Lih Fen Lue
- Neurology top 2%
- Physiology top 5%
- Neurology top 2%
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Co-authors
- Marwan N. SabbaghDouglas G. WalkerThomas G. BeachCharles H. AdlerJohn N. CavinessLucia I. SueLibuse BrachovaJack T. Rogers
- Topics
- Alzheimer's disease research and treatments (4 papers)Parkinson's Disease Mechanisms and Treatments (3 papers)Neurological diseases and metabolism (2 papers)
- Cited by
- NeurologyBiological Psychiatry
- Partner nations
- United StatesJapan
In The Last Decade
Lih Fen Lue
9 papers receiving 1.2k citations
Hit Papers
Peers
Comparison fields: 5 of 79
- Neurology 749
- Physiology 465
- Neurology 408
- Molecular Biology 217
- Cellular and Molecular Neuroscience 211
Countries citing papers authored by Lih Fen Lue
This map shows the geographic impact of Lih Fen Lue's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lih Fen Lue with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lih Fen Lue more than expected).
Fields of papers citing papers by Lih Fen Lue
This network shows the impact of papers produced by Lih Fen Lue. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lih Fen Lue. The network helps show where Lih Fen Lue may publish in the future.
Co-authorship network of co-authors of Lih Fen Lue
This figure shows the co-authorship network connecting the top 25 collaborators of Lih Fen Lue. A scholar is included among the top collaborators of Lih Fen Lue based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lih Fen Lue. Lih Fen Lue is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 65 | |
| 2 | Multi-organ distribution of phosphorylated α-synuclein histopathology in subjects with Lewy body disordersbreakdown → | 690 |
| 3 | 96 | |
| 4 | 78 | |
| 5 | Inflammatory mediators in the Alzheimer's disease brain | 2 |
| 6 | 2 | |
| 7 | 4 | |
| 8 | Inflammation, A beta deposition, and neurofibrillary tangle formation as correlates of Alzheimer's disease neurodegeneration. | 249 |
| 9 | 64 |
About Lih Fen Lue
Lih Fen Lue is a scholar working on Biological Psychiatry, Neurology and Physiology, having authored 9 papers that have together received 1.3k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (4 papers), Parkinson's Disease Mechanisms and Treatments (3 papers) and Neurological diseases and metabolism (2 papers). The work is most often cited by research in Neurology (408 citations), Neurology (749 citations) and Biological Psychiatry (66 citations). Lih Fen Lue has collaborated with scholars based in United States and Japan. Frequent co-authors include Marwan N. Sabbagh, Douglas G. Walker, Thomas G. Beach, Charles H. Adler, John N. Caviness, Lucia I. Sue, Libuse Brachova, Jack T. Rogers, Charles L. White and Linda Vedders. Their work appears in journals such as Acta Neuropathologica, Neurobiology of Aging and Expert Opinion on Investigational Drugs.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.