Lee Levitt

949 total citations
24 papers, 667 citations indexed

About

Lee Levitt is a scholar working on Hematology, Immunology and Oncology. According to data from OpenAlex, Lee Levitt has authored 24 papers receiving a total of 667 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Hematology, 10 papers in Immunology and 6 papers in Oncology. Recurrent topics in Lee Levitt's work include Immune Cell Function and Interaction (9 papers), Immune Response and Inflammation (5 papers) and T-cell and B-cell Immunology (5 papers). Lee Levitt is often cited by papers focused on Immune Cell Function and Interaction (9 papers), Immune Response and Inflammation (5 papers) and T-cell and B-cell Immunology (5 papers). Lee Levitt collaborates with scholars based in United States, Germany and France. Lee Levitt's co-authors include Peter J. Quesenberry, Jin‐hong Park, Albert Y. Lin, George A. Fisher, K Kaushansky, M Shatsky, Sam So, N A Brophy, Christopher Biggs and Torunn I. Yock and has published in prestigious journals such as New England Journal of Medicine, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Lee Levitt

24 papers receiving 616 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Lee Levitt United States	13	205	200	183	132	85	24	667
John Scopes United Kingdom	13	311 1.5×	291 1.5×	211 1.2×	196 1.5×	139 1.6×	18	756
Yuh‐Ching Twu Taiwan	18	269 1.3×	263 1.3×	175 1.0×	56 0.4×	83 1.0×	36	737
M.A. Gross United States	12	184 0.9×	156 0.8×	306 1.7×	139 1.1×	52 0.6×	21	687
J.P. Salier France	12	138 0.7×	258 1.3×	141 0.8×	36 0.3×	68 0.8×	30	538
Greenberger Js United States	13	245 1.2×	303 1.5×	228 1.2×	134 1.0×	86 1.0×	26	701
Gertraud Fröschl Austria	15	401 2.0×	597 3.0×	193 1.1×	269 2.0×	55 0.6×	17	1.3k
Masato Yagita Japan	17	188 0.9×	243 1.2×	618 3.4×	290 2.2×	74 0.9×	52	1.1k
H Uchino Japan	13	47 0.2×	203 1.0×	147 0.8×	134 1.0×	44 0.5×	22	527
Ágota Szepesi Hungary	19	112 0.5×	368 1.8×	244 1.3×	317 2.4×	216 2.5×	49	997
Yoshiko Matsumoto Japan	16	166 0.8×	382 1.9×	231 1.3×	194 1.5×	107 1.3×	39	897

Countries citing papers authored by Lee Levitt

Since Specialization

Citations

This map shows the geographic impact of Lee Levitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lee Levitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lee Levitt more than expected).

Fields of papers citing papers by Lee Levitt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lee Levitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lee Levitt. The network helps show where Lee Levitt may publish in the future.

Co-authorship network of co-authors of Lee Levitt

This figure shows the co-authorship network connecting the top 25 collaborators of Lee Levitt. A scholar is included among the top collaborators of Lee Levitt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lee Levitt. Lee Levitt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Lin, Albert Y., George A. Fisher, Samuel So, Christopher S. Tang, & Lee Levitt. (2008). Phase II Study of Imatinib in Unresectable Hepatocellular Carcinoma. American Journal of Clinical Oncology. 31(1). 84–88. 28 indexed citations

Lin, Allen, George A. Fisher, Sam So, Chao Tang, & Lee Levitt. (2005). A phase II study of imatinib mesylate (IM) in patients (pts) with unresectable hepatocellular carcinoma (HCC). Journal of Clinical Oncology. 23(16_suppl). 4223–4223. 3 indexed citations

Lin, Albert Y., N A Brophy, George A. Fisher, et al.. (2004). Phase II study of thalidomide in patients with unresectable hepatocellular carcinoma. Cancer. 103(1). 119–125. 51 indexed citations

Dilloo, Dagmar, et al.. (1996). Differential production of interleukin-3 in human T lymphocytes following either CD3 or CD2 receptor activation.. PubMed. 24(4). 537–43. 3 indexed citations

Miyajima, Ikuko, Lee Levitt, Takahiko Hara, et al.. (1995). The murine interleukin-3 receptor alpha subunit gene: chromosomal localization, genomic structure, and promoter function. Blood. 85(5). 1246–1253. 36 indexed citations

Park, Jin‐hong & Lee Levitt. (1993). Overexpression of mitogen-activated protein kinase (ERK1) enhances T- cell cytokine gene expression: role of AP1, NF-AT, and NF-KB. Blood. 82(8). 2470–2477. 48 indexed citations

Park, Jin‐hong, K Kaushansky, & Lee Levitt. (1993). Transcriptional regulation of interleukin 3 (IL3) in primary human T lymphocytes. Role of AP-1- and octamer-binding proteins in control of IL3 gene expression.. Journal of Biological Chemistry. 268(9). 6299–6308. 30 indexed citations

Dilloo, Dagmar, et al.. (1991). Differential regulation of lymphokine production by distinct subunits of the T cell interleukin 2 receptor.. Journal of Clinical Investigation. 87(6). 2114–2121. 27 indexed citations

Burdach, S., Lee Levitt, V. Wahn, & U. Göbel. (1989). Lymphokin-vermittelte Suppression der Erythropoese durch normale T-Lymphozyten und bei Retrovirus-assoziierter lymphoproliferativer Erkrankung. Oncology Research and Treatment. 12(3). 116–119. 1 indexed citations

10.

Levitt, Lee, et al.. (1988). T cell regulated hematopoiesis--molecular interactions in hematopoietic control by CD2 and interleukin 2.. PubMed. 56–67. 2 indexed citations

11.

Burdach, Stefan, et al.. (1988). The T-cell CD2 determinant mediates inhibition of erythropoiesis by the lymphokine cascade. Blood. 72(2). 770–775. 7 indexed citations

12.

Burdach, Stefan, et al.. (1988). The T-cell CD2 determinant mediates inhibition of erythropoiesis by the lymphokine cascade. Blood. 72(2). 770–775. 3 indexed citations

13.

Alter, Milton, Sue Min Lai, Gary Friday, et al.. (1987). Acute stroke, hematocrit, and blood pressure.. Stroke. 18(3). 565–569. 18 indexed citations

14.

Levitt, Lee, et al.. (1987). Augmentation of in vitro human marrow erythropoiesis under physiological oxygen tensions is mediated by monocytes and T lymphocytes. Blood. 69(3). 899–907. 48 indexed citations

15.

Burdach, Stefan, et al.. (1987). Receptor-specific modulation of myelopoiesis by recombinant DNA-derived interleukin 2.. The Journal of Immunology. 139(2). 452–458. 13 indexed citations

16.

Levitt, Lee. (1985). Public relations as a source of power. Public Relations Review. 11(3). 3–9. 8 indexed citations

17.

Quesenberry, PJ, et al.. (1984). Studies of the control of hemopoiesis in Dexter cultures.. PubMed. 18. 171–93. 1 indexed citations

18.

Quesenberry, P. J., Lee Levitt, Francis C. Monette, et al.. (1979). The use of stem cell assays to monitor the proliferative potential of bone marrow cells.. PubMed. 7 Suppl 5. 210–27. 2 indexed citations

19.

Quesenberry, Peter J. & Lee Levitt. (1979). Hematopoietic Stem Cells. New England Journal of Medicine. 301(16). 868–872. 52 indexed citations

20.

Quesenberry, Peter J. & Lee Levitt. (1979). Hematopoietic Stem Cells. New England Journal of Medicine. 301(14). 755–760. 152 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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