(1). 46204–46204. 54 indexed citations
18.
Zhang, Yuan, Jing Xu, Yuxuan Luo, et al.. (2014). Overexpression of Mitofilin in the Mouse Heart Promotes Cardiac Hypertrophy in Response to Hypertrophic Stimuli. Antioxidants and Redox Signaling. 21(12). 1693–1707. 16 indexed citations
19.
Li, Li, Huina Zhang, Hou‐Zao Chen, et al.. (2011). SIRT1 Acts as a Modulator of Neointima Formation Following Vascular Injury in Mice. Circulation Research. 108(10). 1180–1189. 151 indexed citations
20.
Liu, Jinjing, Xi Yang, Ruifeng Yang, et al.. (2011). Two novel cis-elements involved in hepatocyte nuclear factor 4α regulation of acyl-coenzyme A:cholesterol acyltransferase 2 expression. Acta Biochimica et Biophysica Sinica. 44(2). 162–171. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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