Lora E. Bakeeva

1.3k total citations
16 papers, 680 citations indexed

About

Lora E. Bakeeva is a scholar working on Molecular Biology, Clinical Biochemistry and Physiology. According to data from OpenAlex, Lora E. Bakeeva has authored 16 papers receiving a total of 680 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 5 papers in Clinical Biochemistry and 3 papers in Physiology. Recurrent topics in Lora E. Bakeeva's work include Mitochondrial Function and Pathology (10 papers), Metabolism and Genetic Disorders (5 papers) and ATP Synthase and ATPases Research (3 papers). Lora E. Bakeeva is often cited by papers focused on Mitochondrial Function and Pathology (10 papers), Metabolism and Genetic Disorders (5 papers) and ATP Synthase and ATPases Research (3 papers). Lora E. Bakeeva collaborates with scholars based in Russia, Germany and Sweden. Lora E. Bakeeva's co-authors include Vladimir P. Skulachev, Olga Yu. Pletjushkina, Boris V. Chernyak, V. B. Saprunova, Lennart Brodin, Oleg Shupliakov, Andrei D. Vinogradov, Vera G. Grivennikova, Irina Gostimskaya and M. Yu. Vyssokikh and has published in prestigious journals such as Physiological Reviews, Analytical Biochemistry and Philosophical Transactions of the Royal Society B Biological Sciences.

In The Last Decade

Lora E. Bakeeva

14 papers receiving 666 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lora E. Bakeeva Russia 11 516 135 105 75 73 16 680
Keiichi Shibagaki Japan 8 226 0.4× 201 1.5× 61 0.6× 55 0.7× 35 0.5× 9 488
Carlos Buesa Spain 18 686 1.3× 131 1.0× 51 0.5× 127 1.7× 46 0.6× 38 969
Gillian Groeger Ireland 12 374 0.7× 96 0.7× 23 0.2× 105 1.4× 28 0.4× 12 646
Deepa V. Dabir United States 14 664 1.3× 286 2.1× 81 0.8× 168 2.2× 60 0.8× 17 1.0k
Yu. S. Chentsov Russia 8 529 1.0× 142 1.1× 145 1.4× 79 1.1× 29 0.4× 31 615
Lavoisier Ramos‐Espiritu United States 10 334 0.6× 138 1.0× 14 0.1× 105 1.4× 30 0.4× 12 597
Gábor Zsurka Germany 23 1.3k 2.4× 168 1.2× 487 4.6× 242 3.2× 60 0.8× 45 1.7k
Madhuparna Roy India 11 303 0.6× 118 0.9× 49 0.5× 71 0.9× 43 0.6× 23 473
Inês Pimenta de Castro United Kingdom 8 293 0.6× 129 1.0× 31 0.3× 67 0.9× 166 2.3× 9 501
Thomas Rival France 15 507 1.0× 210 1.6× 56 0.5× 394 5.3× 164 2.2× 18 1.0k

Countries citing papers authored by Lora E. Bakeeva

Since Specialization
Citations

This map shows the geographic impact of Lora E. Bakeeva's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lora E. Bakeeva with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lora E. Bakeeva more than expected).

Fields of papers citing papers by Lora E. Bakeeva

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lora E. Bakeeva. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lora E. Bakeeva. The network helps show where Lora E. Bakeeva may publish in the future.

