Lea Bleier
Impact in
- Clinical Biochemistry top 5%
- Metabolism and Genetic Disorders
- Molecular Biology top 10%
- Mitochondrial Function and Pathology
- ATP Synthase and ATPases Research
- Photosynthetic Processes and Mechanisms
- Redox biology and oxidative stress
- Metabolomics and Mass Spectrometry Studies
Papers in ⓘ
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- Metabolism and Genetic Disorders 1
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- Cardiac Arrest and Resuscitation 1
- Co-authors
- Stefan Dröse (5 shared papers)Ulrich Brandt (4 shared papers)Ilka Wittig (6 shared papers)Heinrich Heide (4 shared papers)Mirco Steger (4 shared papers)Jörg Ackermann (2 shared papers)Ina Koch (2 shared papers)Bettina Schwamb (1 shared paper)
- Journals
- Biochimica et Biophysica Acta (BBA) - Bioenergetics (3 papers)Cell Metabolism (1 paper)Molecular Pharmacology (1 paper)Free Radical Biology and Medicine (1 paper)Bioinformatics (1 paper)
- Partner nations
- GermanyNetherlandsUnited States
In The Last Decade
Lea Bleier
8 papers receiving 874 citations
Peers
Comparison fields: 5 of 82
- Clinical Biochemistry 120
- Molecular Biology 698
- Aging 16
- Biochemistry 42
- Cancer Research 66
Countries citing papers authored by Lea Bleier
This map shows the geographic impact of Lea Bleier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lea Bleier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lea Bleier more than expected).
Fields of papers citing papers by Lea Bleier
This network shows the impact of papers produced by Lea Bleier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lea Bleier. The network helps show where Lea Bleier may publish in the future.
Co-authors
The 16 scholars most cited alongside Lea Bleier, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 288 | |
| 2 | 2012 | 224 | |
| 3 | 2014 | 149 | |
| 4 | 2010 | 88 | |
| 5 | 2014 | 63 | |
| 6 | 2011 | 56 | |
| 7 | 2013 | 9 | |
| 8 | 2010 | 3 |
About Lea Bleier
Lea Bleier is a scholar working on Clinical Biochemistry, Emergency Medicine, Molecular Biology, Cancer Research and Spectroscopy, having authored 8 papers that have together received 880 indexed citations. Recurring topics across this work include ATP Synthase and ATPases Research (6 papers), Mitochondrial Function and Pathology (6 papers), Metabolism and Genetic Disorders (1 paper), Cardiac electrophysiology and arrhythmias (1 paper), Coenzyme Q10 studies and effects (1 paper), Cardiac Arrest and Resuscitation (1 paper), Cancer, Hypoxia, and Metabolism (1 paper) and Metabolomics and Mass Spectrometry Studies (1 paper). The work is most often cited by research in Clinical Biochemistry (120 citations), Molecular Biology (698 citations), Aging (16 citations), Biochemistry (42 citations) and Cancer Research (66 citations). Lea Bleier has collaborated with scholars based in Germany, Netherlands and United States. Frequent co-authors include Stefan Dröse, Ulrich Brandt, Ilka Wittig, Heinrich Heide, Mirco Steger, Jörg Ackermann, Ina Koch, Bettina Schwamb, Andreas S. Reichert and Martin Zörnig. Their work appears in journals such as Biochimica et Biophysica Acta (BBA) - Bioenergetics, Cell Metabolism, Molecular Pharmacology, Free Radical Biology and Medicine and Bioinformatics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.