Li‐En Shao
Impact in
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- TGF-β signaling in diseases
- Bone Metabolism and Diseases
- Kruppel-like factors research
- Glycosylation and Glycoproteins Research
- Histone Deacetylase Inhibitors Research
- Metabolism, Diabetes, and Cancer
- Redox biology and oxidative stress
- Hematology top 10%
Papers in
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- Hematopoietic Stem Cell Transplantation 3
- Acute Myeloid Leukemia Research 2
Li‐En Shao
21 papers receiving 780 citations
Peers
Comparison fields: 5 of 70
- Molecular Biology 635
- Hematology 70
- Oncology 150
- Immunology 104
- Genetics 44
Countries citing papers authored by Li‐En Shao
This map shows the geographic impact of Li‐En Shao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Li‐En Shao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Li‐En Shao more than expected).
Fields of papers citing papers by Li‐En Shao
This network shows the impact of papers produced by Li‐En Shao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Li‐En Shao. The network helps show where Li‐En Shao may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Li‐En Shao, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 28 | |
| 2 | 2008 | 6 | |
| 3 | 3-amino thioacridone inhibits DNA synthesis and induce DNA damage in T-cell acute lymphoblastic leukemia (T-ALL) in a p16-dependent manner. | 2004 | 6 |
| 4 | 2004 | 1 | |
| 5 | 2002 | 29 | |
| 6 | 2002 | 64 | |
| 7 | Thioredoxin expression in primary T-cell acute lymphoblastic leukemia and its therapeutic implication. | 2001 | 53 |
| 8 | 2001 | 14 | |
| 9 | 2000 | 5 | |
| 10 | 2000 | 9 | |
| 11 | 2000 | 1 | |
| 12 | 1999 | 1 | |
| 13 | 1998 | 12 | |
| 14 | 1998 | 39 | |
| 15 | 1994 | 22 | |
| 16 | 1993 | 1 | |
| 17 | 1992 | 35 | |
| 18 | Regulation of production of activin A in human marrow stromal cells and monocytes. | 1992 | 82 |
| 19 | 1991 | 12 | |
| 20 | 1987 | 248 |
About Li‐En Shao
Li‐En Shao is a scholar working on Hematology, Immunology, Biotechnology, Molecular Biology and Physiology, having authored 21 papers that have together received 803 indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (5 papers), TGF-β signaling in diseases (5 papers), Metabolism, Diabetes, and Cancer (4 papers), Hematopoietic Stem Cell Transplantation (3 papers), Kruppel-like factors research (3 papers), Glycosylation and Glycoproteins Research (2 papers), Acute Myeloid Leukemia Research (2 papers) and Cancer Research and Treatments (2 papers). The work is most often cited by research in Molecular Biology (635 citations), Hematology (70 citations), Oncology (150 citations), Immunology (104 citations) and Genetics (44 citations). Li‐En Shao has collaborated with scholars based in United States, Taiwan and United Kingdom. Frequent co-authors include John Yu, Alice L. Yu, Joan Vaughan, Wylie Vale, V. Lemas, Jean Rivier, Normand L. Frigon, David Sehy, Mitchell B. Diccianni and Tetsuya S. Tanaka. Their work appears in journals such as Journal of Cellular Biochemistry, Annals of the New York Academy of Sciences, Leukemia Research, Cytokine and Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.