Laura Llaó Cid

659 total citations
8 papers, 108 citations indexed

About

Laura Llaó Cid is a scholar working on Immunology, Genetics and Oncology. According to data from OpenAlex, Laura Llaó Cid has authored 8 papers receiving a total of 108 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 6 papers in Genetics and 4 papers in Oncology. Recurrent topics in Laura Llaó Cid's work include Chronic Lymphocytic Leukemia Research (6 papers), Immune Cell Function and Interaction (5 papers) and CAR-T cell therapy research (4 papers). Laura Llaó Cid is often cited by papers focused on Chronic Lymphocytic Leukemia Research (6 papers), Immune Cell Function and Interaction (5 papers) and CAR-T cell therapy research (4 papers). Laura Llaó Cid collaborates with scholars based in Germany, Spain and Sweden. Laura Llaó Cid's co-authors include Philipp M. Roessner, Martina Seiffert, Dolors Colomer, Bola S. Hanna, Stephan Stilgenbauer, Peter Lichter, Selcen Öztürk, Sascha Dietrich, Tobias Roider and Ana Ízcue and has published in prestigious journals such as Blood, FEBS Letters and Frontiers in Immunology.

In The Last Decade

Laura Llaó Cid

8 papers receiving 108 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Laura Llaó Cid Germany	5	67	48	46	28	21	8	108
Emily M. Thrash United States	6	54 0.8×	44 0.9×	40 0.9×	60 2.1×	20 1.0×	8	116
Rosalie de Beaumont United States	3	43 0.6×	51 1.1×	19 0.4×	31 1.1×	34 1.6×	3	104
Brittney Dienes United States	3	53 0.8×	29 0.6×	36 0.8×	14 0.5×	36 1.7×	11	89
Esther Hian Li Chan Singapore	6	28 0.4×	66 1.4×	17 0.4×	27 1.0×	43 2.0×	17	106
Sophie Langdon United Kingdom	2	51 0.8×	68 1.4×	14 0.3×	42 1.5×	19 0.9×	3	111
D. H. Kim South Korea	2	47 0.7×	45 0.9×	68 1.5×	50 1.8×	80 3.8×	2	156
Zhiming Li China	7	27 0.4×	40 0.8×	22 0.5×	11 0.4×	65 3.1×	12	87
Anna Reischer Germany	5	36 0.5×	32 0.7×	13 0.3×	24 0.9×	6 0.3×	8	85
Ralph Schulz Germany	5	66 1.0×	37 0.8×	59 1.3×	29 1.0×	35 1.7×	9	119
Reinhard Dummer Italy	6	50 0.7×	118 2.5×	17 0.4×	56 2.0×	6 0.3×	13	148

Countries citing papers authored by Laura Llaó Cid

Since Specialization

Citations

This map shows the geographic impact of Laura Llaó Cid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Laura Llaó Cid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Laura Llaó Cid more than expected).

Fields of papers citing papers by Laura Llaó Cid

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Laura Llaó Cid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Laura Llaó Cid. The network helps show where Laura Llaó Cid may publish in the future.

Co-authorship network of co-authors of Laura Llaó Cid

This figure shows the co-authorship network connecting the top 25 collaborators of Laura Llaó Cid. A scholar is included among the top collaborators of Laura Llaó Cid based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Laura Llaó Cid. Laura Llaó Cid is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Cid, Laura Llaó. (2025). Single‐cell insights into the role of T cells in B‐cell malignancies. FEBS Letters. 599(20). 2932–2951. 1 indexed citations

2.

Cid, Laura Llaó, Tobias Roider, Dolors Colomer, et al.. (2022). Single-Cell Omics Analyses Identify the TIM-3 Ligand Galectin-9 As Novel Immunotherapy Target for Chronic Lymphocytic Leukemia. Blood. 140(Supplement 1). 1805–1806. 1 indexed citations

3.

Roessner, Philipp M., Laura Llaó Cid, Tobias Roider, et al.. (2021). EOMES and IL-10 regulate antitumor activity of T regulatory type 1 CD4+ T cells in chronic lymphocytic leukemia. Leukemia. 35(8). 2311–2324. 30 indexed citations

4.

Cid, Laura Llaó, Philipp M. Roessner, Vicente Chapaprieta, et al.. (2021). EOMES is essential for antitumor activity of CD8+ T cells in chronic lymphocytic leukemia. Leukemia. 35(11). 3152–3162. 29 indexed citations

5.

Aydin, Ebru, et al.. (2021). Phosphoinositide 3-Kinase Signaling in the Tumor Microenvironment: What Do We Need to Consider When Treating Chronic Lymphocytic Leukemia With PI3K Inhibitors?. Frontiers in Immunology. 11. 595818–595818. 17 indexed citations

6.

Roessner, Philipp M., Bola S. Hanna, Selcen Öztürk, et al.. (2019). TBET‐expressing Th1 CD4⁺T cells accumulate in chronic lymphocytic leukaemia without affecting disease progression in Eµ‐TCL1 mice. British Journal of Haematology. 189(1). 133–145. 13 indexed citations

7.

Cid, Laura Llaó, Bola S. Hanna, Murat Iskar, et al.. (2019). CD8⁺ T-cells of CLL-bearing mice acquire a transcriptional program of T-cell activation and exhaustion. Leukemia & lymphoma. 61(2). 351–356. 16 indexed citations

8.

Haderk, Franziska, Laura Llaó Cid, Murat Iskar, et al.. (2016). CLL Exosome-Derived Y RNA hY4 Induces TLR7/8-Mediated Inflammation and PD-L1 Expression in Monocytes. Blood. 128(22). 3217–3217. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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