Li‐En Shao

929 total citations
21 papers, 803 citations indexed

About

Li‐En Shao is a scholar working on Molecular Biology, Hematology and Physiology. According to data from OpenAlex, Li‐En Shao has authored 21 papers receiving a total of 803 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 8 papers in Hematology and 5 papers in Physiology. Recurrent topics in Li‐En Shao's work include Erythrocyte Function and Pathophysiology (5 papers), TGF-β signaling in diseases (5 papers) and Metabolism, Diabetes, and Cancer (4 papers). Li‐En Shao is often cited by papers focused on Erythrocyte Function and Pathophysiology (5 papers), TGF-β signaling in diseases (5 papers) and Metabolism, Diabetes, and Cancer (4 papers). Li‐En Shao collaborates with scholars based in United States, Taiwan and United Kingdom. Li‐En Shao's co-authors include John Yu, Alice L. Yu, Joan Vaughan, Wylie Vale, V. Lemas, Jean Rivier, Normand L. Frigon, David Sehy, Mitchell B. Diccianni and Tetsuya S. Tanaka and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Blood.

In The Last Decade

Li‐En Shao

21 papers receiving 780 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Li‐En Shao United States 13 635 150 104 70 59 21 803
J A Truglia United States 13 406 0.6× 93 0.6× 120 1.2× 75 1.1× 55 0.9× 16 814
T Umiel United States 13 260 0.4× 158 1.1× 187 1.8× 78 1.1× 41 0.7× 27 642
S.H. Rosenberg United States 8 356 0.6× 182 1.2× 58 0.6× 140 2.0× 60 1.0× 12 588
Sandrine Jayne United Kingdom 18 578 0.9× 142 0.9× 104 1.0× 101 1.4× 65 1.1× 37 876
Paraskevi Kotsi Greece 10 428 0.7× 141 0.9× 217 2.1× 89 1.3× 78 1.3× 19 725
Valentina Rossi Italy 15 397 0.6× 239 1.6× 63 0.6× 79 1.1× 73 1.2× 26 773
Laura M. Rosenberg United States 6 639 1.0× 303 2.0× 91 0.9× 42 0.6× 83 1.4× 8 980
Pierluigi Benedetti Panici Italy 6 654 1.0× 194 1.3× 48 0.5× 40 0.6× 114 1.9× 9 837
Roberts Ab United States 11 376 0.6× 118 0.8× 45 0.4× 53 0.8× 54 0.9× 14 521
Zhijie Xiao China 18 569 0.9× 315 2.1× 136 1.3× 53 0.8× 125 2.1× 37 992

Countries citing papers authored by Li‐En Shao

Since Specialization
Citations

This map shows the geographic impact of Li‐En Shao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Li‐En Shao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Li‐En Shao more than expected).

Fields of papers citing papers by Li‐En Shao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Li‐En Shao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Li‐En Shao. The network helps show where Li‐En Shao may publish in the future.

