Lin He

4.8k total citations
70 papers, 2.6k citations indexed

About

Lin He is a scholar working on Molecular Biology, Public Health, Environmental and Occupational Health and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Lin He has authored 70 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 13 papers in Public Health, Environmental and Occupational Health and 10 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Lin He's work include Reproductive Biology and Fertility (12 papers), Epigenetics and DNA Methylation (7 papers) and Birth, Development, and Health (5 papers). Lin He is often cited by papers focused on Reproductive Biology and Fertility (12 papers), Epigenetics and DNA Methylation (7 papers) and Birth, Development, and Health (5 papers). Lin He collaborates with scholars based in China, United States and United Kingdom. Lin He's co-authors include Dawei Li, David Collier, Lei Wang, Qing Sang, Biaobang Chen, Yanping Kuang, Jin Li, Bin Li, Zhizhou Zhang and Zheng Yan and has published in prestigious journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

Lin He

67 papers receiving 2.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lin He China 30 1.4k 705 510 403 336 70 2.6k
Xinzhi Zhao China 32 1.3k 0.9× 281 0.4× 467 0.9× 275 0.7× 269 0.8× 95 2.8k
Thomas B. Thompson United States 35 2.5k 1.8× 407 0.6× 772 1.5× 137 0.3× 206 0.6× 95 3.9k
Hao Huang China 32 1.4k 1.0× 328 0.5× 194 0.4× 251 0.6× 375 1.1× 170 3.4k
Fan Guo China 30 2.9k 2.1× 372 0.5× 655 1.3× 385 1.0× 91 0.3× 94 4.2k
Elena Rossi Italy 37 1.4k 1.0× 297 0.4× 1.2k 2.3× 505 1.3× 95 0.3× 141 3.9k
Dagmar Wachten Germany 28 962 0.7× 369 0.5× 464 0.9× 137 0.3× 440 1.3× 60 1.9k
Robert O. Heuckeroth United States 43 2.9k 2.1× 237 0.3× 704 1.4× 156 0.4× 87 0.3× 91 6.8k
Patrizia Bianchi Italy 25 650 0.5× 1.6k 2.3× 638 1.3× 397 1.0× 1.4k 4.2× 53 2.9k
Mark S. Roberson United States 35 1.6k 1.1× 708 1.0× 1.2k 2.4× 233 0.6× 997 3.0× 94 3.8k
Didier Wion France 35 1.7k 1.2× 156 0.2× 360 0.7× 224 0.6× 59 0.2× 87 4.3k

Countries citing papers authored by Lin He

Since Specialization
Citations

This map shows the geographic impact of Lin He's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lin He with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lin He more than expected).

Fields of papers citing papers by Lin He

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lin He. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lin He. The network helps show where Lin He may publish in the future.

Co-authorship network of co-authors of Lin He

This figure shows the co-authorship network connecting the top 25 collaborators of Lin He. A scholar is included among the top collaborators of Lin He based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lin He. Lin He is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Zhihua, Ran Yu, Zhi-Jing Wu, et al.. (2024). COX15 deficiency causes oocyte ferroptosis. Proceedings of the National Academy of Sciences. 121(45). e2406174121–e2406174121. 10 indexed citations
3.
4.
Wang, Hu, Geng Tian, Bo Xu, et al.. (2022). Offspring production of haploid spermatid-like cells derived from mouse female germline stem cells with chromatin condensation. Cell & Bioscience. 12(1). 5–5. 4 indexed citations
5.
Mu, Jian, Wenjing Wang, Biaobang Chen, et al.. (2019). Mutations in NLRP2 and NLRP5 cause female infertility characterised by early embryonic arrest. Journal of Medical Genetics. 56(7). 471–480. 102 indexed citations
6.
Deng, Qi, Ping Li, Jiajia Liu, et al.. (2019). Activation of hedgehog signaling in mesenchymal stem cells induces cartilage and bone tumor formation via Wnt/β-Catenin. eLife. 8. 87 indexed citations
7.
Sang, Qing, Bin Li, Yanping Kuang, et al.. (2018). Homozygous Mutations in WEE2 Cause Fertilization Failure and Female Infertility. The American Journal of Human Genetics. 102(4). 649–657. 136 indexed citations
8.
Hu, Jiaxin, Yan Bi, Lei Shi, et al.. (2018). No association of GRIN2A polymorphisms with the major depressive disorder in the Chinese Han origin. Psychiatric Genetics. 28(6). 120–121. 2 indexed citations
9.
Chen, Biaobang, Zhihua Zhang, Xiaoxi Sun, et al.. (2017). Biallelic Mutations in PATL2 Cause Female Infertility Characterized by Oocyte Maturation Arrest. The American Journal of Human Genetics. 101(4). 609–615. 112 indexed citations
11.
Sang, Qing, Ruizhi Feng, Luo Guo, et al.. (2015). Identification of a novel compound heterozygous mutation in PTPRQ in a DFNB84 family with prelingual sensorineural hearing impairment. Molecular Genetics and Genomics. 290(3). 1135–1139. 12 indexed citations
12.
Zheng, Jun, Wei‐Guo Ma, Mingjie Chen, et al.. (2014). Identification and Surgical Repair of Familial Thoracic Aortic Aneurysm and Dissection Caused by TGFBR1 Mutation. Annals of Vascular Surgery. 28(8). 1909–1912. 5 indexed citations
13.
Yang, Chao, Minle Li, Xuemei Tong, et al.. (2014). CYP2S1 depletion enhances colorectal cell proliferation is associated with PGE2-mediated activation of β-catenin signaling. Experimental Cell Research. 331(2). 377–386. 16 indexed citations
14.
Wang, Honglian, Yiran Xu, Mingjie Chen, et al.. (2013). Genetic association study of adaptor protein complex 4 with cerebral palsy in a Han Chinese population. Molecular Biology Reports. 40(11). 6459–6467. 10 indexed citations
15.
Wu, Xi, Kefu Tang, Yang Li, et al.. (2012). Quantitative assessment of the effect of LRRK2 exonic variants on the risk of Parkinson’s disease: A meta-analysis. Parkinsonism & Related Disorders. 18(6). 722–730. 26 indexed citations
16.
Liu, Huijuan, Wai Fook Leong, Lili Li, et al.. (2012). c-Abl promotes osteoblast expansion by differentially regulating canonical and non-canonical BMP pathways and p16INK4a expression. Nature Cell Biology. 14(7). 727–737. 50 indexed citations
17.
Shen, Qin, Xinzhi Zhao, Lan Yu, et al.. (2012). Association of Leukocyte Telomere Length with Type 2 Diabetes in Mainland Chinese Populations. The Journal of Clinical Endocrinology & Metabolism. 97(4). 1371–1374. 59 indexed citations
18.
Shen, Qin, Zhou Zhang, Lan Yu, et al.. (2011). Common variants near TERC are associated with leukocyte telomere length in the Chinese Han population. European Journal of Human Genetics. 19(6). 721–723. 50 indexed citations
19.
Ji, Baohu, Zhao Zhang, Ming Zhang, et al.. (2009). Differential expression profiling of the synaptosome proteome in a rat model of antipsychotic resistance. Brain Research. 1295. 170–178. 12 indexed citations
20.
Ji, Baohu, et al.. (2006). A Novel Deletion Mutation in CCM1 Gene (krit1) is Detected in a Chinese Family with Cerebral Cavernous Malformations. Acta Genetica Sinica. 33(2). 105–110. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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