Laure Willen

1.2k total citations
24 papers, 789 citations indexed

About

Laure Willen is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Laure Willen has authored 24 papers receiving a total of 789 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 11 papers in Immunology and 5 papers in Cancer Research. Recurrent topics in Laure Willen's work include T-cell and B-cell Immunology (7 papers), dental development and anomalies (6 papers) and Immunodeficiency and Autoimmune Disorders (5 papers). Laure Willen is often cited by papers focused on T-cell and B-cell Immunology (7 papers), dental development and anomalies (6 papers) and Immunodeficiency and Autoimmune Disorders (5 papers). Laure Willen collaborates with scholars based in Switzerland, Germany and United States. Laure Willen's co-authors include Pascal Schneider, Aubry Tardivel, Cristian R. Smulski, Fabienne Mackay, Claudia Bossen, Martin Scott, Helen Leung, Hans Acha‐Orbea, Teresa G. Cachero and Max Dobles and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

Laure Willen

24 papers receiving 776 citations

Peers

Laure Willen
Mihm Mc United States
Jeffrey A. Carrell United States
W Strober United States
C Irlé Switzerland
Patricia Lugar United States
Mohammad Luqman United States
Mihm Mc United States
Laure Willen
Citations per year, relative to Laure Willen Laure Willen (= 1×) peers Mihm Mc

Countries citing papers authored by Laure Willen

Since Specialization
Citations

This map shows the geographic impact of Laure Willen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Laure Willen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Laure Willen more than expected).

Fields of papers citing papers by Laure Willen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Laure Willen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Laure Willen. The network helps show where Laure Willen may publish in the future.