Co-authorship network of co-authors of Li‐En Shao

This figure shows the co-authorship network connecting the top 25 collaborators of Li‐En Shao. A scholar is included among the top collaborators of Li‐En Shao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Li‐En Shao. Li‐En Shao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lai, Chao-Yang, et al.. (2011). Identification of Tumorigenic Cells in KrasG12D -Induced Lung Adenocarcinoma. Cancer Research. 71(23). 7250–7258. 28 indexed citations
2.
Chou, Ting‐Chao, et al.. (2008). BO-0742, a derivative of AHMA and N-mustard, has selective toxicity to drug sensitive and drug resistant leukemia cells and solid tumors. Cancer Letters. 276(2). 204–211. 6 indexed citations
3.
Diccianni, Mitchell B., John Yu, Li‐En Shao, et al.. (2004). 3-amino thioacridone inhibits DNA synthesis and induce DNA damage in T-cell acute lymphoblastic leukemia (T-ALL) in a p16-dependent manner.. PubMed. 4(3). 223–37. 6 indexed citations
4.
5.
Shao, Li‐En, et al.. (2002). Thioredoxin‐Related Regulation of NO/NOS Activities. Annals of the New York Academy of Sciences. 962(1). 140–150. 29 indexed citations
6.
Batova, Ayse, Li‐En Shao, Mitchell B. Diccianni, et al.. (2002). The histone deacetylase inhibitor AN-9 has selective toxicity to acute leukemia and drug-resistant primary leukemia and cancer cell lines. Blood. 100(9). 3319–3324. 64 indexed citations
7.
Shao, Li‐En, Mitchell B. Diccianni, Tetsuya S. Tanaka, et al.. (2001). Thioredoxin expression in primary T-cell acute lymphoblastic leukemia and its therapeutic implication.. Cancer Research. 61(19). 7333–8. 53 indexed citations
8.
Dialynas, Deno P., Li‐En Shao, Glenn F. Billman, & John Yu. (2001). Engraftment of Human T‐Cell Acute Lymphoblastic Leukemia in Immunodeficient NOD/SCID Mice Which Have Been Preconditioned by Injection of Human Cord Blood. Stem Cells. 19(5). 443–452. 14 indexed citations
9.
Shao, Li‐En, et al.. (2000). Human cord blood conditioned medium enhances leukemia colony formation in vitro. Leukemia Research. 24(12). 1041–1048. 5 indexed citations
10.
Liu, Fang, Li‐En Shao, & John Yu. (2000). Truncated activin type II receptor inhibits erythroid differentiation in K562 cells. Journal of Cellular Biochemistry. 78(1). 24–33. 9 indexed citations
11.
Liu, Fang, Li‐En Shao, & John Yu. (2000). Truncated activin type II receptor inhibits erythroid differentiation in K562 cells. Journal of Cellular Biochemistry. 78(1). 24–33. 1 indexed citations
12.
Dialynas, Deno P., et al.. (1999). FUNCTIONAL AND BIOCHEMICAL CHARACTERIZATION OF A NOVEL HUMAN MACROPHAGE-DERIVED NEGATIVE REGULATOR OF HAEMATOPOIESIS. Cytokine. 11(12). 985–995. 1 indexed citations
13.
Dolter, K E, et al.. (1998). Analysis of activin A gene expression in human bone marrow stromal cells. Journal of Cellular Biochemistry. 70(1). 8–21. 12 indexed citations
14.
15.
Shao, Li‐En, et al.. (1994). Detection of Functional and Dimeric Activin A in Human Marrow Microenvironment. Annals of the New York Academy of Sciences. 718(1). 285–299. 22 indexed citations
16.
Sehy, David, Li‐En Shao, Darryl Rideout, & John Yu. (1993). Sensitivity of committed hematopoietic progenitor cells in vitro (BFU-E, CFU-E, CFU-GM) and two human carcinoma cell lines toward rhodamine-123 and phosphonium salt II-41. Leukemia Research. 17(3). 247–253. 1 indexed citations
17.
Sehy, David, Li‐En Shao, Weimin Tsai, John Yu, & Alice L. Yu. (1992). Activin A‐induced diffrentiation in K562 cells is associated with a transient hypophosphorylation of RB protein and the concomitant block of cell cycle at G1 phase. Journal of Cellular Biochemistry. 50(3). 255–265. 35 indexed citations
18.
Shao, Li‐En, Normand L. Frigon, David Sehy, et al.. (1992). Regulation of production of activin A in human marrow stromal cells and monocytes.. PubMed. 20(10). 1235–42. 82 indexed citations
19.
Yu, John, Li‐En Shao, Normand L. Frigon, et al.. (1991). Specific Roles of Activin/Inhibin in Human Erythropoiesis in Vitroa. Annals of the New York Academy of Sciences. 628(1). 199–211. 12 indexed citations
20.
Yu, John, Li‐En Shao, V. Lemas, et al.. (1987). Importance of FSH-releasing protein and inhibin in erythrodifferentiation. Nature. 330(6150). 765–767. 248 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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