Co-authorship network of co-authors of Lora E. Bakeeva

This figure shows the co-authorship network connecting the top 25 collaborators of Lora E. Bakeeva. A scholar is included among the top collaborators of Lora E. Bakeeva based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lora E. Bakeeva. Lora E. Bakeeva is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Popkov, Vasily A., Susanne Holtze, Thomas B. Hildebrandt, et al.. (2022). Unique Features of the Tissue Structure in the Naked Mole Rat (Heterocephalus glaber): Hypertrophy of the Endoplasmic Reticulum and Spatial Mitochondrial Rearrangements in Hepatocytes. International Journal of Molecular Sciences. 23(16). 9067–9067. 2 indexed citations
3.
Sheval, Eugene V., Susanne Holtze, Thomas B. Hildebrandt, et al.. (2020). Mitochondria in the Nuclei of Rat Myocardial Cells. Cells. 9(3). 712–712. 11 indexed citations
4.
Bakeeva, Lora E., et al.. (2019). Delayed Onset of Age-Dependent Changes in Ultrastructure of Myocardial Mitochondria as One of the Neotenic Features in Naked Mole Rats (Heterocephalus glaber). International Journal of Molecular Sciences. 20(3). 566–566. 7 indexed citations
5.
Shabalina, Irina G., M. Yu. Vyssokikh, Robert I. Csikasz, et al.. (2017). Improved health-span and lifespan in mtDNA mutator mice treated with the mitochondrially targeted antioxidant SkQ1. Aging. 9(2). 315–339. 81 indexed citations
6.
Skulachev, Vladimir P., Susanne Holtze, M. Yu. Vyssokikh, et al.. (2017). Neoteny, Prolongation of Youth: From Naked Mole Rats to “Naked Apes” (Humans). Physiological Reviews. 97(2). 699–720. 79 indexed citations
7.
Bakeeva, Lora E., et al.. (2016). Mitochondria-targeted antioxidant SkQ1 reduces age-related alterations in the ultrastructure of the lacrimal gland. Oncotarget. 7(49). 80208–80222. 10 indexed citations
8.
Колосова, Н. Г., et al.. (2014). Antioxidant SkQ1 delays sarcopenia-associated damage of mitochondrial ultrastructure. Aging. 6(2). 140–148. 43 indexed citations
9.
Knorre, Dmitry A., et al.. (2011). Accumulation of dodecyltriphenylphosphonium in mitochondria induces their swelling and ROS-dependent growth inhibition in yeast. Journal of Bioenergetics and Biomembranes. 43(2). 175–180. 8 indexed citations
10.
Фурсова, А. Ж., et al.. (2010). Alterations of retinal pigment epithelium cause AMD-like retinopathy in senescence-accelerated OXYS rats. Aging. 3(1). 44–54. 60 indexed citations
11.
Lyamzaev, Konstantin G., V. B. Saprunova, Lora E. Bakeeva, et al.. (2008). Novel mechanism of elimination of malfunctioning mitochondria (mitoptosis): Formation of mitoptotic bodies and extrusion of mitochondrial material from the cell. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1777(7-8). 817–825. 97 indexed citations
12.
Pletjushkina, Olga Yu., Konstantin G. Lyamzaev, V. B. Saprunova, et al.. (2008). S9.11 Novel mechanism of elimination of malfunctioning mitochondria (mitoptosis): Formation of mitoptotic bodies and extrusion of mitochondrial material from the cell. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1777. S57–S57. 1 indexed citations
13.
Skulachev, Vladimir P., Lora E. Bakeeva, Boris V. Chernyak, et al.. (2003). Thread-grain transition of mitochondrial reticulum as a step of mitoptosis and apoptosis. Molecular and Cellular Biochemistry. 256-257(1-2). 341–358. 117 indexed citations
14.
Gostimskaya, Irina, et al.. (2003). In situ assay of the intramitochondrial enzymes: use of alamethicin for permeabilization of mitochondria. Analytical Biochemistry. 313(1). 46–52. 84 indexed citations
15.
Brodin, Lennart, Lora E. Bakeeva, & Oleg Shupliakov. (1999). Presynaptic mitochondria and the temporal pattern of neurotransmitter release. Philosophical Transactions of the Royal Society B Biological Sciences. 354(1381). 365–372. 61 indexed citations
16.
Сыроешкин, А. В., Lora E. Bakeeva, & Dmitry A. Cherepanov. (1998). Contraction transitions of F1-F0 ATPase during catalytic turnover. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1409(2). 59–71. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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