Co-authorship network of co-authors of Laure Willen

This figure shows the co-authorship network connecting the top 25 collaborators of Laure Willen. A scholar is included among the top collaborators of Laure Willen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Laure Willen. Laure Willen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schuepbach‐Mallepell, Sonia, Laure Willen, Christine Kowalczyk‐Quintas, et al.. (2024). Unique and redundant roles of mouse BCMA, TACI, BAFF, APRIL, and IL-6 in supporting antibody-producing cells in different tissues. Proceedings of the National Academy of Sciences. 121(29). e2404309121–e2404309121. 11 indexed citations
2.
Heppelmann, Maike, Marion Hewicker‐Trautwein, Maren Hellige, et al.. (2023). A Missense Mutation in the Collagen Triple Helix of EDA Is Associated with X-Linked Recessive Hypohidrotic Ectodermal Dysplasia in Fleckvieh Cattle. Genes. 15(1). 8–8. 1 indexed citations
3.
Smulski, Cristian R., Luyao Zhang, Michele Vigolo, et al.. (2022). Ligand-independent oligomerization of TACI is controlled by the transmembrane domain and regulates proliferation of activated B cells. Cell Reports. 38(13). 110583–110583. 11 indexed citations
4.
Papasouliotis, Orestis, et al.. (2022). Kinetics of free and ligand-bound atacicept in human serum. Frontiers in Immunology. 13. 1035556–1035556. 2 indexed citations
5.
Schneider, Holm, et al.. (2022). Functional and clinical analysis of five EDA variants associated with ectodermal dysplasia but with a hard-to-predict significance. Frontiers in Genetics. 13. 934395–934395. 4 indexed citations
6.
Kowalczyk‐Quintas, Christine, et al.. (2018). Inhibition of Membrane-Bound BAFF by the Anti-BAFF Antibody Belimumab. Frontiers in Immunology. 9. 2698–2698. 30 indexed citations
7.
Smulski, Cristian R., Lea Seidel, Harald Staiger, et al.. (2017). BAFF- and TACI-Dependent Processing of BAFFR by ADAM Proteases Regulates the Survival of B Cells. Cell Reports. 18(9). 2189–2202. 54 indexed citations
8.
Smulski, Cristian R., Marion Décossas, Julien Beyrath, et al.. (2017). Hetero-oligomerization between the TNF receptor superfamily members CD40, Fas and TRAILR2 modulate CD40 signalling. Cell Death and Disease. 8(2). e2601–e2601. 25 indexed citations
9.
Kowalczyk‐Quintas, Christine, Sonia Schuepbach‐Mallepell, Michele Vigolo, et al.. (2016). Antibodies That Block or Activate Mouse B Cell Activating Factor of the Tumor Necrosis Factor (TNF) Family (BAFF), Respectively, Induce B Cell Depletion or B Cell Hyperplasia. Journal of Biological Chemistry. 291(38). 19826–19834. 15 indexed citations
10.
Chypre, Mélanie, Laure Willen, Kathryn A. Knoop, et al.. (2016). Characterization and application of two RANK-specific antibodies with different biological activities. Immunology Letters. 171. 5–14. 3 indexed citations
11.
Schuepbach‐Mallepell, Sonia, Laure Willen, Michele Vigolo, et al.. (2015). Stoichiometry of Heteromeric BAFF and APRIL Cytokines Dictates Their Receptor Binding and Signaling Properties. Journal of Biological Chemistry. 290(26). 16330–16342. 37 indexed citations
12.
Kowalczyk‐Quintas, Christine, Laure Willen, Anh Thu Dang, et al.. (2014). Generation and Characterization of Function-blocking Anti-ectodysplasin A (EDA) Monoclonal Antibodies That Induce Ectodermal Dysplasia. Journal of Biological Chemistry. 289(7). 4273–4285. 24 indexed citations
13.
Schneider, Pascal, Laure Willen, & Cristian R. Smulski. (2014). Tools and Techniques to Study Ligand–Receptor Interactions and Receptor Activation by TNF Superfamily Members. Methods in enzymology on CD-ROM/Methods in enzymology. 545. 103–125. 33 indexed citations
14.
Kowalczyk‐Quintas, Christine, Sonia Schuepbach‐Mallepell, Laure Willen, et al.. (2014). Pharmacological Stimulation of Edar Signaling in the Adult Enhances Sebaceous Gland Size and Function. Journal of Investigative Dermatology. 135(2). 359–368. 8 indexed citations
15.
Nys, Josquin, Cristian R. Smulski, Aubry Tardivel, et al.. (2013). No Evidence That Soluble TACI Induces Signalling via Membrane-Expressed BAFF and APRIL in Myeloid Cells. PLoS ONE. 8(4). e61350–e61350. 14 indexed citations
16.
Pellegrini, Maria, Laure Willen, Mai Perroud, et al.. (2013). Structure of the extracellular domains of human and Xenopus Fn14: implications in the evolution of TWEAK and Fn14 interactions. FEBS Journal. 280(8). 1818–1829. 5 indexed citations
17.
Dunkel, N., Laure Willen, Margret L. Casal, et al.. (2011). Molecular and Therapeutic Characterization of Anti-ectodysplasin A Receptor (EDAR) Agonist Monoclonal Antibodies. Journal of Biological Chemistry. 286(35). 30769–30779. 32 indexed citations
18.
Bossen, Claudia, Aubry Tardivel, Laure Willen, et al.. (2010). Mutation of the BAFF furin cleavage site impairs B‐cell homeostasis and antibody responses. European Journal of Immunology. 41(3). 787–797. 50 indexed citations
19.
Mues, Gabriele, Aubry Tardivel, Laure Willen, et al.. (2009). Functional analysis of Ectodysplasin-A mutations causing selective tooth agenesis. European Journal of Human Genetics. 18(1). 19–25. 58 indexed citations
20.
Swee, Lee Kim, Aubry Tardivel, Laure Willen, et al.. (2009). Biological Activity of Ectodysplasin A Is Conditioned by Its Collagen and Heparan Sulfate Proteoglycan-binding Domains. Journal of Biological Chemistry. 284(40). 27567–27576. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026