Lewis R. Chase
- Molecular Biology top 5%
- Nephrology top 1%
- Genetics top 5%
- Oncology top 10%
- Physiology top 10%
- Co-authors
- G.D. AurbachG. Leland MelsonAnthony S. PagliaraAlton L. SteinerDavid KipnisEduardo SlatopolskyGordon M. TomkinsD K Granner
- Topics
- Parathyroid Disorders and Treatments (12 papers)Ion Transport and Channel Regulation (5 papers)Metabolism, Diabetes, and Cancer (5 papers)
- Partner nations
- United StatesPolandUnited Kingdom
In The Last Decade
Lewis R. Chase
23 papers receiving 2.3k citations
Hit Papers
Peers
Comparison fields: 5 of 111
- Molecular Biology 1.3k
- Nephrology 597
- Genetics 501
- Oncology 385
- Physiology 331
Countries citing papers authored by Lewis R. Chase
This map shows the geographic impact of Lewis R. Chase's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lewis R. Chase with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lewis R. Chase more than expected).
Fields of papers citing papers by Lewis R. Chase
This network shows the impact of papers produced by Lewis R. Chase. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lewis R. Chase. The network helps show where Lewis R. Chase may publish in the future.
Co-authorship network of co-authors of Lewis R. Chase
This figure shows the co-authorship network connecting the top 25 collaborators of Lewis R. Chase. A scholar is included among the top collaborators of Lewis R. Chase based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lewis R. Chase. Lewis R. Chase is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 29 | |
| 2 | 10 | |
| 3 | 27 | |
| 4 | Phosphate accumulation by the isolated renal tubule and the effects of parathyroid hormone and cyclic adenosine monophosphate. | 3 |
| 5 | 9 | |
| 6 | 4 | |
| 7 | 32 | |
| 8 | 129 | |
| 9 | 19 | |
| 10 | 29 | |
| 11 | Radioimmunoassay for Cyclic Nucleotidesbreakdown → | 513 |
| 12 | 45 | |
| 13 | 109 | |
| 14 | 17 | |
| 15 | 172 | |
| 16 | 348 | |
| 17 | 102 | |
| 18 | 323 | |
| 19 | 307 | |
| 20 | 67 |
About Lewis R. Chase
Lewis R. Chase is a scholar working on Nephrology, Biochemistry and Pathology and Forensic Medicine, having authored 23 papers that have together received 2.6k indexed citations. Recurring topics across this work include Parathyroid Disorders and Treatments (12 papers), Ion Transport and Channel Regulation (5 papers) and Metabolism, Diabetes, and Cancer (5 papers). The work is most often cited by research in Nephrology (597 citations), Physiology (150 citations) and Molecular Biology (1.3k citations). Lewis R. Chase has collaborated with scholars based in United States, Poland and United Kingdom. Frequent co-authors include G.D. Aurbach, G. Leland Melson, Anthony S. Pagliara, Alton L. Steiner, David Kipnis, Eduardo Slatopolsky, Gordon M. Tomkins, D K Granner, Bruce M. Eberhart and L V Avioli. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.